Deletions within the 5'UTR of coxsackievirus B3: consequences for virus translation and replication.
about
Coxsackievirus B3 inhibits antigen presentation in vivo, exerting a profound and selective effect on the MHC class I pathway.A leaderless genome identified during persistent bovine coronavirus infection is associated with attenuation of gene expression.Modification of the untranslated regions of human enterovirus 71 impairs growth in a cell-specific manner.Wild-type coxsackievirus infection dramatically alters the abundance, heterogeneity, and immunostimulatory capacity of conventional dendritic cells in vivoCoxsackievirus can exploit LC3 in both autophagy-dependent and -independent manners in vivo.Mutational Disruption of cis-Acting Replication Element 2C in Coxsackievirus B3 Leads to 5'-Terminal Genomic Deletions.Replication of coxsackievirus B3 in primary cell cultures generates novel viral genome deletionsEnumeration and functional evaluation of virus-specific CD4+ and CD8+ T cells in lymphoid and peripheral sites of coxsackievirus B3 infectionCoxsackievirus B3 infects the bone marrow and diminishes the restorative capacity of erythroid and lymphoid progenitors.Ribosomal Initiation Complex Assembly within the Wild-Strain of Coxsackievirus B3 and Live-Attenuated Sabin3-like IRESes during the Initiation of TranslationViral persistence and chronic immunopathology in the adult central nervous system following Coxsackievirus infection during the neonatal period.In vivo ablation of type I interferon receptor from cardiomyocytes delays coxsackieviral clearance and accelerates myocardial disease.Role of RNA structure motifs in IRES-dependent translation initiation of the coxsackievirus B3: new insights for developing live-attenuated strains for vaccines and gene therapy.Coxsackievirus B3 replication and pathogenesis.5' terminal deletions in the genome of a coxsackievirus B2 strain occurred naturally in human heart.Functional Consequences of RNA 5'-Terminal Deletions on Coxsackievirus B3 RNA Replication and Ribonucleoprotein Complex Formation.Enterovirus but not Parvovirus B19 is associated with idiopathic dilated cardiomyopathy and endomyocardial CD3, CD68, or HLA-DR expression.Immunological and pathological consequences of coxsackievirus RNA persistence in the heart.Impaired binding of standard initiation factors eIF3b, eIF4G and eIF4B to domain V of the live-attenuated coxsackievirus B3 Sabin3-like IRES--alternatives for 5'UTR-related cardiovirulence mechanisms.Coxsackievirus infection induces autophagy-like vesicles and megaphagosomes in pancreatic acinar cells in vivo.MCPIP1 inhibits coxsackievirus B3 replication by targeting viral RNA and negatively regulates virus-induced inflammation.
P2860
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P2860
Deletions within the 5'UTR of coxsackievirus B3: consequences for virus translation and replication.
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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2006年学术文章
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2006年学术文章
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name
Deletions within the 5'UTR of ...... s translation and replication.
@ast
Deletions within the 5'UTR of ...... s translation and replication.
@en
type
label
Deletions within the 5'UTR of ...... s translation and replication.
@ast
Deletions within the 5'UTR of ...... s translation and replication.
@en
prefLabel
Deletions within the 5'UTR of ...... s translation and replication.
@ast
Deletions within the 5'UTR of ...... s translation and replication.
@en
P2093
P2860
P1433
P1476
Deletions within the 5'UTR of ...... s translation and replication.
@en
P2093
Christopher T Cornell
Isabelle P Hunziker
J Lindsay Whitton
P2860
P304
P356
10.1016/J.VIROL.2006.09.041
P407
P577
2006-11-03T00:00:00Z