A metabolomic perspective of griseofulvin-induced liver injury in mice.
about
Ferrochelatase is a therapeutic target for ocular neovascularization.Griseofulvin impairs intraerythrocytic growth of Plasmodium falciparum through ferrochelatase inhibition but lacks activity in an experimental human infection study.Chronic Treatment with Isoniazid Causes Protoporphyrin IX Accumulation in Mouse Liver.The Opportunities of Metabolomics in Drug Safety Evaluation.
P2860
A metabolomic perspective of griseofulvin-induced liver injury in mice.
description
2015 nî lūn-bûn
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name
A metabolomic perspective of griseofulvin-induced liver injury in mice.
@ast
A metabolomic perspective of griseofulvin-induced liver injury in mice.
@en
type
label
A metabolomic perspective of griseofulvin-induced liver injury in mice.
@ast
A metabolomic perspective of griseofulvin-induced liver injury in mice.
@en
prefLabel
A metabolomic perspective of griseofulvin-induced liver injury in mice.
@ast
A metabolomic perspective of griseofulvin-induced liver injury in mice.
@en
P2093
P2860
P1476
A metabolomic perspective of griseofulvin-induced liver injury in mice
@en
P2093
P2860
P304
P356
10.1016/J.BCP.2015.09.002
P407
P577
2015-09-05T00:00:00Z