Inhibition of histone deacetylase-induced myocardial repair is mediated by c-kit in infarcted hearts.
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HDAC and HDAC Inhibitor: From Cancer to Cardiovascular DiseasesHDAC class I inhibitor, Mocetinostat, reverses cardiac fibrosis in heart failure and diminishes CD90+ cardiac myofibroblast activationSpecific inhibition of HDAC4 in cardiac progenitor cells enhances myocardial repairs.Stimulation of glucagon-like peptide-1 receptor through exendin-4 preserves myocardial performance and prevents cardiac remodeling in infarcted myocardium.HDAC inhibition elicits myocardial protective effect through modulation of MKK3/Akt-1.HDAC4 degradation mediates HDAC inhibition-induced protective effects against hypoxia/reoxygenation injury.Anti-Fibrotic Effects of Class I HDAC Inhibitor, Mocetinostat Is Associated with IL-6/Stat3 Signaling in Ischemic Heart Failure.Histone deacetylase (HDAC) inhibition improves myocardial function and prevents cardiac remodeling in diabetic mice.Irisin Ameliorates Hypoxia/Reoxygenation-Induced Injury through Modulation of Histone Deacetylase 4Attenuation of ER stress prevents post-infarction-induced cardiac rupture and remodeling by modulating both cardiac apoptosis and fibrosis.Inhibition of Oct 3/4 mitigates the cardiac progenitor-derived myocardial repair in infarcted myocardium.Exendin-4 induces myocardial protection through MKK3 and Akt-1 in infarcted heartsHDAC-dependent ventricular remodeling.Histone deacetylase inhibition blunts ischemia/reperfusion injury by inducing cardiomyocyte autophagy.c-kit(+) cells: the tell-tale heart of cardiac regeneration?Generation of cardiac progenitor cells through epicardial to mesenchymal transition.The Impact of Environmental Factors in Influencing Epigenetics Related to Oxidative States in the Cardiovascular System.Transplantation of Epigenetically Modified Adult Cardiac c-Kit+ Cells Retards Remodeling and Improves Cardiac Function in Ischemic Heart Failure Model.New Aspects of Vascular Calcification: Histone Deacetylases and Beyond.Sodium Butyrate Protects -Against High Fat Diet-Induced Cardiac Dysfunction and Metabolic Disorders in Type II Diabetic Mice.Epigenetics and precision medicine in cardiovascular patients: from basic concepts to the clinical arena.
P2860
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P2860
Inhibition of histone deacetylase-induced myocardial repair is mediated by c-kit in infarcted hearts.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
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2012年學術文章
@zh-hant
name
Inhibition of histone deacetyl ...... by c-kit in infarcted hearts.
@ast
Inhibition of histone deacetyl ...... by c-kit in infarcted hearts.
@en
type
label
Inhibition of histone deacetyl ...... by c-kit in infarcted hearts.
@ast
Inhibition of histone deacetyl ...... by c-kit in infarcted hearts.
@en
prefLabel
Inhibition of histone deacetyl ...... by c-kit in infarcted hearts.
@ast
Inhibition of histone deacetyl ...... by c-kit in infarcted hearts.
@en
P2093
P2860
P356
P1476
Inhibition of histone deacetyl ...... by c-kit in infarcted hearts.
@en
P2093
Gang J Qin
Ling Zhang
Patrycja M Dubielecka
Ting C Zhao
Y Eugene Chin
Yigang Wang
P2860
P304
39338-39348
P356
10.1074/JBC.M112.379115
P407
P577
2012-09-28T00:00:00Z