The peripheral deletion of autoreactive CD8+ T cells induced by cross-presentation of self-antigens involves signaling through CD95 (Fas, Apo-1).
about
Signalling through CD30 protects against autoimmune diabetes mediated by CD8 T cellsInduction of immunological tolerance by apoptotic cells requires caspase-dependent oxidation of high-mobility group box-1 proteinThe intriguing biology of the tumour necrosis factor/tumour necrosis factor receptor superfamily: players, rules and the gamesUbiquitin in the immune systemCell death in the maintenance and abrogation of tolerance: the five Ws of dying cellsLymphopenia-induced proliferation in aire-deficient mice helps to explain their autoimmunity and differences from human patientsMutation in the Fas pathway impairs CD8+ T cell memory.CD30 prevents T-cell responses to non-lymphoid tissues.Role of dendritic cells in the induction and maintenance of autoimmune diseases.Autoaggression and tumor rejection: it takes more than self-specific T-cell activation.The use of carboxyfluorescein diacetate succinimidyl ester to determine the site, duration and cell type responsible for antigen presentation in vivo.Cutting edge: merocytic dendritic cells break T cell tolerance to beta cell antigens in nonobese diabetic mouse diabetes.Review article: molecular signals and genetic reprogramming in peripheral T-cell differentiation.Autoimmunity and the immunotherapy of cancer: targeting the "self" to destroy the "other".The immune tolerance network and rheumatic disease: immune tolerance comes to the clinic.Dendritic cells under investigation in autoimmune disease.Cellular requirements for diabetes induction in DO11.10xRIPmOVA mice.Antibody-enhanced cross-presentation of self antigen breaks T cell tolerance.Immunologic ignorance of vascular endothelial cells expressing minor histocompatibility antigenUrinary bladder epithelium antigen induces CD8+ T cell tolerance, activation, and autoimmune response.In situ tolerance within the central nervous system as a mechanism for preventing autoimmunity.Peripheral deletion of autoreactive CD8 T cells by cross presentation of self-antigen occurs by a Bcl-2-inhibitable pathway mediated by BimConsequences of cell death: exposure to necrotic tumor cells, but not primary tissue cells or apoptotic cells, induces the maturation of immunostimulatory dendritic cellsImmunotherapy with dendritic cells directed against tumor antigens shared with normal host cells results in severe autoimmune disease.Phenotypic and functional analysis of CD8(+) T cells undergoing peripheral deletion in response to cross-presentation of self-antigen.Cross-tolerance: a pathway for inducing tolerance to peripheral tissue antigensB cells directly tolerize CD8(+) T cells.CD8 T cell ignorance or tolerance to islet antigens depends on antigen dose.Pro-inflammatory self-reactive T cells are found within murine TCR-αβ(+) CD4(-) CD8(-) PD-1(+) cells.Apoptosis as a mechanism of T-regulatory cell homeostasis and suppression.Donor Cell Composition and Reactivity Predict Risk of Acute Graft-versus-Host Disease after Allogeneic Hematopoietic Stem Cell Transplantation.The ambiguity in immunology.The potential role of dendritic cells in the therapy of Type 1 diabetes.Self-antigen presentation by dendritic cells in autoimmunity.Harnessing the properties of dendritic cells in the pursuit of immunological tolerance.A distinct role of CD4+ Th17- and Th17-stimulated CD8+ CTL in the pathogenesis of type 1 diabetes and experimental autoimmune encephalomyelitis.Cross-presentation of cell-associated antigens by mouse splenic dendritic cell populations.Functional specialization of islet dendritic cell subsetsRegulation of CD30 antigen expression and its potential significance for human disease.Incomplete Killing And Enhanced Activation of Islet-Reactive CD8+ T Cells by FasL-Expressing Dendritic Cells Limits Protection from Diabetes.
P2860
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P2860
The peripheral deletion of autoreactive CD8+ T cells induced by cross-presentation of self-antigens involves signaling through CD95 (Fas, Apo-1).
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh
1998年學術文章
@zh-hant
name
The peripheral deletion of aut ...... ing through CD95 (Fas, Apo-1).
@ast
The peripheral deletion of aut ...... ing through CD95 (Fas, Apo-1).
@en
type
label
The peripheral deletion of aut ...... ing through CD95 (Fas, Apo-1).
@ast
The peripheral deletion of aut ...... ing through CD95 (Fas, Apo-1).
@en
prefLabel
The peripheral deletion of aut ...... ing through CD95 (Fas, Apo-1).
@ast
The peripheral deletion of aut ...... ing through CD95 (Fas, Apo-1).
@en
P2093
P2860
P356
P1476
The peripheral deletion of aut ...... ing through CD95 (Fas, Apo-1).
@en
P2093
P2860
P304
P356
10.1084/JEM.188.2.415
P407
P577
1998-07-01T00:00:00Z