Decreased age-related cardiac dysfunction, myocardial nitrative stress, inflammatory gene expression, and apoptosis in mice lacking fatty acid amide hydrolase.
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The endocannabinoid system in normal and pathological brain ageingMeasurement of cardiac function using pressure-volume conductance catheter technique in mice and ratsThe endocannabinoid/endovanilloid N-arachidonoyl dopamine (NADA) and synthetic cannabinoid WIN55,212-2 abate the inflammatory activation of human endothelial cellsEnzymatic pathways that regulate endocannabinoid signaling in the nervous systemCerebral microhemorrhages: mechanisms, consequences, and prevention.Lipid droplets are novel sites of N-acylethanolamine inactivation by fatty acid amide hydrolase-2.Targeting cannabinoid signaling for peritoneal dialysis-induced oxidative stress and fibrosis.Age-dependent alterations in fibrillar collagen content and myocardial diastolic function: role of SPARC in post-synthetic procollagen processing.The endocannabinoid system: 'NO' longer anonymous in the control of nitrergic signalling?CB2 receptor activation ameliorates the proinflammatory activity in acute lung injury induced by paraquat.Cannabinoid-2 receptor limits inflammation, oxidative/nitrosative stress, and cell death in nephropathy.Cardioprotection and ageingThe cannabinoid WIN55,212-2 protects against oxidized LDL-induced inflammatory response in murine macrophages.Mechanisms of vascular aging: new perspectives.A heart that beats for 500 years: age-related changes in cardiac proteasome activity, oxidative protein damage and expression of heat shock proteins, inflammatory factors, and mitochondrial complexes in Arctica islandica, the longest-living noncolonMathematical modeling of left ventricular dimensional changes in mice during aging.Role of endocannabinoids and cannabinoid-1 receptors in cerebrocortical blood flow regulation.Myofilament calcium sensitization delays decompensated hypertrophy differently between the sexes following myocardial infarctionCB2 cannabinoid receptor agonist JWH-015 modulates human monocyte migration through defined intracellular signaling pathways.Endocannabinoid system in cardiovascular disorders - new pharmacotherapeutic opportunities.Cannabinoid receptors in acute and chronic complications of atherosclerosis.Pleiotropic effects of the CB2 cannabinoid receptor activation on human monocyte migration: implications for atherosclerosis and inflammatory diseases.Age-related cardiac muscle sarcopenia: Combining experimental and mathematical modeling to identify mechanisms.Endocannabinoids and liver disease. III. Endocannabinoid effects on immune cells: implications for inflammatory liver diseasesInfant birth weight and third trimester maternal plasma markers of vascular integrity: the MIREC study.Testing hypotheses of aging in long-lived mice of the genus Peromyscus: association between longevity and mitochondrial stress resistance, ROS detoxification pathways, and DNA repair efficiencyVasoprotective effects of resveratrol and SIRT1: attenuation of cigarette smoke-induced oxidative stress and proinflammatory phenotypic alterationsThe role of the endocannabinoid system in atherosclerosis.Cannabinoid receptor CB2 protects against balloon-induced neointima formation.Role of superoxide, nitric oxide, and peroxynitrite in doxorubicin-induced cell death in vivo and in vitroEndocannabinoids and liver disease. V. endocannabinoids as mediators of vascular and cardiac abnormalities in cirrhosis.Oxidative stress and accelerated vascular aging: implications for cigarette smoking.Loss of cardioprotection with ageing.The case for the development of novel analgesic agents targeting both fatty acid amide hydrolase and either cyclooxygenase or TRPV1.Endocannabinoids and the heart.The emerging role of the endocannabinoid system in cardiovascular disease.Cardiac Physiology of Aging: Extracellular Considerations.Seeing through the smoke: Human and animal studies of cannabis use and endocannabinoid signalling in corticolimbic networks.Role of TFEB Mediated Autophagy, Oxidative Stress, Inflammation, and Cell Death in Endotoxin Induced Myocardial Toxicity of Young and Aged Mice.Activation of Endocannabinoid Receptor 2 as a Mechanism of Propofol Pretreatment-Induced Cardioprotection against Ischemia-Reperfusion Injury in Rats
P2860
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P2860
Decreased age-related cardiac dysfunction, myocardial nitrative stress, inflammatory gene expression, and apoptosis in mice lacking fatty acid amide hydrolase.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Decreased age-related cardiac ...... ng fatty acid amide hydrolase.
@ast
Decreased age-related cardiac ...... ng fatty acid amide hydrolase.
@en
type
label
Decreased age-related cardiac ...... ng fatty acid amide hydrolase.
@ast
Decreased age-related cardiac ...... ng fatty acid amide hydrolase.
@en
prefLabel
Decreased age-related cardiac ...... ng fatty acid amide hydrolase.
@ast
Decreased age-related cardiac ...... ng fatty acid amide hydrolase.
@en
P2093
P2860
P50
P1476
Decreased age-related cardiac ...... ng fatty acid amide hydrolase.
@en
P2093
Anna Csiszár
György Haskó
Lucas Liaudet
Mohanraj Rajesh
Sándor Bátkai
Zoltan Ungvári
P2860
P304
P356
10.1152/AJPHEART.00373.2007
P577
2007-04-13T00:00:00Z