Impaired clearance of apoptotic cells promotes synergy between atherogenesis and autoimmune disease - Wikidata - awgldk - TriplyDB

Impaired clearance of apoptotic cells promotes synergy between atherogenesis and autoimmune disease

Impaired clearance of apoptotic cells promotes synergy between atherogenesis and autoimmune disease

http://www.wikidata.org/entity/Q36399434

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Lysophosphatidylcholine acyltransferase 3 knockdown-mediated liver lysophosphatidylcholine accumulation promotes very low density lipoprotein production by enhancing microsomal triglyceride transfer protein expression Low-density lipoprotein receptor-related protein 1 prevents early atherosclerosis by limiting lesional apoptosis and inflammatory Ly-6Chigh monocytosis: evidence that the effects are not apolipoprotein E dependent Inhibition of lipoprotein-associated phospholipase A2 reduces complex coronary atherosclerotic plaque development Liver X receptor and peroxisome proliferator-activated receptor as integrators of lipid homeostasis and immunity Cardiovascular disease in lupus: insights and updates Chemerin peptides promote phagocytosis in a ChemR23- and Syk-dependent manner The effect of mycophenolate mofetil on disease development in the gld.apoE (-/-) mouse model of accelerated atherosclerosis and systemic lupus erythematosus The peroxisome proliferator-activated receptor gamma agonist rosiglitazone ameliorates murine lupus by induction of adiponectin ACAT inhibition reduces the progression of preexisting, advanced atherosclerotic mouse lesions without plaque or systemic toxicity.Cholesterol-induced membrane microvesicles as novel carriers of damage-associated molecular patterns: mechanisms of formation, action, and detoxification.Increased sensitivity of Apolipoprotein E knockout mice to swainsonine dependent immunomodulation.Macrophage death and defective inflammation resolution in atherosclerosis.TLR4 ligands induce IFN-alpha production by mouse conventional dendritic cells and human monocytes after IFN-beta priming Treatment with apolipoprotein A-1 mimetic peptide reduces lupus-like manifestations in a murine lupus model of accelerated atherosclerosis.Tyro3 receptor tyrosine kinases in the heterogeneity of apoptotic cell uptake.Adiponectin modulates inflammatory reactions via calreticulin receptor-dependent clearance of early apoptotic bodies.The Role of Efferocytosis in Atherosclerosis.Insulin resistance and atherosclerosis.IRF5 deficiency ameliorates lupus but promotes atherosclerosis and metabolic dysfunction in a mouse model of lupus-associated atherosclerosis Hematopoietic Fas deficiency does not affect experimental atherosclerotic lesion formation despite inducing a proatherogenic state.Lupus-prone New Zealand Black/New Zealand White F1 mice display endothelial dysfunction and abnormal phenotype and function of endothelial progenitor cells.The immunomodulatory parasitic worm product ES-62 reduces lupus-associated accelerated atherosclerosis in a mouse model Atherosclerosis in systemic lupus erythematosus.Hypercholesterolemia and ApoE deficiency result in severe infection with Lyme disease and relapsing-fever Borrelia.Apoptotic neutrophils and T cells sequester chemokines during immune response resolution through modulation of CCR5 expression.Plasmacytoid dendritic cells in atherosclerosis.Type I interferons modulate vascular function, repair, thrombosis, and plaque progression in murine models of lupus and atherosclerosis Apoptotic cells with oxidation-specific epitopes are immunogenic and proinflammatory A novel mouse model that develops spontaneous arthritis and is predisposed towards atherosclerosis.Distinct roles for complement in glomerulonephritis and atherosclerosis revealed in mice with a combination of lupus and hyperlipidemia.17-β Estradiol reduces atherosclerosis without exacerbating lupus in ovariectomized systemic lupus erythematosus-susceptible LDLr(-/-) mice.Accelerated atherosclerosis in ApoE deficient lupus mouse models.The link between antibodies to OxLDL and natural protection against pneumococci depends on D(H) gene conservation.Trypanosoma cruzi infection is enhanced by vector saliva through immunosuppressant mechanisms mediated by lysophosphatidylcholine Autoimmunity in atherosclerosis: a protective response losing control?Accelerated atherosclerosis is independent of feeding high fat diet in systemic lupus erythematosus-susceptible LDLr(-/-) mice.The inextricable link between atherosclerosis and prototypical inflammatory diseases rheumatoid arthritis and systemic lupus erythematosus.Immune and inflammatory mechanisms of atherosclerosis (*)Extravascular inflammation does not increase atherosclerosis in apoE-deficient mice.G2A and LPC: regulatory functions in immunity.

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P2860

Impaired clearance of apoptotic cells promotes synergy between atherogenesis and autoimmune disease

http://www.wikidata.org/entity/Q36399434

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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Impaired clearance of apoptoti ...... genesis and autoimmune disease

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Impaired clearance of apoptoti ...... genesis and autoimmune disease

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Impaired clearance of apoptoti ...... genesis and autoimmune disease

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Impaired clearance of apoptoti ...... genesis and autoimmune disease

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Impaired clearance of apoptoti ...... genesis and autoimmune disease

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Impaired clearance of apoptoti ...... genesis and autoimmune disease

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P1433

Journal of Experimental Medicine

P1476

Impaired clearance of apoptoti ...... genesis and autoimmune disease

@en

P2093

Bénédicte Hugel

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Ian Rifkin

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Jean-Marie Freyssinet

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John J Castellot

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Kaori Sato

http://www.w3.org/2001/XMLSchema#string

Kenneth Walsh

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Ramon Bonegio

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Tamar Aprahamian

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P2860

Atherosclerosis — An Inflammatory Disease

Mutation of DNASE1 in people with systemic lupus erythematosus

A composite picture of TcR alpha/beta(+) CD4(-)CD8(-) T Cells (alpha/beta-DNTCs) in humans with autoimmune lymphoproliferative syndrome

Severe hypercholesterolemia and atherosclerosis in apolipoprotein E-deficient mice created by homologous recombination in ES cells

Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand

The pathogenesis of atherosclerosis: a perspective for the 1990s

The Fas death factor

Mutations in Fas associated with human lymphoproliferative syndrome and autoimmunity.

Fas gene mutations in the Canale-Smith syndrome, an inherited lymphoproliferative disorder associated with autoimmunity.

Th1 CD4+ lymphocytes delete activated macrophages through the Fas/APO-1 antigen pathway.

P304

1121-1131

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10.1084/JEM.20031557

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8

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199

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8606870

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15096538

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apoptotic process

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2211887

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