Transposon mutagenesis identifies genes that transform neural stem cells into glioma-initiating cells.
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Epigenetic pathways and glioblastoma treatmentGlioblastoma Stem Cells Microenvironment: The Paracrine Roles of the Niche in Drug and RadioresistanceThe utility of transposon mutagenesis for cancer studies in the era of genome editing.In vivo models of brain tumors: roles of genetically engineered mouse models in understanding tumor biology and use in preclinical studiesGenetic signature of histiocytic sarcoma revealed by a sleeping beauty transposon genetic screen in miceTransposon mutagenesis identifies genes and cellular processes driving epithelial-mesenchymal transition in hepatocellular carcinoma.Sleeping Beauty mouse models identify candidate genes involved in gliomagenesis.Whole genome analyses of a well-differentiated liposarcoma reveals novel SYT1 and DDR2 rearrangements.Transposon mediated integration of plasmid DNA into the subventricular zone of neonatal mice to generate novel models of glioblastoma.Sequencing methods and datasets to improve functional interpretation of sleeping beauty mutagenesis screens.Ex vivo gene therapy using patient iPSC-derived NSCs reverses pathology in the brain of a homologous mouse modelEstablishment of human iPSC-based models for the study and targeting of glioma initiating cellsMesenchymal differentiation mediated by NF-κB promotes radiation resistance in glioblastoma.Paediatric and adult glioblastoma: multiform (epi)genomic culprits emerge.Cancer gene discovery: exploiting insertional mutagenesis.Brain stem cells as the cell of origin in glioma.Sleeping Beauty transposon insertional mutagenesis based mouse models for cancer gene discoveryRole of MEK partner-1 in cancer stemness through MEK/ERK pathway in cancerous neural stem cells, expressing EGFRviii.Reprogramming strategies for the establishment of novel human cancer models.Role and Therapeutic Targeting of the HGF/MET Pathway in Glioblastoma.Transcriptionally regulated and nontoxic delivery of the hyperactive Sleeping Beauty Transposase.An Efficient In Vitro Transposition Method by a Transcriptionally Regulated Sleeping Beauty System Packaged into an Integration Defective Lentiviral Vector.RNA helicase DHX15 acts as a tumour suppressor in glioma.The La and related RNA-binding proteins (LARPs): structures, functions, and evolving perspectives.Spontaneous development of intratumoral heterogeneity in a transposon-induced mouse model of glioma.
P2860
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P2860
Transposon mutagenesis identifies genes that transform neural stem cells into glioma-initiating cells.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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name
Transposon mutagenesis identif ...... into glioma-initiating cells.
@ast
Transposon mutagenesis identif ...... into glioma-initiating cells.
@en
type
label
Transposon mutagenesis identif ...... into glioma-initiating cells.
@ast
Transposon mutagenesis identif ...... into glioma-initiating cells.
@en
prefLabel
Transposon mutagenesis identif ...... into glioma-initiating cells.
@ast
Transposon mutagenesis identif ...... into glioma-initiating cells.
@en
P2093
P2860
P50
P356
P1476
Transposon mutagenesis identif ...... into glioma-initiating cells.
@en
P2093
Christopher Chin Kuan Yew
David J Adams
Haruna Takeda
Jerrold M Ward
Kazuko Ueno
Masao Nagasaki
Naoki Nariai
Sumiko Watanabe
P2860
P304
E2998-3007
P356
10.1073/PNAS.1215899109
P407
P577
2012-10-08T00:00:00Z