Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.
about
Periodontal disease, tooth loss, and cancer risk in male health professionals: a prospective cohort studyA review of the relationship between tooth loss, periodontal disease, and cancerRoles of Commensal Microbiota in Pancreas Homeostasis and Pancreatic PathologiesLinks demystified: Periodontitis and cancerInflammatory mechanisms linking periodontal diseases to cardiovascular diseasesPeriodontal Disease and Tooth Loss as Risks for Cancer: A Systematic Review of the LiteratureAnchoring and length regulation of Porphyromonas gingivalis Mfa1 fimbriae by the downstream gene product Mfa2.Active invasion of oral and aortic tissues by Porphyromonas gingivalis in mice causally links periodontitis and atherosclerosis.High-throughput sequencing reveals key genes and immune homeostatic pathways activated in myeloid dendritic cells by Porphyromonas gingivalis 381 and its fimbrial mutants.Hepatocyte-specific IKKβ expression aggravates atherosclerosis development in APOE*3-Leiden miceLipoproteins and lipoprotein metabolism in periodontal disease.Evaluation of the effect of andrographolide on atherosclerotic rabbits induced by Porphyromonas gingivalisThe oxidative stress product carboxyethylpyrrole potentiates TLR2/TLR1 inflammatory signaling in macrophagesReview: Pathogen-induced inflammation at sites distant from oral infection: bacterial persistence and induction of cell-specific innate immune inflammatory pathways.Porphyromonas gingivalis peptidoglycans induce excessive activation of the innate immune system in silkworm larvae.Immune dysregulation mediated by the oral microbiome: potential link to chronic inflammation and atherosclerosis.The sinR ortholog PGN_0088 encodes a transcriptional regulator that inhibits polysaccharide synthesis in Porphyromonas gingivalis ATCC 33277 biofilmsAnti-P. gingivalis response correlates with atherosclerosis.The atherogenic bacterium Porphyromonas gingivalis evades circulating phagocytes by adhering to erythrocytes.Impact of monocytic cells on recovery of uncultivable bacteria from atherosclerotic lesions.Porphyromonas gingivalis evasion of autophagy and intracellular killing by human myeloid dendritic cells involves DC-SIGN-TLR2 crosstalk.Periodontal disease and pregnancy outcomes: time to move on?Oxidized LDLs inhibit TLR-induced IL-10 production by monocytes: a new aspect of pathogen-accelerated atherosclerosis.Protective role for TLR4 signaling in atherosclerosis progression as revealed by infection with a common oral pathogen.Microbial carriage state of peripheral blood dendritic cells (DCs) in chronic periodontitis influences DC differentiation, atherogenic potential.Complement-targeted therapeutics in periodontitisInsights into the virulence of oral biofilms: discoveries from proteomics.Nasal immunization with Porphyromonas gingivalis outer membrane protein decreases P. gingivalis-induced atherosclerosis and inflammation in spontaneously hyperlipidemic miceAcute toxicity and the effect of andrographolide on Porphyromonas gingivalis-induced hyperlipidemia in rats.Variations of oral microbiota are associated with pancreatic diseases including pancreatic cancerSubversion of innate immunity by periodontopathic bacteria via exploitation of complement receptor-3Induction of distinct TLR2-mediated proinflammatory and proadhesive signaling pathways in response to Porphyromonas gingivalis fimbriae.Role of human CD36 in bacterial recognition, phagocytosis, and pathogen-induced JNK-mediated signaling.Coffee, glucose homeostasis, and insulin resistance: physiological mechanisms and mediators.Translational mini-review series on immunology of vascular disease: inflammation, infections and Toll-like receptors in cardiovascular disease.Secondary lymphoid organ homing phenotype of human myeloid dendritic cells disrupted by an intracellular oral pathogen.Disruption of immune regulation by microbial pathogens and resulting chronic inflammation.Apolipoprotein A-I inhibits LPS-induced atherosclerosis in ApoE(-/-) mice possibly via activated STAT3-mediated upregulation of tristetraprolin.Complement and periodontitis.Signaling events in pathogen-induced macrophage foam cell formation.
P2860
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P2860
Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.
@ast
Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.
@en
type
label
Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.
@ast
Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.
@en
prefLabel
Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.
@ast
Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.
@en
P2093
P2860
P1476
Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis
@en
P2093
P2860
P304
P356
10.1177/154405910608500202
P407
P577
2006-02-01T00:00:00Z