Macrophage p38 mitogen-activated protein kinase activity regulates invariant natural killer T-cell responses during Borrelia burgdorferi infection
about
CD4+ T cells promote antibody production but not sustained affinity maturation during Borrelia burgdorferi infection.Ikaros mediates the DNA methylation-independent silencing of MCJ/DNAJC15 gene expression in macrophages.The early dissemination defect attributed to disruption of decorin-binding proteins is abolished in chronic murine Lyme borreliosis.The MEK/ERK pathway is the primary conduit for Borrelia burgdorferi-induced inflammation and P53-mediated apoptosis in oligodendrocytes.Infection with the Lyme disease pathogen suppresses innate immunity in mice with diet-induced obesity.A multi-omic analysis reveals the regulatory role of CD180 during the response of macrophages to Borrelia burgdorferi.
P2860
Macrophage p38 mitogen-activated protein kinase activity regulates invariant natural killer T-cell responses during Borrelia burgdorferi infection
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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name
Macrophage p38 mitogen-activat ...... Borrelia burgdorferi infection
@ast
Macrophage p38 mitogen-activat ...... Borrelia burgdorferi infection
@en
type
label
Macrophage p38 mitogen-activat ...... Borrelia burgdorferi infection
@ast
Macrophage p38 mitogen-activat ...... Borrelia burgdorferi infection
@en
prefLabel
Macrophage p38 mitogen-activat ...... Borrelia burgdorferi infection
@ast
Macrophage p38 mitogen-activat ...... Borrelia burgdorferi infection
@en
P2093
P2860
P921
P356
P1476
Macrophage p38 mitogen-activat ...... Borrelia burgdorferi infection
@en
P2093
Chris M Olson
Dietrich Conze
Kelly Hawley
Mercedes Rincón
Michael N Hedrick
Nicolás Navasa
Tonya C Bates
P2860
P304
P356
10.1093/INFDIS/JIS332
P407
P50
P577
2012-05-02T00:00:00Z