Extracellular chromatin is an important mediator of ischemic stroke in mice. - Wikidata - awgldk - TriplyDB

Extracellular chromatin is an important mediator of ischemic stroke in mice.

Extracellular chromatin is an important mediator of ischemic stroke in mice.

http://www.wikidata.org/entity/Q36387981

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Next-generation antithrombotics in ischemic stroke: preclinical perspective on 'bleeding-free antithrombosis'Sepsis and ARDS: The Dark Side of Histones Damage-associated molecular pattern-activated neutrophil extracellular trap exacerbates sterile inflammatory liver injury.Elevated homocysteine levels in type 2 diabetes induce constitutive neutrophil extracellular traps.Inter-α inhibitor protein and its associated glycosaminoglycans protect against histone-induced injury AN INVESTIGATION OF EXTRACELLULAR HISTONES IN PIG-TO-BABOON ORGAN XENOTRANSPLANTATION.Thrombosis: tangled up in NETs.PAD4-deficiency does not affect bacteremia in polymicrobial sepsis and ameliorates endotoxemic shock Arterial thrombosis is accelerated in mice deficient in histidine-rich glycoprotein Release and activity of histone in diseases.Expression of functional tissue factor by neutrophil extracellular traps in culprit artery of acute myocardial infarction.Mitochondrial Damage-Associated Molecular Patterns (MTDs) Are Released during Hepatic Ischemia Reperfusion and Induce Inflammatory Responses.Effect of DNase I treatment and neutrophil depletion on acute limb ischemia-reperfusion injury in mice.Extracellular DNA traps are associated with the pathogenesis of TRALI in humans and mice.NETosis promotes cancer-associated arterial microthrombosis presenting as ischemic stroke with troponin elevation.Epigenetic mechanisms in cerebral ischemia.Dynamic change of neutrophil to lymphocyte ratio and hemorrhagic transformation after thrombolysis in stroke.Circulating histones are major mediators of systemic inflammation and cellular injury in patients with acute liver failure.Extracellular DNA traps released by acute promyelocytic leukemia cells through autophagy VWF-mediated leukocyte recruitment with chromatin decondensation by PAD4 increases myocardial ischemia/reperfusion injury in mice.Thrombosis, Neuroinflammation, and Poststroke Infection: The Multifaceted Role of Neutrophils in Stroke.New insights into modulation of thrombin formation.Neutrophil extracellular traps: a walk on the wild side of exercise immunology.Platelets and Their Interactions with Other Immune Cells The role of extracellular histone in organ injury.Validation of an enzyme-linked immunosorbent assay for the quantification of citrullinated histone H3 as a marker for neutrophil extracellular traps in human plasma.Extracellular histones, cell-free DNA, or nucleosomes: differences in immunostimulation.HMGB1 and Histones Play a Significant Role in Inducing Systemic Inflammation and Multiple Organ Dysfunctions in Severe Acute Pancreatitis.HMGB1 and Extracellular Histones Significantly Contribute to Systemic Inflammation and Multiple Organ Failure in Acute Liver Failure Antihistone Properties of C1 Esterase Inhibitor Protect against Lung Injury.Hepatic radiofrequency ablation causes an increase of circulating histones in patients with hepatocellular carcinoma.Cell-Free DNA and DNase Activity in Dogs with Immune-Mediated Hemolytic Anemia.Extracellular RNA facilitates hypoxia-induced leukocyte adhesion and infiltration in the lung through TLR3-IFN-γ-STAT1 signaling pathway.Release of neutrophil extracellular traps by neutrophils stimulated with antiphospholipid antibodies: a newly identified mechanism of thrombosis in the antiphospholipid syndrome.Peptidylarginine deiminase 4 promotes age-related organ fibrosis Dying cells and extracellular histones in AKI: beyond a NET effect?Circulating histones exacerbate inflammation in mice with acute liver failure.Therapeutic targeting of extracellular DNA improves the outcome of intestinal ischemic reperfusion injury in neonatal rats.Cell-free DNA as a biomarker in stroke: Current status, problems and perspectives.Neutrophil extracellular traps in ischemic stroke thrombi.

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P2860

Extracellular chromatin is an important mediator of ischemic stroke in mice.

http://www.wikidata.org/entity/Q36387981

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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name

Extracellular chromatin is an important mediator of ischemic stroke in mice.

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Extracellular chromatin is an important mediator of ischemic stroke in mice.

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type

label

Extracellular chromatin is an important mediator of ischemic stroke in mice.

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Extracellular chromatin is an important mediator of ischemic stroke in mice.

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prefLabel

Extracellular chromatin is an important mediator of ischemic stroke in mice.

@ast

Extracellular chromatin is an important mediator of ischemic stroke in mice.

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ATVBAHA.112.250993

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Arteriosclerosis, Thrombosis, and Vascular Biology

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Extracellular chromatin is an important mediator of ischemic stroke in mice.

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P2093

Denisa D Wagner

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Georgette L Suidan

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Marc Monestier

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Simon F De Meyer

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Tobias A Fuchs

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P2860

Efficacy and Safety of Anticoagulant Treatment in Acute Cardioembolic Stroke: A Meta-Analysis of Randomized Controlled Trials

Neutrophil extracellular traps promote deep vein thrombosis in mice

Extracellular DNA traps promote thrombosis.

Platelet polyphosphates are proinflammatory and procoagulant mediators in vivo

Global and regional burden of disease and risk factors, 2001: systematic analysis of population health data

Targeting coagulation factor XII provides protection from pathological thrombosis in cerebral ischemia without interfering with hemostasis

The role of hypoxia and platelets in air travel-related venous thromboembolism.

Histones induce rapid and profound thrombocytopenia in mice

von Willebrand factor: an emerging target in stroke therapy.

Extracellular histones are major mediators of death in sepsis.

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