Fas and Fas ligand interactions suppress melanoma lung metastasis. - Wikidata - awgldk - TriplyDB

Fas and Fas ligand interactions suppress melanoma lung metastasis.

Fas and Fas ligand interactions suppress melanoma lung metastasis.

http://www.wikidata.org/entity/Q36401618

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FAP-1 association with Fas (Apo-1) inhibits Fas expression on the cell surface Expression of SCC-S2, an antiapoptotic molecule, correlates with enhanced proliferation and tumorigenicity of MDA-MB 435 cells TIP49b, a regulator of activating transcription factor 2 response to stress and DNA damage Transcriptional switch by activating transcription factor 2-derived peptide sensitizes melanoma cells to apoptosis and inhibits their tumorigenicity Metastasis: a question of life or death Decitabine and vorinostat cooperate to sensitize colon carcinoma cells to Fas ligand-induced apoptosis in vitro and tumor suppression in vivo NF-κB directly regulates Fas transcription to modulate Fas-mediated apoptosis and tumor suppression Matrilysin [MMP-7] expression selects for cells with reduced sensitivity to apoptosis CCL9 Induced by TGFβ Signaling in Myeloid Cells Enhances Tumor Cell Survival in the Premetastatic Organ.FAS/FASL expression profile as a prognostic marker in squamous cell carcinoma of the oral cavity.Engineering a waste management enzyme to overcome cancer resistance to apoptosis: adding DNase1 to the anti-cancer toolbox.Inducible resistance to Fas-mediated apoptosis in B cells.Ionizing radiation as a response-enhancing agent for CD95-mediated apoptosis.Induction of tumour cell apoptosis by matrix metalloproteinase inhibitors: new tricks from a (not so) old drug.Alterations in molecular killing mechanisms: implications in skin disease.An ATF2-derived peptide sensitizes melanomas to apoptosis and inhibits their growth and metastasis.Immunity to cancer: attack and escape in T lymphocyte-tumor cell interaction.Apoptosis, autophagy, necroptosis, and cancer metastasis Impaired CD95 expression predisposes for recurrence in curatively resected colon carcinoma: clinical evidence for immunoselection and CD95L mediated control of minimal residual disease.Loss of Fas-ligand expression in mouse keratinocytes during UV carcinogenesis Corruption of the Fas pathway delays the pulmonary clearance of murine osteosarcoma cells, enhances their metastatic potential, and reduces the effect of aerosol gemcitabine.TRAIL-R deficiency in mice enhances lymph node metastasis without affecting primary tumor development Elucidating the autoimmune and antitumor effector mechanisms of a treatment based on cytotoxic T lymphocyte antigen-4 blockade in combination with a B16 melanoma vaccine: comparison of prophylaxis and therapy Induction of lymphocyte apoptosis by tumor cell secretion of FasL-bearing microvesicles.The inhibitor of death receptor signaling, FLICE-inhibitory protein defines a new class of tumor progression factors.Unphosphorylated STAT1 promotes sarcoma development through repressing expression of Fas and bad and conferring apoptotic resistance.Granulocytes mediates the Fas-L-associated apoptosis during lung metastasis of melanoma that determines the metastatic behaviour Cellular liaisons of natural killer lymphocytes in immunology and immunotherapy of cancer.VEGF165 is necessary to the metastatic potential of Fas(-) osteosarcoma cells but will not rescue the Fas(+) cells Epigenetics and colorectal cancer pathogenesis Regulation of Fas expression by STAT3 and c-Jun is mediated by phosphatidylinositol 3-kinase-AKT signaling.The MLL1-H3K4me3 Axis-Mediated PD-L1 Expression and Pancreatic Cancer Immune Evasion.Circulating and disseminated tumour cells - mechanisms of immune surveillance and escape.Methotrexate inhibits the viability of human melanoma cell lines and enhances Fas/Fas-ligand expression, apoptosis and response to interferon-alpha: rationale for its use in combination therapy.Reduced FAS transcription in clones of U937 cells that have acquired resistance to Fas-induced apoptosis.Downregulation of IFN-gammaR in association with loss of Fas function is linked to tumor progression.The EphB4 receptor-tyrosine kinase promotes the migration of melanoma cells through Rho-mediated actin cytoskeleton reorganization.Role of apoptosis resistance in immune evasion and metastasis of colorectal cancer.Analysis of alternative splicing events for cancer diagnosis using a multiplexing nanophotonic biosensor Molecular alterations associated with osteosarcoma development.

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P2860

Fas and Fas ligand interactions suppress melanoma lung metastasis.

http://www.wikidata.org/entity/Q36401618

description

1998 nî lūn-bûn

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1998年の論文

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年學術文章

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1998年學術文章

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1998年學術文章

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name

Fas and Fas ligand interactions suppress melanoma lung metastasis.

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Fas and Fas ligand interactions suppress melanoma lung metastasis.

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type

label

Fas and Fas ligand interactions suppress melanoma lung metastasis.

@ast

Fas and Fas ligand interactions suppress melanoma lung metastasis.

@en

prefLabel

Fas and Fas ligand interactions suppress melanoma lung metastasis.

@ast

Fas and Fas ligand interactions suppress melanoma lung metastasis.

@en

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Journal of Experimental Medicine

P1476

Fas and Fas ligand interactions suppress melanoma lung metastasis.

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P2093

J E Price

http://www.w3.org/2001/XMLSchema#string

K L van Golen

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L B Owen-Schaub

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L L Hill

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P2860

Inhibition of death receptor signals by cellular FLIP

Apoptosis in the pathogenesis and treatment of disease

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

FAP-1: a protein tyrosine phosphatase that associates with Fas

Apoptosis by death factor

Speculations on the role of ultraviolet radiation in the development of malignant melanoma.

CD28 costimulation can promote T cell survival by enhancing the expression of Bcl-XL.

Why do cancer cells metastasize into particular organs?

Fas and Fas ligand in embryos and adult mice: ligand expression in several immune-privileged tissues and coexpression in adult tissues characterized by apoptotic cell turnover.

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1717-1723

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10.1084/JEM.188.9.1717

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188

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13483504

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9802983

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2212505

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