The cellular ataxia telangiectasia-mutated kinase promotes epstein-barr virus lytic reactivation in response to multiple different types of lytic reactivation-inducing stimuli
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Modulation of DNA damage and repair pathways by human tumour virusesRegulation of the latent-lytic switch in Epstein-Barr virusEpstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesisReactive oxygen species mediate Epstein-Barr virus reactivation by N-methyl-N'-nitro-N-nitrosoguanidineIdentification of Novel Small Organic Compounds with Diverse Structures for the Induction of Epstein-Barr Virus (EBV) Lytic Cycle in EBV-Positive Epithelial MalignanciesActivation and repression of Epstein-Barr Virus and Kaposi's sarcoma-associated herpesvirus lytic cycles by short- and medium-chain fatty acidsA locus encompassing the Epstein-Barr virus bglf4 kinase regulates expression of genes encoding viral structural proteinsDNA Damage Signaling Is Induced in the Absence of Epstein-Barr Virus (EBV) Lytic DNA Replication and in Response to Expression of ZEBRA.Targeted therapy for Epstein-Barr virus-associated gastric carcinoma using low-dose gemcitabine-induced lytic activation.LMP1-Induced Sumoylation Influences the Maintenance of Epstein-Barr Virus Latency through KAP1ATM facilitates mouse gammaherpesvirus reactivation from myeloid cells during chronic infection.Oncogenic Herpesvirus Utilizes Stress-Induced Cell Cycle Checkpoints for Efficient Lytic ReplicationChloroquine triggers Epstein-Barr virus replication through phosphorylation of KAP1/TRIM28 in Burkitt lymphoma cellsMurine Gammaherpesvirus 68 LANA and SOX Homologs Counteract ATM-Driven p53 Activity during Lytic Viral ReplicationHuman papillomaviruses: research priorities for the next decade.Epstein-Barr virus induces global changes in cellular mRNA isoform usage that are important for the maintenance of latency.Potential of protein kinase inhibitors for treating herpesvirus-associated disease.Tumor Suppressor p53 Stimulates the Expression of Epstein-Barr Virus Latent Membrane Protein 1.H2AX phosphorylation and DNA damage kinase activity are dispensable for herpes simplex virus replication.Inhibition of class I histone deacetylases by romidepsin potently induces Epstein-Barr virus lytic cycle and mediates enhanced cell death with ganciclovir.Activation of DNA Damage Response Pathways during Lytic Replication of KSHV.Role of ATM in the formation of the replication compartment during lytic replication of Epstein-Barr virus in nasopharyngeal epithelial cells.EBV-related lymphomas: new approaches to treatment.Take your PIKK: tumour viruses and DNA damage response pathways.ATM supports gammaherpesvirus replication by attenuating type I interferon pathway.The Translesion Polymerase Pol η Is Required for Efficient Epstein-Barr Virus Infectivity and Is Regulated by the Viral Deubiquitinating Enzyme BPLF1.Small molecule perturbation of the CAND1-Cullin1-ubiquitin cycle stabilizes p53 and triggers Epstein-Barr virus reactivation.B Cell-Specific Expression of Ataxia-Telangiectasia Mutated Protein Kinase Promotes Chronic Gammaherpesvirus Infection.Epstein-Barr Virus Rta-Mediated Accumulation of DNA Methylation Interferes with CTCF Binding in both Host and Viral Genomes.The Rad6/18 ubiquitin complex interacts with the Epstein-Barr virus deubiquitinating enzyme, BPLF1, and contributes to virus infectivity.Epstein-Barr Virus Hijacks DNA Damage Response Transducers to Orchestrate Its Life Cycle.EBV-encoded miRNAs target ATM-mediated response in nasopharyngeal carcinoma.A Screen for Epstein-Barr Virus Proteins that Inhibit the DNA Damage Response Reveals a Novel Histone Binding Protein.Investigating genetic-and-epigenetic networks, and the cellular mechanisms occurring in Epstein-Barr virus-infected human B lymphocytes via big data mining and genome-wide two-sided NGS data identification
P2860
Q26825783-038C9D21-ABD5-42FD-ACBA-F93FC9BEC7C8Q27021366-963B3661-1D28-426E-A49D-B97B55FD60B9Q28079510-EE9E6099-70B5-4B35-956B-6FFF9F0B33BAQ28537881-98E74CFF-E096-4626-AC84-64764FC0936CQ28551855-88B342B4-1D2C-4045-B578-D1796313E024Q33900360-3937A9A2-F467-44AE-8C79-D0BCB19B8C69Q34105467-1D6B3459-CF47-494F-A76D-805EEFAE4F12Q35624112-A587053C-9623-4D5A-8CC2-D7249F81072FQ35795380-6E1D103B-373F-4E95-943B-58A492417952Q35861199-1175348B-B746-4B9A-BB96-8A0FEAFC2A31Q35893600-F43ACF5A-F5AA-4509-AD79-C37292045AE1Q35927487-DB2312C0-0888-4AF6-A9CA-EA98D6A4CDB3Q36294186-125C01D8-7EF0-4573-98F7-950EC66802C9Q36736774-41A59E35-1353-42A0-9BD1-BC5D0C9B875BQ37241711-72C6ADB2-FA8A-44A0-92AB-988284D33EE6Q37254393-059D1877-0177-498F-9083-007DC85B91EBQ38100859-7FC57CF3-FE32-403B-80A6-78AD55B0E34CQ38631755-576986B2-F76F-4D1D-B198-949C53CC129FQ38799103-41626845-0FF7-4F16-B067-AB35A7600935Q38849926-A01576CB-FACF-4569-9DF8-FF31D2C66554Q38865828-DACD266D-ED28-4389-A9D0-7F1BFC8479EDQ38943928-F2DC51C0-84DC-48CA-9E98-F005A18C8745Q39441281-42EC92AB-D845-4CC9-9498-51E0B0A9FEC6Q40046642-2D7E2A47-6425-4E42-B621-CB944EF707CAQ40113142-5B1D2696-77E9-4D3E-8B19-1BDDD324D7B8Q40116028-F6786E41-428A-4B5E-BB4B-B93EC6974982Q40120026-6BBAAE6C-4FCC-4CA8-8FB9-A33C2099882CQ40125358-2D487E37-0749-4FC7-A16B-5B969EF4CB53Q40207035-1A43BB82-C75F-4F82-95F9-DB83929DCE0AQ42222047-B24E5414-7F0D-4BD4-8B7D-BA8BACADE64BQ45324762-034A0B1D-D076-43FD-878F-3AFD155263FFQ47259217-40751E25-1674-4FB9-9316-EA481D9E32E3Q54216760-6757ADBC-6AC7-4C54-A488-AD8AC3E8D549Q56888557-510A7D77-B30E-4182-AF60-5DE3FDDA10EA
P2860
The cellular ataxia telangiectasia-mutated kinase promotes epstein-barr virus lytic reactivation in response to multiple different types of lytic reactivation-inducing stimuli
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
The cellular ataxia telangiect ...... reactivation-inducing stimuli
@ast
The cellular ataxia telangiect ...... reactivation-inducing stimuli
@en
type
label
The cellular ataxia telangiect ...... reactivation-inducing stimuli
@ast
The cellular ataxia telangiect ...... reactivation-inducing stimuli
@en
prefLabel
The cellular ataxia telangiect ...... reactivation-inducing stimuli
@ast
The cellular ataxia telangiect ...... reactivation-inducing stimuli
@en
P2093
P2860
P356
P1433
P1476
The cellular ataxia telangiect ...... reactivation-inducing stimuli
@en
P2093
Elizabeth A Barlow
Shannon C Kenney
Stacy R Hagemeier
P2860
P304
13360-13370
P356
10.1128/JVI.01850-12
P407
P577
2012-09-26T00:00:00Z