Anorexia/cachexia of chronic diseases: a role for the TGF-β family cytokine MIC-1/GDF15.
about
Molecular pathways leading to loss of skeletal muscle mass in cancer cachexia--can findings from animal models be translated to humans?The anorectic actions of the TGFβ cytokine MIC-1/GDF15 require an intact brainstem area postrema and nucleus of the solitary tractMuscle wasting: an overview of recent developments in basic research.DNA from dead cancer cells induces TLR9-mediated invasion and inflammation in living cancer cellsMIC-1/GDF15 in Barrett's oesophagus and oesophageal adenocarcinomaMacrophage inhibitory cytokine-1 (MIC-1/GDF15) gene deletion promotes cancer growth in TRAMP prostate cancer prone miceSerum Levels of Human MIC-1/GDF15 Vary in a Diurnal Pattern, Do Not Display a Profile Suggestive of a Satiety Factor and Are Related to BMI.Plasma growth differentiation factor 15 is associated with weight loss and mortality in cancer patients.Pancreatic cancer cell-derived IGFBP-3 contributes to muscle wastingMAP3K11/GDF15 axis is a critical driver of cancer cachexiaGrowth differentiation factor-15 is associated with muscle mass in chronic obstructive pulmonary disease and promotes muscle wasting in vivo.GFRAL is the receptor for GDF15 and the ligand promotes weight loss in mice and nonhuman primates.Cachexia in patients with oesophageal cancer.Anorexia-cachexia and obesity treatment may be two sides of the same coin: role of the TGF-b superfamily cytokine MIC-1/GDF15.Muscle wasting in ageing and chronic illness.Association between GDF-15 levels and changes in vascular and physical function in older patients with hypertension.Relationship between efficacy outcomes and weight gain during treatment of advanced, non-squamous, non-small-cell lung cancer patients.Cancer cachexia-when proteasomal inhibition is not enough.Anorexia of ageing: a key component in the pathogenesis of both sarcopenia and cachexia.A New Transgenic Mouse Model of Heart Failure and Cardiac Cachexia Raised by Sustained Activation of Met Tyrosine Kinase in the Heart.Treatment with the TGF-b superfamily cytokine MIC-1/GDF15 reduces the adiposity and corrects the metabolic dysfunction of mice with diet-induced obesity.New extracellular factors in glioblastoma multiforme development: neurotensin, growth differentiation factor-15, sphingosine-1-phosphate and cytomegalovirus infection.
P2860
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P2860
Anorexia/cachexia of chronic diseases: a role for the TGF-β family cytokine MIC-1/GDF15.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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2012年學術文章
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name
Anorexia/cachexia of chronic diseases: a role for the TGF-β family cytokine MIC-1/GDF15.
@ast
Anorexia/cachexia of chronic diseases: a role for the TGF-β family cytokine MIC-1/GDF15.
@en
type
label
Anorexia/cachexia of chronic diseases: a role for the TGF-β family cytokine MIC-1/GDF15.
@ast
Anorexia/cachexia of chronic diseases: a role for the TGF-β family cytokine MIC-1/GDF15.
@en
prefLabel
Anorexia/cachexia of chronic diseases: a role for the TGF-β family cytokine MIC-1/GDF15.
@ast
Anorexia/cachexia of chronic diseases: a role for the TGF-β family cytokine MIC-1/GDF15.
@en
P2093
P2860
P1476
Anorexia/cachexia of chronic diseases: a role for the TGF-β family cytokine MIC-1/GDF15.
@en
P2093
David A Brown
Hong Ping Zhang
K K Michelle Lee-Ng
Kate Harriott
Lele Jiang
Rakesh Manandhar
Samuel N Breit
Vicky W W Tsai
Yasmin Husaini
P2860
P2888
P304
P356
10.1007/S13539-012-0082-6
P577
2012-08-31T00:00:00Z