Ovalbumin sensitization changes the inflammatory response to subsequent parainfluenza infection. Eosinophils mediate airway hyperresponsiveness, m(2) muscarinic receptor dysfunction, and antiviral effects

Ovalbumin sensitization changes the inflammatory response to subsequent parainfluenza infection. Eosinophils mediate airway hyperresponsiveness, m(2) muscarinic receptor dysfunction, and antiviral effects

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Antigen sensitization influences organophosphorus pesticide-induced airway hyperreactivity The influence of sensitization on mechanisms of organophosphorus pesticide-induced airway hyperreactivity Asthma increases susceptibility to heterologous but not homologous secondary influenza Eosinophils Promote Antiviral Immunity in Mice Infected with Influenza A Virus IFN-γ increases susceptibility to influenza A infection through suppression of group II innate lymphoid cells.Metabolomic biomarkers in a model of asthma exacerbation: urine nuclear magnetic resonance.Eosinophil survival and apoptosis in health and disease.MDA5 and TLR3 initiate pro-inflammatory signaling pathways leading to rhinovirus-induced airways inflammation and hyperresponsiveness.Muscarinic receptor antagonists: effects on pulmonary function.Neuropeptide-induced chemotaxis of eosinophils in pulmonary diseases.Asthma, asthma medication and autonomic nervous system dysfunction.Neuronal eotaxin and the effects of CCR3 antagonist on airway hyperreactivity and M2 receptor dysfunction.Antiviral potential of chemokines.Animal models of virus-induced chronic airway disease The airway epithelium: soldier in the fight against respiratory viruses Eosinophils: multifaceted biological properties and roles in health and disease The expanding role(s) of eosinophils in health and disease.Eosinophils: offenders or general bystanders in allergic airway disease and pulmonary immunity?Airway neural plasticity: the nerves they are a-changin'.Eosinophils: changing perspectives in health and disease.Host defense function of the airway epithelium in health and disease: clinical background.Rhinovirus infection of allergen-sensitized and -challenged mice induces eotaxin release from functionally polarized macrophages.Anti-interleukin-5 monoclonal antibodies: preclinical and clinical evidence in asthma models.Review article: The eosinophil as a therapeutic target in gastrointestinal disease.The clinical significance of eosinophils in the amniotic fluid in preterm labor The Pneumonia Virus of Mice (PVM) model of acute respiratory infection.Interaction between allergy and innate immunity: model for eosinophil regulation of epithelial cell interferon expression Targeting eosinophils in asthma Respiratory viruses and eosinophils: exploring the connections.Retinoic acid prevents virus-induced airway hyperreactivity and M2 receptor dysfunction via anti-inflammatory and antiviral effects Pulmonary eosinophils and their role in immunopathologic responses to formalin-inactivated pneumonia virus of mice.Pneumoviruses infect eosinophils and elicit MyD88-dependent release of chemoattractant cytokines and interleukin-6.Biology of the eosinophil Eosinophils and their interactions with respiratory virus pathogens.Activated mouse eosinophils protect against lethal respiratory virus infection.Allergy, asthma, and inflammation: which inflammatory cell type is more important?The role of eosinophils in non-parasitic infections.Antimicrobial activity of human eosinophil granule proteins: involvement in host defence against pathogens.Eosinophils: multifunctional and distinctive properties Fc gamma receptors in respiratory syncytial virus infections: implications for innate immunity.

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P2860

Ovalbumin sensitization changes the inflammatory response to subsequent parainfluenza infection. Eosinophils mediate airway hyperresponsiveness, m(2) muscarinic receptor dysfunction, and antiviral effects

Item

http://www.wikidata.org/entity/Q36375538

description

1999 nî lūn-bûn

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1999年の論文

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1999年学术文章

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1999年学术文章

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1999年学术文章

@zh-hans

1999年学术文章

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1999年学术文章

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1999年學術文章

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1999年學術文章

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1999年學術文章

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name

Ovalbumin sensitization change ...... unction, and antiviral effects

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Ovalbumin sensitization change ...... unction, and antiviral effects

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Ovalbumin sensitization change ...... unction, and antiviral effects

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Ovalbumin sensitization change ...... unction, and antiviral effects

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Ovalbumin sensitization change ...... unction, and antiviral effects

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Ovalbumin sensitization change ...... unction, and antiviral effects

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P1476

Ovalbumin sensitization change ...... unction, and antiviral effects

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P2093

A D Fryer

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B L Yost

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D B Jacoby

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D J Adamko

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G J Gleich

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P2860

Biochemical and functional similarities between human eosinophil-derived neurotoxin and eosinophil cationic protein: homology with ribonuclease

Rhinovirus and respiratory syncytial virus in wheezing children requiring emergency care. IgE and eosinophil analyses.

Influenza virus-specific CD4+ T helper type 2 T lymphocytes do not promote recovery from experimental virus infection.

Cell-specific expression of RANTES, MCP-1, and MIP-1alpha by lower airway epithelial cells and eosinophils infected with respiratory syncytial virus.

Human eosinophil major basic protein is an endogenous allosteric antagonist at the inhibitory muscarinic M2 receptor.

Nitric oxide inhibits rhinovirus-induced cytokine production and viral replication in a human respiratory epithelial cell line.

Increased nitric oxide in exhaled air of asthmatic patients.

T-cell-replacing factor (interleukin 5) induces expression of interleukin 2 receptors on murine splenic B cells.

Interleukin 5, a T-cell-derived B-cell differentiation factor also induces cytotoxic T lymphocytes

Eosinophil cationic protein/RNase 3 is another RNase A-family ribonuclease with direct antiviral activity.

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1465-1478

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scholarly article

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10.1084/JEM.190.10.1465

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English

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190

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1999-11-01T00:00:00Z

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10562321

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P921

inflammation

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2195693

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Ovalbumin sensitization changes the inflammatory response to subsequent parainfluenza infection. Eosinophils mediate airway hyperresponsiveness, m(2) muscarinic receptor dysfunction, and antiviral effects

about

P2860

P2860

Ovalbumin sensitization changes the inflammatory response to subsequent parainfluenza infection. Eosinophils mediate airway hyperresponsiveness, m(2) muscarinic receptor dysfunction, and antiviral effects

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