Prevention and treatment of experimental autoimmune encephalomyelitis by soluble CD83
about
The dark side of dendritic cells: development and exploitation of tolerogenic activity that favor tumor outgrowth and immune escape.Genes involved in the balance between neuronal survival and death during inflammationCD83 modulates B cell function in vitro: increased IL-10 and reduced Ig secretion by CD83Tg B cells.Influence of dendritic cells on viral pathogenicityA Kinetic Study of CD83 Reveals an Upregulation and Higher Production of sCD83 in Lymphocytes from Pregnant MiceAn increase of CD83+ dendritic cells ex vivo correlates with increased regulatory T cells in patients with active eosinophilic granulomatosis and polyangiitis.CD83 increases MHC II and CD86 on dendritic cells by opposing IL-10-driven MARCH1-mediated ubiquitination and degradation.The transmembrane E3 ligase GRAIL ubiquitinates and degrades CD83 on CD4 T cells.Dendritic cell CD83 homotypic interactions regulate inflammation and promote mucosal homeostasisThe expression and characterization of functionally active soluble CD83 by Pichia pastoris using high-density fermentation.CD83-stimulated monocytes suppress T-cell immune responses through production of prostaglandin E2.L Particles Transmit Viral Proteins from Herpes Simplex Virus 1-Infected Mature Dendritic Cells to Uninfected Bystander Cells, Inducing CD83 DownmodulationDancing with the enemy: the interplay of herpes simplex virus with dendritic cells.The CD83 reporter mouse elucidates the activity of the CD83 promoter in B, T, and dendritic cell populations in vivo.Dendritic cell CD83: a therapeutic target or innocent bystander?Th-1 polarization is regulated by dendritic-cell comparison of MHC class I and class II antigens.Thymic CD4 T cell selection requires attenuation of March8-mediated MHCII turnover in cortical epithelial cells through CD83.Heat-induced reactivation of HSV-1 in latent mice: upregulation in the TG of CD83 and other immune response genes and their LAT-ICP0 locus.CD83 polymorphisms and cervical cancer risk.Soluble CD83 Inhibits T Cell Activation by Binding to the TLR4/MD-2 Complex on CD14+ Monocytes.Multiple interferon regulatory factor and NF-κB sites cooperate in mediating cell-type- and maturation-specific activation of the human CD83 promoter in dendritic cells.Phosphorylation of the HuR ligand APRIL by casein kinase 2 regulates CD83 expression.Herpes simplex virus type 1 induces CD83 degradation in mature dendritic cells with immediate-early kinetics via the cellular proteasome.Sex Bias in Pathogenesis of Autoimmune Neuroinflammation: Relevance for Dimethyl Fumarate Immunomodulatory/Anti-oxidant Action.Tolerogenic dendritic cells pulsed with enterobacterial extract suppress development of colitis in the severe combined immunodeficiency transfer model.The Major Immediate-Early Protein IE2 of Human Cytomegalovirus Is Sufficient to Induce Proteasomal Degradation of CD83 on Mature Dendritic CellsCD83 Modulates B Cell Activation and Germinal Center Responses.Thymic stromal lymphopoietin deficiency attenuates experimental autoimmune encephalomyelitis.Post-transcriptional regulation of CD83 expression by AUF1 proteinsSoluble CD14 and CD83 from human neonatal antigen-presenting cells are inducible by commensal bacteria and suppress allergen-induced human neonatal Th2 differentiation.Inhaled iloprost suppresses the cardinal features of asthma via inhibition of airway dendritic cell function.NK cells are negatively regulated by sCD83 in experimental autoimmune uveitisTargeting CD83 for the treatment of graft-versus-host disease.Dexamethasone/1alpha-25-dihydroxyvitamin D3-treated dendritic cells suppress colitis in the SCID T-cell transfer model.Involvement of Caveolin-1 in CD83 Internalization in Mouse Dendritic Cells.NFATc1 deficiency in T cells protects mice from experimental autoimmune encephalomyelitis.Increased levels of circulating soluble CD14 but not CD83 in infants are associated with early intestinal colonization with Staphylococcus aureus.Tolerogenic Transcriptional Signatures of Steady-State and Pathogen-Induced Dendritic Cells.An Expanded Neuroimmunomodulation Axis: sCD83-Indoleamine 2,3-Dioxygenase—Kynurenine Pathway and Updates of Kynurenine Pathway in Neurologic Diseases.
P2860
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P2860
Prevention and treatment of experimental autoimmune encephalomyelitis by soluble CD83
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Prevention and treatment of experimental autoimmune encephalomyelitis by soluble CD83
@ast
Prevention and treatment of experimental autoimmune encephalomyelitis by soluble CD83
@en
type
label
Prevention and treatment of experimental autoimmune encephalomyelitis by soluble CD83
@ast
Prevention and treatment of experimental autoimmune encephalomyelitis by soluble CD83
@en
prefLabel
Prevention and treatment of experimental autoimmune encephalomyelitis by soluble CD83
@ast
Prevention and treatment of experimental autoimmune encephalomyelitis by soluble CD83
@en
P2093
P2860
P356
P1476
Prevention and treatment of experimental autoimmune encephalomyelitis by soluble CD83
@en
P2093
Alexander Steinkasserer
Antje Golka
Elisabeth Zinser
Manfred B Lutz
Matthias Lechmann
P2860
P304
P356
10.1084/JEM.20030973
P407
P577
2004-08-01T00:00:00Z