CD4+ effector cells default to the Th2 pathway in interferon gamma-deficient mice infected with Leishmania major - Wikidata - awgldk - TriplyDB

CD4+ effector cells default to the Th2 pathway in interferon gamma-deficient mice infected with Leishmania major

CD4+ effector cells default to the Th2 pathway in interferon gamma-deficient mice infected with Leishmania major

http://www.wikidata.org/entity/Q36362926

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Mutation in the signal-transducing chain of the interferon-gamma receptor and susceptibility to mycobacterial infection Cytokine production and dysregulation in HIV pathogenesis: lessons for development of therapeutics and vaccines Distinct dendritic cell subsets actively induce Th2 polarization IL-22 Protects against Tissue Damage during Cutaneous Leishmaniasis The host-protective effect of arabinosylated lipoarabinomannan against Leishmania donovani infection is associated with restoration of IFN-γ responsiveness iNOS-producing inflammatory dendritic cells constitute the major infected cell type during the chronic Leishmania major infection phase of C57BL/6 resistant mice Roles of endogenous gamma interferon and macrophage microbicidal mechanisms in host response to chemotherapy in experimental visceral leishmaniasis Successful therapy of chronic, nonhealing murine cutaneous leishmaniasis with sodium stibogluconate and gamma interferon depends on continued interleukin-12 production Schistosome-derived omega-1 drives Th2 polarization by suppressing protein synthesis following internalization by the mannose receptor.Collective nitric oxide production provides tissue-wide immunity during Leishmania infection.Enzyme and cytokine effects on the impaired onset of the murine foreign-body reaction to dermal sheep collagen.Leishmania major infection of inbred mice: unmasking genetic determinants of infectious diseases.The gamma interferon receptor is required for the protective pulmonary inflammatory response to Cryptococcus neoformans.The transcription factors Thpok and LRF are necessary and partly redundant for T helper cell differentiation Some of the early events underlying Th2 cell maturation and susceptibility to Leishmania major infection in BALB/c mice.Regulation of T helper cell differentiation by respiratory tract dendritic cells.Increased capacity for interleukin-2 synthesis parallels disease progression in mice infected with Leishmania major Gamma interferon (IFN-gamma) receptor null-mutant mice are more susceptible to herpes simplex virus type 1 infection than IFN-gamma ligand null-mutant mice Protective T-cell-based immunity induced in neonatal mice by a single replicative cycle of herpes simplex virus The immune response modifier imiquimod requires STAT-1 for induction of interferon, interferon-stimulated genes, and interleukin-6.Replication-defective adenovirus infection reduces Helicobacter felis colonization in the mouse in a gamma interferon- and interleukin-12-dependent manner.Bartonella henselae-specific cell-mediated immune responses display a predominantly Th1 phenotype in experimentally infected C57BL/6 mice.Autoimmune mechanisms of depletion of CD4 cells in HIV infection.Interleukin-4 (IL-4) and IL-10 collude in vaccine failure for novel exacerbatory antigens in murine Leishmania major infection Redundant Notch1 and Notch2 signaling is necessary for IFNγ secretion by T helper 1 cells during infection with Leishmania major.MHC class II restricted innate-like double negative T cells contribute to optimal primary and secondary immunity to Leishmania major Deception and manipulation: the arms of leishmania, a successful parasite Interferon-gamma and interlukin-4 patterns in BALB/c mice suffering from cutaneous leishmaniasis treated with cantharidin.Role of cytokines in the innate immune response to intracellular pathogens Blocking junctional adhesion molecule C enhances dendritic cell migration and boosts the immune responses against Leishmania major.CD40-CD40 ligand costimulation is not required for initiation and maintenance of a Th1-type response to Leishmania major infection.Deficiency of Leishmania phosphoglycans influences the magnitude but does not affect the quality of secondary (memory) anti-Leishmania immunity Perforin and gamma interferon are critical CD8+ T-cell-mediated responses in vaccine-induced immunity against Leishmania amazonensis infection.Differential induction of TLR3-dependent innate immune signaling by closely related parasite species.Dissemination of Chlamydia trachomatis chronic genital tract infection in gamma interferon gene knockout mice.Enhanced replication of Leishmania amazonensis amastigotes in gamma interferon-stimulated murine macrophages: implications for the pathogenesis of cutaneous leishmaniasis.Profiles of healing and nonhealing Cryptosporidium parvum infection in C57BL/6 mice with functional B and T lymphocytes: the extent of gamma interferon modulation determines the outcome of infection.Interferon Gamma +874T/A Polymorphism Increases the Risk of Hepatitis Virus-Related Diseases: Evidence from a Meta-Analysis Reduced transplant arteriosclerosis in murine cardiac allografts placed in interferon-gamma knockout recipients.Indigenous microbiota and Leishmaniasis.

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P2860

CD4+ effector cells default to the Th2 pathway in interferon gamma-deficient mice infected with Leishmania major

http://www.wikidata.org/entity/Q36362926

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1994 nî lūn-bûn

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1994年の論文

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1994年学术文章

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1994年学术文章

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1994年学术文章

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1994年学术文章

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1994年学术文章

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1994年學術文章

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1994年學術文章

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1994年學術文章

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CD4+ effector cells default to ...... infected with Leishmania major

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CD4+ effector cells default to ...... infected with Leishmania major

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CD4+ effector cells default to ...... infected with Leishmania major

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CD4+ effector cells default to ...... infected with Leishmania major

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CD4+ effector cells default to ...... infected with Leishmania major

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P1433

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P1433

Journal of Experimental Medicine

P1476

CD4+ effector cells default to ...... infected with Leishmania major

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P2093

D K Dalton

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R M Locksley

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S L Reiner

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S Zheng

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Z E Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Production of natural killer cell stimulatory factor (interleukin 12) by peripheral blood mononuclear cells

Multiple defects of immune cell function in mice with disrupted interferon-gamma genes

TH1 and TH2 cells: different patterns of lymphokine secretion lead to different functional properties

Development of TH1 CD4+ T cells through IL-12 produced by Listeria-induced macrophages

The presence of interleukin 4 during in vitro priming determines the lymphokine-producing potential of CD4+ T cells from T cell receptor transgenic mice.

Cure of murine leishmaniasis with anti-interleukin 4 monoclonal antibody. Evidence for a T cell-dependent, interferon gamma-independent mechanism.

Generation of interleukin 4 (IL-4)-producing cells in vivo and in vitro: IL-2 and IL-4 are required for in vitro generation of IL-4-producing cells.

Natural killer cells are a source of interferon gamma that drives differentiation of CD4+ T cell subsets and induces early resistance to Leishmania major in mice

Mice that lack the interferon-gamma receptor have profoundly altered responses to infection with Bacillus Calmette-Guérin and subsequent challenge with lipopolysaccharide.

Interleukin 4 suppresses interleukin 2 and interferon gamma production by naive T cells stimulated by accessory cell-dependent receptor engagement

P304

1367-1371

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scholarly article

P356

10.1084/JEM.179.4.1367

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P433

4

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P478

179

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1994-04-01T00:00:00Z

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15291558

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P698

7908325

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P932

2191434

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