Increased excitability of acidified skeletal muscle: role of chloride conductance. - Wikidata - awgldk - TriplyDB

Increased excitability of acidified skeletal muscle: role of chloride conductance.

Increased excitability of acidified skeletal muscle: role of chloride conductance.

http://www.wikidata.org/entity/Q36412593

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Clinical review: practical approach to hyponatraemia and hypernatraemia in critically ill patients Role of physiological ClC-1 Cl- ion channel regulation for the excitability and function of working skeletal muscle Channelopathies of skeletal muscle excitability Rapid Ca2+ flux through the transverse tubular membrane, activated by individual action potentials in mammalian skeletal muscle.Regional differences in osmotic behavior in brain during acute hyponatremia: an in vivo MRI-study of brain and skeletal muscle in pigs.CLC channels and transporters: proteins with borderline personalities.The Sick and the Weak: Neuropathies/Myopathies in the Critically Ill.Binding of ATP to the CBS domains in the C-terminal region of CLC-1.An analysis of the relationships between subthreshold electrical properties and excitability in skeletal muscle.Na+,K+-pump stimulation improves contractility in isolated muscles of mice with hyperkalemic periodic paralysis Relationships between resting conductances, excitability, and t-system ionic homeostasis in skeletal muscle.Properties of single FDB fibers following a collagenase digestion for studying contractility, fatigue, and pCa-sarcomere shortening relationship.ClC-1 chloride channels: state-of-the-art research and future challenges.Excitation of skeletal muscle is a self-limiting process, due to run-down of Na+, K+ gradients, recoverable by stimulation of the Na+, K+ pumps.Niflumic acid inhibits chloride conductance of rat skeletal muscle by directly inhibiting the CLC-1 channel and by increasing intracellular calcium.Intracellular β-nicotinamide adenine dinucleotide inhibits the skeletal muscle ClC-1 chloride channel.Cytoplasmic ATP inhibition of CLC-1 is enhanced by low pH Understanding the physiology of the asymptomatic diaphragm of the M1592V hyperkalemic periodic paralysis mouse.The muscle chloride channel ClC-1 is not directly regulated by intracellular ATP Resting potential-dependent regulation of the voltage sensitivity of sodium channel gating in rat skeletal muscle in vivo.Excitation-induced exchange of Na+, K+, and Cl- in rat EDL muscle in vitro and in vivo: physiology and pathophysiology.ClC-1 chloride channel: Matching its properties to a role in skeletal muscle.Defects in Neuromuscular Transmission May Underlie Motor Dysfunction in Spinal and Bulbar Muscular Atrophy ATP inhibition of CLC-1 is controlled by oxidation and reduction Do multiple ionic interactions contribute to skeletal muscle fatigue?Comparison of regulated passive membrane conductance in action potential-firing fast- and slow-twitch muscle Regulation of ClC-1 and KATP channels in action potential-firing fast-twitch muscle fibers Periodic limb movements are associated with vasomotor symptoms.Sarcolemmal ATP-sensitive potassium channels modulate skeletal muscle function under low-intensity workloads.A peripheral governor regulates muscle contraction.Cell biology and physiology of CLC chloride channels and transporters.Hypoxia and Its Acid-Base Consequences: From Mountains to Malignancy.Limitations in intense exercise performance of athletes - effect of speed endurance training on ion handling and fatigue development.Inhibition of skeletal muscle ClC-1 chloride channels by low intracellular pH and ATP.KATP channel deficiency in mouse FDB causes an impairment of energy metabolism during fatigue.To ATP or not to ATP: this is the question Disuse of rat muscle in vivo reduces protein kinase C activity controlling the sarcolemma chloride conductance.Lactate per se improves the excitability of depolarized rat skeletal muscle by reducing the Cl- conductance.Effect of purinergic receptor activation on Na+-K+ pump activity, excitability, and function in depolarized skeletal muscle.Clearance of extracellular K+ during muscle contraction--roles of membrane transport and diffusion.

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P2860

Increased excitability of acidified skeletal muscle: role of chloride conductance.

http://www.wikidata.org/entity/Q36412593

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2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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Increased excitability of acidified skeletal muscle: role of chloride conductance.

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Increased excitability of acidified skeletal muscle: role of chloride conductance.

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Increased excitability of acidified skeletal muscle: role of chloride conductance.

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The Journal of General Physiology

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Increased excitability of acidified skeletal muscle: role of chloride conductance.

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Frank de Paoli

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Ole B Nielsen

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Thomas H Pedersen

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P2860

The effect of sodium ions on the electrical activity of the giant axon of the squid

The skeletal muscle chloride channel in dominant and recessive human myotonia

Protective effects of lactic acid on force production in rat skeletal muscle

Voltage-gated ion channels and hereditary disease.

Dynamics and consequences of potassium shifts in skeletal muscle and heart during exercise.

Molecular structure and physiological function of chloride channels.

The resting membrane parameters of human intercostal muscle at low, normal, and high extracellular potassium.

Intracellular acidosis enhances the excitability of working muscle.

A study of the reinnervation of fast and slow mammalian muscles

Ionic blockage of sodium channels in nerve.

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237-246

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scholarly article

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10.1085/JGP.200409173

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2

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125

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