CD1d-dependent activation of NKT cells aggravates atherosclerosis. - Wikidata - awgldk - TriplyDB

CD1d-dependent activation of NKT cells aggravates atherosclerosis.

CD1d-dependent activation of NKT cells aggravates atherosclerosis.

http://www.wikidata.org/entity/Q36399122

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The role of apolipoprotein E in Guillain-Barré syndrome and experimental autoimmune neuritis Immunoregulation of NKT Cells in Systemic Lupus Erythematosus T cells in atherosclerosis Relationships between Th1 or Th2 iNKT cell activity and structures of CD1d-antigen complexes: meta-analysis of CD1d-glycolipids dynamics simulations Apolipoprotein-mediated pathways of lipid antigen presentation Once Upon a Time: The Adaptive Immune Response in Atherosclerosis--a Fairy Tale No More Lymphocyte recruitment into the aortic wall before and during development of atherosclerosis is partially L-selectin dependent.Deficiency of invariant V alpha 14 natural killer T cells decreases atherosclerosis in LDL receptor null mice Macrophage plasticity in experimental atherosclerosis.Innate and adaptive immunity in atherosclerosis.Inflammation in atherosclerosis: from pathophysiology to practice NKG2A inhibits invariant NKT cell activation in hepatic injury.Statins impair CD1d-mediated antigen presentation through the inhibition of prenylation.The stimulating adventure of KRN 7000.The immune-modulatory role of apolipoprotein E with emphasis on multiple sclerosis and experimental autoimmune encephalomyelitis.Primary deficiency of microsomal triglyceride transfer protein in human abetalipoproteinemia is associated with loss of CD1 function.Impact of CD1d deficiency on metabolism.Deficiency of antigen-presenting cell invariant chain reduces atherosclerosis in mice.Development and function of invariant natural killer T cells producing T(h)2- and T(h)17-cytokines Type II NKT cells stimulate diet-induced obesity by mediating adipose tissue inflammation, steatohepatitis and insulin resistance.Development of spontaneous anergy in invariant natural killer T cells in a mouse model of dyslipidemia.The role of natural killer T cells in costimulation blockade-based mixed chimerism Invariant natural killer T (iNKT) cells in HAART-treated, HIV-positive patients with bone and cardiovascular impairment.Role of Innate T Cells in Anti-Bacterial Immunity.Activation of CD11b+ Kupffer cells/macrophages as a common cause for exacerbation of TNF/Fas-ligand-dependent hepatitis in hypercholesterolemic mice.The IL-33/ST2 pathway: therapeutic target and novel biomarker.Lymphocytes in atherosclerosis.Invariant natural killer T cells from children with versus without food allergy exhibit differential responsiveness to milk-derived sphingomyelin Deleting TCR alpha beta+ or CD4+ T lymphocytes leads to opposite effects on site-specific atherosclerosis in female apolipoprotein E-deficient mice.Lymphocyte migration into atherosclerotic plaque.Beta-glucosylceramide: a novel method for enhancement of natural killer T lymphoycte plasticity in murine models of immune-mediated disorders Characterization of the natural killer T-cell response in an adoptive transfer model of atherosclerosis.Natural killer T cells as targets for immunotherapy of autoimmune diseases.IFN-gamma-mediated negative feedback regulation of NKT-cell function by CD94/NKG2.Going both ways: immune regulation via CD1d-dependent NKT cells.Thematic review series: the immune system and atherogenesis. Immune function in atherogenesis.Genetic basis of atherosclerosis: insights from mice and humans Activation of invariant natural killer T cells by lipid excess promotes tissue inflammation, insulin resistance, and hepatic steatosis in obese mice.alpha-Galactosylceramide therapy for autoimmune diseases: prospects and obstacles.Thematic review series: the immune system and atherogenesis. The unusual suspects:an overview of the minor leukocyte populations in atherosclerosis.

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P2860

CD1d-dependent activation of NKT cells aggravates atherosclerosis.

http://www.wikidata.org/entity/Q36399122

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2004 nî lūn-bûn

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CD1d-dependent activation of NKT cells aggravates atherosclerosis.

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CD1d-dependent activation of NKT cells aggravates atherosclerosis.

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CD1d-dependent activation of NKT cells aggravates atherosclerosis.

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CD1d-dependent activation of NKT cells aggravates atherosclerosis.

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CD1d-dependent activation of NKT cells aggravates atherosclerosis.

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CD1d-dependent activation of NKT cells aggravates atherosclerosis.

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Journal of Experimental Medicine

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CD1d-dependent activation of NKT cells aggravates atherosclerosis

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P2093

Emmanuel Tupin

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Gabrielle Paulsson Berne

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Göran K Hansson

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Hans-Gustaf Ljunggren

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Mats Rudling

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Rima Elhage

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P2860

CD1d-restricted and TCR-mediated activation of valpha14 NKT cells by glycosylceramides

CD1d1 mutant mice are deficient in natural T cells that promptly produce IL-4

NKT cells: facts, functions and fallacies.

Immune mechanisms in atherosclerosis.

Interleukin-10 deficiency increases atherosclerosis, thrombosis, and low-density lipoproteins in apolipoprotein E knockout mice.

The unconventional lifestyle of NKT cells.

Innate and acquired immunity in atherogenesis.

CD1-restricted antigen presentation: an oily matter.

NKT cell subsets in infection and inflammation.

Interleukins in atherosclerosis: molecular pathways and therapeutic potential.

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417-422

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10.1084/JEM.20030997

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3

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199

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Antonino Nicoletti

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2004-01-26T00:00:00Z

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8901636

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14744994

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atherosclerosis

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2211791

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