Glucose-fatty acid interaction in skeletal muscle and adipose tissue in insulin resistance.
about
Viewpoints on the way to the consensus session: where does insulin resistance start? The adipose tissue.Mutant amyloid precursor protein differentially alters adipose biology under obesogenic and non-obesogenic conditions.Two-tiered approach identifies a network of cancer and liver disease-related genes regulated by miR-122.Acute hyperinsulinemia and reduced plasma free fatty acid levels decrease intramuscular triglyceride synthesis.Fat feeding potentiates the diabetogenic effect of dexamethasone in Wistar rats.Exercise improved lipid metabolism and insulin sensitivity in rats fed a high-fat diet by regulating glucose transporter 4 (GLUT4) and musclin expression.The sedentary (r)evolution: Have we lost our metabolic flexibility?Aqueous-Methanol Extracts of Orange-Fleshed Sweet Potato () Ameliorate Oxidative Stress and Modulate Type 2 Diabetes Associated Genes in Insulin Resistant C2C12 Cells
P2860
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P2860
Glucose-fatty acid interaction in skeletal muscle and adipose tissue in insulin resistance.
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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name
Glucose-fatty acid interaction ...... tissue in insulin resistance.
@ast
Glucose-fatty acid interaction ...... tissue in insulin resistance.
@en
type
label
Glucose-fatty acid interaction ...... tissue in insulin resistance.
@ast
Glucose-fatty acid interaction ...... tissue in insulin resistance.
@en
prefLabel
Glucose-fatty acid interaction ...... tissue in insulin resistance.
@ast
Glucose-fatty acid interaction ...... tissue in insulin resistance.
@en
P2093
P921
P1476
Glucose-fatty acid interaction ...... e tissue in insulin resistance
@en
P2093
P577
2006-02-23T00:00:00Z