By altering ocular immune privilege, bone marrow-derived cells pathogenically contribute to DBA/2J pigmentary glaucoma - Wikidata - awgldk - TriplyDB

By altering ocular immune privilege, bone marrow-derived cells pathogenically contribute to DBA/2J pigmentary glaucoma

By altering ocular immune privilege, bone marrow-derived cells pathogenically contribute to DBA/2J pigmentary glaucoma

http://www.wikidata.org/entity/Q36370392

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The Role of the IL-20 Subfamily in Glaucoma Neurodegeneration and Vision Loss after Mild Blunt Trauma in the C57Bl/6 and DBA/2J Mouse Early microglia activation in a mouse model of chronic glaucoma.Genetic context determines susceptibility to intraocular pressure elevation in a mouse pigmentary glaucoma Progressive ganglion cell loss and optic nerve degeneration in DBA/2J mice is variable and asymmetric Absence of glaucoma in DBA/2J mice homozygous for wild-type versions of Gpnmb and Tyrp1 Inducible nitric oxide synthase, Nos2, does not mediate optic neuropathy and retinopathy in the DBA/2J glaucoma model.Characterization of intraocular immunopathology following intracameral inoculation with alloantigen.Bioinformatic and statistical analysis of the optic nerve head in a primate model of ocular hypertension.GDF15 is elevated in mice following retinal ganglion cell death and in glaucoma patients High-dose radiation with bone marrow transfer prevents neurodegeneration in an inherited glaucoma.The melanoma-associated transmembrane glycoprotein Gpnmb controls trafficking of cellular debris for degradation and is essential for tissue repair Microarray analysis of iris gene expression in mice with mutations influencing pigmentation.Mouse models of retinal ganglion cell death and glaucoma.Exacerbation of blast-induced ocular trauma by an immune response.Determining immune components necessary for progression of pigment dispersing disease to glaucoma in DBA/2J mice Genetic dissection of the Gpnmb network in the eye Neurodegeneration severity can be predicted from early microglia alterations monitored in vivo in a mouse model of chronic glaucoma.Glycoprotein Nonmetastatic Melanoma B (Gpnmb)-Positive Macrophages Contribute to the Balance between Fibrosis and Fibrolysis during the Repair of Acute Liver Injury in Mice.Using genetic mouse models to gain insight into glaucoma: Past results and future possibilities.A vision of cell death: Fas ligand and immune privilege 10 years later.Longitudinal evaluation of retinal ganglion cell function and IOP in the DBA/2J mouse model of glaucoma.Loss of Fractalkine Signaling Exacerbates Axon Transport Dysfunction in a Chronic Model of Glaucoma.Molecular genetics in glaucoma.Virus-mediated EpoR76E gene therapy preserves vision in a glaucoma model by modulating neuroinflammation and decreasing oxidative stress.New insights into mononuclear phagocyte biology from the visual system.Virus-mediated EpoR76E Therapy Slows Optic Nerve Axonopathy in Experimental Glaucoma.Early Involvement of Immune/Inflammatory Response Genes in Retinal Degeneration in DBA/2J Mice.IOP-dependent retinal ganglion cell dysfunction in glaucomatous DBA/2J mice.Eye-Directed Overpressure Airwave-Induced Trauma Causes Lasting Damage to the Anterior and Posterior Globe: A Model for Testing Cell-Based Therapies.Aqueous humor suppression of dendritic cell function helps maintain immune regulation in the eye during human uveitis Involvement of inflammation, degradation, and apoptosis in a mouse model of glaucoma.Norrin protects optic nerve axons from degeneration in a mouse model of glaucoma.Loss of GPNMB Causes Autosomal-Recessive Amyloidosis Cutis Dyschromica in Humans.Neuroinflammation: Microglia and T Cells Get Ready to Tango.Modulation of the immune system for the treatment of glaucoma.Caspases in retinal ganglion cell death and axon regeneration.Molecular Genetics of Pigment Dispersion Syndrome and Pigmentary Glaucoma: New Insights into Mechanisms.

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P2860

By altering ocular immune privilege, bone marrow-derived cells pathogenically contribute to DBA/2J pigmentary glaucoma

http://www.wikidata.org/entity/Q36370392

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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2003年學術文章

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name

By altering ocular immune priv ...... to DBA/2J pigmentary glaucoma

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By altering ocular immune priv ...... to DBA/2J pigmentary glaucoma

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type

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By altering ocular immune priv ...... to DBA/2J pigmentary glaucoma

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By altering ocular immune priv ...... to DBA/2J pigmentary glaucoma

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By altering ocular immune priv ...... to DBA/2J pigmentary glaucoma

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By altering ocular immune priv ...... to DBA/2J pigmentary glaucoma

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By altering ocular immune priv ...... to DBA/2J pigmentary glaucoma

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P2093

Bruce R Ksander

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David V Serreze

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J Wayne Streilein

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Jun-Song Mo

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Meredith Gregory

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Olga V Savinova

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Richard S Smith

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Simon W M John

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P2860

Dendritic cells and the control of immunity

Identification of the genes differentially expressed in human dendritic cell subsets by cDNA subtraction and microarray analysis

Molecular cloning of a dendritic cell-associated transmembrane protein, DC-HIL, that promotes RGD-dependent adhesion of endothelial cells through recognition of heparan sulfate proteoglycans

Mouse genetics: a tool to help unlock the mechanisms of glaucoma.

Human pigmentation genes: identification, structure and consequences of polymorphic variation.

Autoimmunity and the immunotherapy of cancer: targeting the "self" to destroy the "other".

Characterization of a new melanocyte-specific gene (QNR-71) expressed in v-myc-transformed quail neuroretina.

Induction of experimental autoimmune anterior uveitis by a self-antigen: melanin complex without adjuvant.

Regulation of the proinflammatory effects of Fas ligand (CD95L).

Augmentation of intraocular inflammation by melanin.

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1335-1344

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10.1084/JEM.20022041

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10

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197

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Michael Anderson

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2003-05-01T00:00:00Z

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10752427

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12756269

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2193785

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