Exposure to ALS-FTD-CSF generates TDP-43 aggregates in glioblastoma cells through exosomes and TNTs-like structure.
about
Extracellular Vesicles and a Novel Form of Communication in the BrainTunneling nanotube (TNT)-mediated neuron-to neuron transfer of pathological Tau protein assembliesα-synuclein transfer through tunneling nanotubes occurs in SH-SY5Y cells and primary brain pericytes from Parkinson's disease patientsSingle cell imaging and quantification of TDP-43 and α-synuclein intercellular propagation.In vitro prion-like behaviour of TDP-43 in ALS.Exosomes, an Unmasked Culprit in Neurodegenerative Diseases.The role of exosomes in the pathogenesis of Alzheimer' disease.Physiological and pathological roles of exosomes in the nervous system.Exosome secretion is a key pathway for clearance of pathological TDP-43.Disease Mechanisms in ALS: Misfolded SOD1 Transferred Through Exosome-Dependent and Exosome-Independent Pathways.Prion-like propagation as a pathogenic principle in frontotemporal dementia.Prions on the run: How extracellular vesicles serve as delivery vehicles for self-templating protein aggregatesThe spread of prion-like proteins by lysosomes and tunneling nanotubes: Implications for neurodegenerative diseases.Extracellular Vesicles, Tunneling Nanotubes, and Cellular Interplay: Synergies and Missing Links.Isolation of Exosomes and Microvesicles from Cell Culture Systems to Study Prion Transmission.Proteomic analysis of FUS interacting proteins provides insights into FUS function and its role in ALS.Cell-to-cell transfer of SAA1 protein in a cell culture model of systemic AA amyloidosisPerspective Insights of Exosomes in Neurodegenerative Diseases: A Critical Appraisal.Tunneling nanotubes spread fibrillar α-synuclein by intercellular trafficking of lysosomes.Let's make microglia great again in neurodegenerative disorders.Etiogenic factors present in the cerebrospinal fluid from amyotrophic lateral sclerosis patients induce predominantly pro-inflammatory responses in microglia.Robustness and Vulnerability of the Autoregulatory System That Maintains Nuclear TDP-43 Levels: A Trade-off Hypothesis for ALS Pathology Based on in Silico Data.Pathomechanisms of TDP-43 in neurodegeneration.Exosomes in cancer: small vesicular transporters for cancer progression and metastasis, biomarkers in cancer therapeutics.Unconventional Secretion and Intercellular Transfer of Mutant Huntingtin.Aggregated SOD1 causes selective death of cultured human motor neuronsRole of Extracellular Vesicles in Amyotrophic Lateral Sclerosis
P2860
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P2860
Exposure to ALS-FTD-CSF generates TDP-43 aggregates in glioblastoma cells through exosomes and TNTs-like structure.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年学术文章
@wuu
2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
@zh
2015年學術文章
@zh-hant
name
Exposure to ALS-FTD-CSF genera ...... somes and TNTs-like structure.
@ast
Exposure to ALS-FTD-CSF genera ...... somes and TNTs-like structure.
@en
type
label
Exposure to ALS-FTD-CSF genera ...... somes and TNTs-like structure.
@ast
Exposure to ALS-FTD-CSF genera ...... somes and TNTs-like structure.
@en
prefLabel
Exposure to ALS-FTD-CSF genera ...... somes and TNTs-like structure.
@ast
Exposure to ALS-FTD-CSF genera ...... somes and TNTs-like structure.
@en
P2093
P2860
P356
P1433
P1476
Exposure to ALS-FTD-CSF genera ...... osomes and TNTs-like structure
@en
P2093
Ekokobe Fonkem
Jingzheng Yin
Junfang Teng
Mingming Ma
Robert K F Teng
Xuebing Ding
Xuejing Wang
P2860
P304
24178-24191
P356
10.18632/ONCOTARGET.4680
P407
P577
2015-09-01T00:00:00Z