Blocking chemokine responsive to gamma-2/interferon (IFN)-gamma inducible protein and monokine induced by IFN-gamma activity in vivo reduces the pathogenetic but not the antiviral potential of hepatitis B virus-specific cytotoxic T lymphocytes.
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Platelets mediate cytotoxic T lymphocyte-induced liver damage.HBV pathogenesis in animal models: recent advances on the role of plateletsImmune-mediated Liver Injury in Hepatitis B Virus InfectionMouse models of hepatitis B virus infection comprising host-virus immunologic interactionsHepatitis B: future curative strategiesIntrahepatic Transcriptional Signature Associated with Response to Interferon-α Treatment in the Woodchuck Model of Chronic Hepatitis BKupffer cells hasten resolution of liver immunopathology in mouse models of viral hepatitisStealth and cunning: hepatitis B and hepatitis C viruses.Plasma chemokine levels correlate with the outcome of antiviral therapy in patients with hepatitis CThe L60V Variation in Hepatitis B Virus Core Protein Elicits New Epitope-Specific Cytotoxic T Lymphocytes and Enhances Viral ReplicationAntiplatelet drug therapy moderates immune-mediated liver disease and inhibits viral clearance in mice infected with a replication-deficient adenovirusImmunology of hepatitis B virus and hepatitis C virus infectionKIR and HLA loci are associated with hepatocellular carcinoma development in patients with hepatitis B virus infection: a case-control studyDifferent antiviral effects of IFNα subtypes in a mouse model of HBV infection.Genomic analysis of the host response to hepatitis B virus infection.Hepatitis B virus protein X-induced expression of the CXC chemokine IP-10 is mediated through activation of NF-kappaB and increases migration of leukocytes.The Role of Innate Lymphoid Cells in Immune-Mediated Liver Diseases.Expression profiles of circulating cytokines, chemokines and immune cells in patients with hepatitis B virus infectionToll-like receptor 8 agonist and bacteria trigger potent activation of innate immune cells in human liver.Mutations to bid cleavage sites protect hepatocytes from apoptosis after ischemia/reperfusion injuryLicensing virus-specific T cells to secrete the neutrophil attracting chemokine CXCL-8 during hepatitis B virus infectionGene expression during the priming phase of liver regeneration after partial hepatectomy in mice.Depletion of neutrophils blocks the recruitment of antigen-nonspecific cells into the liver without affecting the antiviral activity of hepatitis B virus-specific cytotoxic T lymphocytesImmune control and failure in HCV infection--tipping the balance.Polyfunctional type-1, -2, and -17 CD8⁺ T cell responses to apoptotic self-antigens correlate with the chronic evolution of hepatitis C virus infection.Searching for interferon-induced genes that inhibit hepatitis B virus replication in transgenic mouse hepatocytes.Lymphocyte deficiency limits Epstein-Barr virus latent membrane protein 1 induced chronic inflammation and carcinogenic pathology in vivoActive lytic infection of human primary tonsillar B cells by KSHV and its noncytolytic control by activated CD4+ T cells.Immune response and tolerance during chronic hepatitis B virus infection.Regulation of hepatocyte fate by interferon-γ.CXCL9 associated with sustained virological response in chronic hepatitis B patients receiving peginterferon alfa-2a therapy: a pilot studyHCV immunology--death and the maiden T cell.The Fgl2/fibroleukin prothrombinase contributes to immunologically mediated thrombosis in experimental and human viral hepatitisExpression and function of chemokines during viral infections: from molecular mechanisms to in vivo function.A proinflammatory role for interleukin-22 in the immune response to hepatitis B virus.Metabolic regulation of hepatitis B immunopathology by myeloid-derived suppressor cells.The level of viral antigen presented by hepatocytes influences CD8 T-cell function.Pathogenesis of chronic viral hepatitis: differential roles of T cells and NK cellsEmerging Role of Interleukin 22 in Hepatitis B Virus Infection: a Double-edged Sword.Tumor necrosis factor activates a conserved innate antiviral response to hepatitis B virus that destabilizes nucleocapsids and reduces nuclear viral DNA.
P2860
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P2860
Blocking chemokine responsive to gamma-2/interferon (IFN)-gamma inducible protein and monokine induced by IFN-gamma activity in vivo reduces the pathogenetic but not the antiviral potential of hepatitis B virus-specific cytotoxic T lymphocytes.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Blocking chemokine responsive ...... cific cytotoxic T lymphocytes.
@ast
Blocking chemokine responsive ...... cific cytotoxic T lymphocytes.
@en
type
label
Blocking chemokine responsive ...... cific cytotoxic T lymphocytes.
@ast
Blocking chemokine responsive ...... cific cytotoxic T lymphocytes.
@en
prefLabel
Blocking chemokine responsive ...... cific cytotoxic T lymphocytes.
@ast
Blocking chemokine responsive ...... cific cytotoxic T lymphocytes.
@en
P2093
P2860
P921
P356
P1476
Blocking chemokine responsive ...... ecific cytotoxic T lymphocytes
@en
P2093
P2860
P304
P356
10.1084/JEM.194.12.1755
P407
P577
2001-12-01T00:00:00Z