Down-regulation of 14-3-3zeta suppresses anchorage-independent growth of lung cancer cells through anoikis activation - Wikidata - awgldk - TriplyDB

Down-regulation of 14-3-3zeta suppresses anchorage-independent growth of lung cancer cells through anoikis activation

Down-regulation of 14-3-3zeta suppresses anchorage-independent growth of lung cancer cells through anoikis activation

http://www.wikidata.org/entity/Q36423378

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Proteomic dissection of cell type-specific H2AX-interacting protein complex associated with hepatocellular carcinoma Rsk-mediated phosphorylation and 14-3-3ɛ binding of Apaf-1 suppresses cytochrome c-induced apoptosis A novel binding factor of 14-3-3beta functions as a transcriptional repressor and promotes anchorage-independent growth, tumorigenicity, and metastasis The interaction between casein kinase Ialpha and 14-3-3 is phosphorylation dependent 14-3-3 interacts with LKB1 via recognizing phosphorylated threonine 336 residue and suppresses LKB1 kinase function miR-451 protects against erythroid oxidant stress by repressing 14-3-3zeta 14-3-3zeta mediates resistance of diffuse large B cell lymphoma to an anthracycline-based chemotherapeutic regimen The role of the myofibroblast in tumor stroma remodeling Involvement of 14-3-3 Proteins in Regulating Tumor Progression of Hepatocellular Carcinoma Metabolic-stress-induced rearrangement of the 14-3-3ζ interactome promotes autophagy via a ULK1- and AMPK-regulated 14-3-3ζ interaction with phosphorylated Atg9 Three-way interaction between 14-3-3 proteins, the N-terminal region of tyrosine hydroxylase, and negatively charged membranes 14-3-3 isoforms bind directly exon B of the 5'-UTR of human surfactant protein A2 mRNA 14-3-3 zeta as novel molecular target for cancer therapy Overexpression of YWHAZ relates to tumor cell proliferation and malignant outcome of gastric carcinoma Barium promotes anchorage-independent growth and invasion of human HaCaT keratinocytes via activation of c-SRC kinase Proteomic analysis of apoptosis induction in human lung cancer cells by recombinant MVL.A network-based biomarker approach for molecular investigation and diagnosis of lung cancer.The anoikis effector Bit1 displays tumor suppressive function in lung cancer cells.A cancer-related protein 14-3-3ζ is a potential tumor-associated antigen in immunodiagnosis of hepatocellular carcinoma.Pro-oncogenic function of HIP-55/Drebrin-like (DBNL) through Ser269/Thr291-phospho-sensor motifs.14-3-3ζ as a prognostic marker and therapeutic target for cancer Profilin-1 overexpression in MDA-MB-231 breast cancer cells is associated with alterations in proteomics biomarkers of cell proliferation, survival, and motility as revealed by global proteomics analyses.14-3-3ε overexpression contributes to epithelial-mesenchymal transition of hepatocellular carcinoma.Proteomic analysis of differentially expressed proteins in peripheral cholangiocarcinoma.Cyclin D1 promotes anchorage-independent cell survival by inhibiting FOXO-mediated anoikis.A time-resolved fluorescence resonance energy transfer assay for high-throughput screening of 14-3-3 protein-protein interaction inhibitors Altered expression of 14-3-3ζ protein in spinal cords of rat fetuses with spina bifida aperta Transcriptional regulation of YWHAZ, the gene encoding 14-3-3ζ.Tamoxifen downregulation of miR-451 increases 14-3-3ζ and promotes breast cancer cell survival and endocrine resistance.14-3-3 proteins as potential therapeutic targets.Direct interaction between scaffolding proteins RACK1 and 14-3-3ζ regulates brain-derived neurotrophic factor (BDNF) transcription Increased expression of 14-3-3β promotes tumor progression and predicts extrahepatic metastasis and worse survival in hepatocellular carcinoma.14-3-3 fusion oncogenes in high-grade endometrial stromal sarcoma.Destabilisation of dimeric 14-3-3 proteins as a novel approach to anti-cancer therapeutics.Regulation of aldo-keto-reductase family 1 B10 by 14-3-3ε and their prognostic impact of hepatocellular carcinoma.The presence of HIV-1 Tat protein second exon delays fas protein-mediated apoptosis in CD4+ T lymphocytes: a potential mechanism for persistent viral production 14-3-3ζ as a predictor of early time to recurrence and distant metastasis in hormone receptor-positive and -negative breast cancers.14-3-3zeta overexpression defines high risk for breast cancer recurrence and promotes cancer cell survival.The 14-3-3 proteins: integrators of diverse signaling cues that impact cell fate and cancer development.14-3-3 Protein regulates cell adhesion in the seminiferous epithelium of rat testes.

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Down-regulation of 14-3-3zeta suppresses anchorage-independent growth of lung cancer cells through anoikis activation

http://www.wikidata.org/entity/Q36423378

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Down-regulation of 14-3-3zeta ...... lls through anoikis activation

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Down-regulation of 14-3-3zeta ...... lls through anoikis activation

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Down-regulation of 14-3-3zeta ...... lls through anoikis activation

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Down-regulation of 14-3-3zeta ...... lls through anoikis activation

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Down-regulation of 14-3-3zeta ...... lls through anoikis activation

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PNAS.0710905105

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Proceedings of the National Academy of Sciences of the United States of America

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Down-regulation of 14-3-3zeta ...... lls through anoikis activation

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Alastair Aitken

http://www.w3.org/2001/XMLSchema#string

Fadlo R Khuri

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Hae Ryoun Park

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Haian Fu

http://www.w3.org/2001/XMLSchema#string

Jing Zhao

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Leon Bernal-Mizrachi

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Shi-Yong Sun

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Yuhong Du

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Zenggang Li

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P2860

The Hallmarks of Cancer

Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function

Comprehensive proteomic analysis of interphase and mitotic 14-3-3-binding proteins

14-3-3 is not essential for Raf-1 function: identification of Raf-1 proteins that are biologically activated in a 14-3-3- and Ras-independent manner

Interaction of 14-3-3 with signaling proteins is mediated by the recognition of phosphoserine

Activation of Raf-1 by 14-3-3 proteins

Interaction of the protein kinase Raf-1 with 14-3-3 proteins

AKT/PKB signaling: navigating downstream

14-3-3epsilon is important for neuronal migration by binding to NUDEL: a molecular explanation for Miller-Dieker syndrome

Efp targets 14-3-3 sigma for proteolysis and promotes breast tumour growth

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162-167

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10.1073/PNAS.0710905105

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105

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2007-12-27T00:00:00Z

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18162532

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2224179

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