Low dose Leishmania major promotes a transient T helper cell type 2 response that is down-regulated by interferon gamma-producing CD8+ T cells
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Post-Genomics and Vaccine Improvement for LeishmaniaStudy of Leishmania pathogenesis in mice: experimental considerationsCytotoxic T cells mediate pathology and metastasis in cutaneous leishmaniasisPlasma levels of interlukin-4 and Interferon-γ in patients with chronic or healed cutaneous leishmaniasis.CD8+ T cells as a source of IFN-γ production in human cutaneous leishmaniasis.Experimental Validation of Multi-Epitope Peptides Including Promising MHC Class I- and II-Restricted Epitopes of Four Known Leishmania infantum Proteins.Subversion mechanisms by which Leishmania parasites can escape the host immune response: a signaling point of view.Leishmaniasis Vaccine: Where are We Today?Subversion of host cell signalling by the protozoan parasite Leishmania.Antigen requirements for efficient priming of CD8+ T cells by Leishmania major-infected dendritic cells.Regulation of CD8+ T cell responses to infection with parasitic protozoa.IFN-{gamma} produced by CD8 T cells induces T-bet-dependent and -independent class switching in B cells in responses to alum-precipitated protein vaccine.Deception and manipulation: the arms of leishmania, a successful parasiteAn early reduction in Treg cells correlates with enhanced local inflammation in cutaneous leishmaniasis in CCR6-deficient mice.CD8+ T cells are preferentially activated during primary low dose leishmania major infection but are completely dispensable during secondary anti-Leishmania immunityDeficiency of Leishmania phosphoglycans influences the magnitude but does not affect the quality of secondary (memory) anti-Leishmania immunityT cell-derived IL-10 determines leishmaniasis disease outcome and is suppressed by a dendritic cell based vaccineProtective and pathological functions of CD8+ T cells in Leishmania braziliensis infection.Engagement of NKG2D on bystander memory CD8 T cells promotes increased immunopathology following Leishmania major infectionIn silico analysis of six known Leishmania major antigens and in vitro evaluation of specific epitopes eliciting HLA-A2 restricted CD8 T cell response.Immunogenicity evaluation of a rationally designed polytope construct encoding HLA-A*0201 restricted epitopes derived from Leishmania major related proteins in HLA-A2/DR1 transgenic mice: steps toward polytope vaccineEpigenetic control of interferon-gamma expression in CD8 T cells.Induction of protective CD4+ T cell-mediated immunity by a Leishmania peptide delivered in recombinant influenza viruses.The development of effector and memory T cells in cutaneous leishmaniasis: the implications for vaccine development.CD8(+) T cells in leishmania infections: friends or foes?Does T Helper Differentiation Correlate with Resistance or Susceptibility to Infection with L. major? Some Insights From the Murine Model.CD8+ T cell cytotoxicity mediates pathology in the skin by inflammasome activation and IL-1β production.Evaluation of the protection conferred by a naturally attenuated Neospora caninum isolate against congenital and cerebral neosporosis in miceImmunologic memory in cutaneous leishmaniasis.Cutaneous Infection with Leishmania major Mediates Heterologous Protection against Visceral Infection with Leishmania infantumCXCL10 is critical for the generation of protective CD8 T cell response induced by antigen pulsed CpG-ODN activated dendritic cells.Cutaneous Leishmania infection: progress in pathogenesis research and experimental therapy.Functional paradox in host-pathogen interaction dictates the fate of parasites.CD8 cytotoxic T cells in cutaneous leishmaniasis.The central memory CD4+ T cell population generated during Leishmania major infection requires IL-12 to produce IFN-gammaParasite-derived arginase influences secondary anti-Leishmania immunity by regulating programmed cell death-1-mediated CD4+ T cell exhaustion.Mechanisms and consequences of persistence of intracellular pathogens: leishmaniasis as an example.Optimized subunit vaccine protects against experimental leishmaniasis.Complement receptor 3 deficiency influences lesion progression during Leishmania major infection in BALB/c miceHuman classical monocytes control the intracellular stage of Leishmania braziliensis by reactive oxygen species.
P2860
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P2860
Low dose Leishmania major promotes a transient T helper cell type 2 response that is down-regulated by interferon gamma-producing CD8+ T cells
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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name
Low dose Leishmania major prom ...... n gamma-producing CD8+ T cells
@ast
Low dose Leishmania major prom ...... n gamma-producing CD8+ T cells
@en
type
label
Low dose Leishmania major prom ...... n gamma-producing CD8+ T cells
@ast
Low dose Leishmania major prom ...... n gamma-producing CD8+ T cells
@en
prefLabel
Low dose Leishmania major prom ...... n gamma-producing CD8+ T cells
@ast
Low dose Leishmania major prom ...... n gamma-producing CD8+ T cells
@en
P2093
P2860
P356
P1476
Low dose Leishmania major prom ...... n gamma-producing CD8+ T cells
@en
P2093
Jude E Uzonna
Karen L Joyce
Phillip Scott
P2860
P304
P356
10.1084/JEM.20040172
P407
P577
2004-06-01T00:00:00Z