t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway. - Wikidata - awgldk - TriplyDB

t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway.

t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway.

http://www.wikidata.org/entity/Q36423956

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Indigofera oblongifolia Prevents Lead Acetate-Induced Hepatotoxicity, Oxidative Stress, Fibrosis and Apoptosis in Rats.Proanthocyanidins Attenuation of Chronic Lead-Induced Liver Oxidative Damage in Kunming Mice via the Nrf2/ARE Pathway.The toxic influence of dibromoacetic acid on the hippocampus and pre-frontal cortex of rat: involvement of neuroinflammation response and oxidative stress.Myricitrin Attenuates High Glucose-Induced Apoptosis through Activating Akt-Nrf2 Signaling in H9c2 Cardiomyocytes.Neuroprotective effect of 5-aminolevulinic acid against low inorganic phosphate in neuroblastoma SH-SY5Y cells.Role of free fatty acids in endothelial dysfunction.Lycium barbarum polysaccharide protects against neurotoxicity via the Nrf2-HO-1 pathway.Nrf2 induces cisplatin resistance via suppressing the iron export related gene SLC40A1 in ovarian cancer cells.Tert-butylhydroquinone post-treatment attenuates neonatal hypoxic-ischemic brain damage in rats.Neurotoxic Agent-Induced Injury in Neurodegenerative Disease Model: Focus on Involvement of Glutamate Receptors Heme Oxygenase 1 in the Nervous System: Does It Favor Neuronal Cell Survival or Induce Neurodegeneration?

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t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway.

http://www.wikidata.org/entity/Q36423956

description

2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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2015年學術文章

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2015年學術文章

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t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway.

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t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway.

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t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway.

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t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway.

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t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway.

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t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway.

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Oxidative medicine and cellular longevity

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t-BHQ Provides Protection against Lead Neurotoxicity via Nrf2/HO-1 Pathway.

@en

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Fang Ye

http://www.w3.org/2001/XMLSchema#string

Jing Yuan

http://www.w3.org/2001/XMLSchema#string

Jun Chen

http://www.w3.org/2001/XMLSchema#string

Lili Li

http://www.w3.org/2001/XMLSchema#string

Xiaoyi Li

http://www.w3.org/2001/XMLSchema#string

P2860

BTB protein Keap1 targets antioxidant transcription factor Nrf2 for ubiquitination by the Cullin 3-Roc1 ligase

Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain

Antioxidant enzymes and human diseases

Differential regulation of glutamate-cysteine ligase subunit expression and increased holoenzyme formation in response to cysteine deprivation

Through metal binding, curcumin protects against lead- and cadmium-induced lipid peroxidation in rat brain homogenates and against lead-induced tissue damage in rat brain.

Oxidative stress and apoptotic changes in primary cultures of rat proximal tubular cells exposed to lead.

Zeaxanthin induces Nrf2-mediated phase II enzymes in protection of cell death.

Effects of antioxidant enzymes in the molecular control of reactive oxygen species toxicology.

Lead neurotoxicity in children: basic mechanisms and clinical correlates.

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2075915

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scholarly article

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10.1155/2016/2075915

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2016

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2015-12-21T00:00:00Z

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287810826

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