Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein.
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Role of S-nitrosylation in apoptosis resistance and carcinogenesisThe Fas death signaling pathway connecting reactive oxygen species generation and FLICE inhibitory protein down-regulationNitric oxide negatively regulates Fas CD95-induced apoptosis through inhibition of ubiquitin-proteasome-mediated degradation of FLICE inhibitory proteinExpression of SCC-S2, an antiapoptotic molecule, correlates with enhanced proliferation and tumorigenicity of MDA-MB 435 cellsFLICE-inhibitory proteins: regulators of death receptor-mediated apoptosisNatural selection of tumor variants in the generation of "tumor escape" phenotypesCellular FLICE-inhibitory protein splice variants inhibit different steps of caspase-8 activation at the CD95 death-inducing signaling complexDecitabine and vorinostat cooperate to sensitize colon carcinoma cells to Fas ligand-induced apoptosis in vitro and tumor suppression in vivoMatrilysin [MMP-7] expression selects for cells with reduced sensitivity to apoptosisTRAIL-Based High Throughput Screening Reveals a Link between TRAIL-Mediated Apoptosis and Glutathione Reductase, a Key Component of Oxidative Stress ResponseRegulation of the cell cycle and apoptosis by the oncogenes of adenovirusRheumatoid arthritis synovial macrophages express the Fas-associated death domain-like interleukin-1beta-converting enzyme-inhibitory protein and are refractory to Fas-mediated apoptosis.Interaction of dietary fatty acids with tumour necrosis factor family cytokines during colon inflammation and cancer.LUCA-15-encoded sequence variants regulate CD95-mediated apoptosis.Blockade of the granzyme B/perforin pathway through overexpression of the serine protease inhibitor PI-9/SPI-6 constitutes a mechanism for immune escape by tumors.CD40 stimulation leads to effective therapy of CD40(-) tumors through induction of strong systemic cytotoxic T lymphocyte immunity.Inducible resistance to Fas-mediated apoptosis in B cells.Inhibition of the intrinsic but not the extrinsic apoptosis pathway accelerates and drives MYC-driven tumorigenesis towards acute myeloid leukemia.Apoptosis and its clinical impact.The caspase 8 inhibitor c-FLIP(L) modulates T-cell receptor-induced proliferation but not activation-induced cell death of lymphocytesIonizing radiation as a response-enhancing agent for CD95-mediated apoptosis.FLIP: a novel regulator of macrophage differentiation and granulocyte homeostasisE1A sensitizes cells to tumor necrosis factor alpha by downregulating c-FLIP S.The role of cellular FLICE inhibitory protein (c-FLIP) in the pathogenesis and treatment of cancer.Role of TNF family ligands in antitumor activity of natural killer cells.Isolation of immunoresistant human glioma cell clones after selection with alloreactive cytotoxic T lymphocytes: cytogenetic and molecular cytogenetic characterization.Mechanisms of malignant glioma immune resistance and sources of immunosuppressionModulating T-cell immunity to tumours: new strategies for monitoring T-cell responses.Tumor escape from killing: role of killer inhibitory receptors and acquisition of tumor resistance to cell death.Induction of apoptosis in cancer: new therapeutic opportunities.Cellular FLICE-inhibitory protein: an attractive therapeutic target?The use of genetic microarray analysis to classify and predict prognosis in haematological malignancies.Impaired CD95 expression predisposes for recurrence in curatively resected colon carcinoma: clinical evidence for immunoselection and CD95L mediated control of minimal residual disease.Immunosuppressive tumor microenvironment in cervical cancer patientsThe role of c-FLIP splice variants in urothelial tumours.The androgen receptor directly targets the cellular Fas/FasL-associated death domain protein-like inhibitory protein gene to promote the androgen-independent growth of prostate cancer cells.Constitutive expression of c-FLIP in Hodgkin and Reed-Sternberg cells.Cellular and functional characterization of immunoresistant human glioma cell clones selected with alloreactive cytotoxic T lymphocytes reveals their up-regulated synthesis of biologically active TGF-beta.Mechanisms of resistance to TRAIL-induced apoptosis in cancer.Modulation of apoptosis as a target for liver disease.
P2860
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P2860
Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein.
@ast
Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein.
@en
type
label
Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein.
@ast
Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein.
@en
prefLabel
Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein.
@ast
Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein.
@en
P2093
P2860
P356
P1476
Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein.
@en
P2093
C J Melief
J P Medema
R Offringa
T van Hall
P2860
P304
P356
10.1084/JEM.190.7.1033
P407
P577
1999-10-01T00:00:00Z