Age-by-disease biological interactions: implications for late-life depression.
about
Hypermethylation of BDNF and SST Genes in the Orbital Frontal Cortex of Older Individuals: A Putative Mechanism for Declining Gene Expression with Age.The effect of gender, age, and symptom severity in late-life depression on the risk of all-cause mortality: the Bambuí Cohort Study of Aging.Increased levels of ascorbic acid in the cerebrospinal fluid of cognitively intact elderly patients with major depression: a preliminary study.The Role of BDNF in Age-Dependent Changes of Excitatory and Inhibitory Synaptic Markers in the Human Prefrontal Cortex.Does physical activity moderate the relationship between depression symptomatology and low back pain? Cohort and co-twin control analyses nested in the longitudinal study of aging Danish twins (LSADT).The role of the nervous system in aging and longevity.
P2860
Age-by-disease biological interactions: implications for late-life depression.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
@wuu
2012年学术文章
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2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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2012年學術文章
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name
Age-by-disease biological interactions: implications for late-life depression.
@ast
Age-by-disease biological interactions: implications for late-life depression.
@en
type
label
Age-by-disease biological interactions: implications for late-life depression.
@ast
Age-by-disease biological interactions: implications for late-life depression.
@en
prefLabel
Age-by-disease biological interactions: implications for late-life depression.
@ast
Age-by-disease biological interactions: implications for late-life depression.
@en
P2860
P356
P1476
Age-by-disease biological interactions: implications for late-life depression
@en
P2093
Etienne Sibille
Hyunjung Oh
P2860
P356
10.3389/FGENE.2012.00237
P577
2012-11-16T00:00:00Z