Spinal cord injury induces serotonin supersensitivity without increasing intrinsic excitability of mouse V2a interneurons.
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Serotonergic regulation of excitability of principal cells of the dorsal cochlear nucleus.Examination of the combined effects of chondroitinase ABC, growth factors and locomotor training following compressive spinal cord injury on neuroanatomical plasticity and kinematics.Aberrant crossed corticospinal facilitation in muscles distant from a spinal cord injury.The role of the serotonergic system in locomotor recovery after spinal cord injuryAdult spinal V2a interneurons show increased excitability and serotonin-dependent bistabilityCompensatory plasticity restores locomotion after chronic removal of descending projections.A dystonia-like movement disorder with brain and spinal neuronal defects is caused by mutation of the mouse laminin β1 subunit, Lamb1.Synthesis, transport, and metabolism of serotonin formed from exogenously applied 5-HTP after spinal cord injury in rats.Serotonergic transmission after spinal cord injury.Neuromodulation of neurons and synapses.Removal of endogenous neuromodulators in a small motor network enhances responsiveness to neuromodulation.Stem cells for spinal cord injury: Strategies to inform differentiation and transplantation.Rehabilitation Strategies after Spinal Cord Injury: Inquiry into the Mechanisms of Success and Failure.A short history of the 5-HT2C receptor: from the choroid plexus to depression, obesity and addiction treatment.Spatiotemporal correlation of spinal network dynamics underlying spasms in chronic spinalized mice.Constitutive activity of 5-HT2C receptors is present after incomplete spinal cord injury but is not modified after chronic SSRI or baclofen treatment.Retracing your footsteps: developmental insights to spinal network plasticity following injury.Serotonin receptor and dendritic plasticity in the spinal cord mediated by chronic serotonergic pharmacotherapy combined with exercise following complete SCI in the adult rat.Locomotor recovery following contusive spinal cord injury does not require oligodendrocyte remyelination
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P2860
Spinal cord injury induces serotonin supersensitivity without increasing intrinsic excitability of mouse V2a interneurons.
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-my
2012年学术文章
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2012年學術文章
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name
Spinal cord injury induces ser ...... ity of mouse V2a interneurons.
@ast
Spinal cord injury induces ser ...... ity of mouse V2a interneurons.
@en
type
label
Spinal cord injury induces ser ...... ity of mouse V2a interneurons.
@ast
Spinal cord injury induces ser ...... ity of mouse V2a interneurons.
@en
prefLabel
Spinal cord injury induces ser ...... ity of mouse V2a interneurons.
@ast
Spinal cord injury induces ser ...... ity of mouse V2a interneurons.
@en
P2093
P2860
P1476
Spinal cord injury induces ser ...... lity of mouse V2a interneurons
@en
P2093
Andreas Husch
Diana N Hong
Gabrielle N Van Patten
Moira M Scaperotti
P2860
P304
13145-13154
P356
10.1523/JNEUROSCI.2995-12.2012
P407
P577
2012-09-01T00:00:00Z