Dendritic cells initiate immune control of epstein-barr virus transformation of B lymphocytes in vitro.
about
Priming of protective T cell responses against virus-induced tumors in mice with human immune system componentsInterference with the Autophagic Process as a Viral Strategy to Escape from the Immune Control: Lesson from Gamma HerpesvirusesFunctional delivery of viral miRNAs via exosomesTonsilar NK cells restrict B cell transformation by the Epstein-Barr virus via IFN-gamma.Dendritic cells during Epstein Barr virus infection.mRNA Structural constraints on EBNA1 synthesis impact on in vivo antigen presentation and early priming of CD8+ T cells.Transfer of antigen from human B cells to dendritic cells.Infection of dendritic cells by a gamma2-herpesvirus induces functional modulationCytolytic T lymphocytes from HLA-B8+ donors frequently recognize the Hodgkin's lymphoma associated latent membrane protein 2 of Epstein Barr virus.Epstein-barr virus nuclear antigen 1: from immunologically invisible to a promising T cell target.EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors.Virus-specific CD4+ T cells: ready for direct attack.Control of Epstein-Barr virus infection in vitro by T helper cells specific for virion glycoproteins.Autophagy and antigen presentation.Robust T-cell stimulation by Epstein-Barr virus-transformed B cells after antigen targeting to DEC-205.Targeting the nuclear antigen 1 of Epstein-Barr virus to the human endocytic receptor DEC-205 stimulates protective T-cell responses.Immune escape by Epstein-Barr virus associated malignancies.Editing antigen presentation: antigen transfer between human B lymphocytes and macrophages mediated by class A scavenger receptors.Critical role of EBNA1-specific CD4+ T cells in the control of mouse Burkitt lymphoma in vivo.Innate immune responses against Epstein Barr virus infectionHerpesviruses placating the unwilling host: manipulation of the MHC class II antigen presentation pathway.Immune control of oncogenic γ-herpesviruses.Programmed death 1 is highly expressed on CD8+CD57+ T cells in patients with stable multiple sclerosis and inhibits their cytotoxic response to EBV.Cytomegalovirus-seropositive children show inhibition of in vitro EBV infection that is associated with CD8+CD57+ T cell enrichment and IFN-γ.The CD8+ T-cell response to an Epstein-Barr virus-related gammaherpesvirus infecting rhesus macaques provides evidence for immune evasion by the EBNA-1 homologue.Successful in vitro priming of EBV-specific CD8+ T cells endowed with strong cytotoxic function from T cells of EBV-seronegative children.
P2860
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P2860
Dendritic cells initiate immune control of epstein-barr virus transformation of B lymphocytes in vitro.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
Dendritic cells initiate immun ...... ion of B lymphocytes in vitro.
@ast
Dendritic cells initiate immun ...... ion of B lymphocytes in vitro.
@en
type
label
Dendritic cells initiate immun ...... ion of B lymphocytes in vitro.
@ast
Dendritic cells initiate immun ...... ion of B lymphocytes in vitro.
@en
prefLabel
Dendritic cells initiate immun ...... ion of B lymphocytes in vitro.
@ast
Dendritic cells initiate immun ...... ion of B lymphocytes in vitro.
@en
P2093
P2860
P356
P1476
Dendritic cells initiate immun ...... ion of B lymphocytes in vitro.
@en
P2093
Casper Paludan
Christian Münz
Kara Bickham
Kiera Goodman
Ming Li Tsang
Ralph M Steinman
Sarah Nikiforow
P2860
P304
P356
10.1084/JEM.20030646
P407
P577
2003-12-01T00:00:00Z