Signals for nausea and emesis: Implications for models of upper gastrointestinal diseases.
about
Why can't rodents vomit? A comparative behavioral, anatomical, and physiological studyWhy is the neurobiology of nausea and vomiting so important?Cannabinoids As Potential Treatment for Chemotherapy-Induced Nausea and VomitingFunctional organization of autonomic neural pathwaysRole of central vagal 5-HT3 receptors in gastrointestinal physiology and pathophysiologyAmylin modulates the mesolimbic dopamine system to control energy balance.Investigating the effect of emetic compounds on chemotaxis in Dictyostelium identifies a non-sentient model for bitter and hot tastant researchRegulation of nausea and vomiting by cannabinoids and the endocannabinoid systemFood restriction, refeeding, and gastric fill fail to affect emesis in musk shrews.Chemotherapy agent cisplatin induces 48-h Fos expression in the brain of a vomiting species, the house musk shrew (Suncus murinus).Zebrafish: an emerging technology for in vivo pharmacological assessment to identify potential safety liabilities in early drug discovery.Synthetic and natural antioxidants attenuate cisplatin-induced vomitingTNFalpha: a trigger of autonomic dysfunctionRotavirus stimulates release of serotonin (5-HT) from human enterochromaffin cells and activates brain structures involved in nausea and vomitingAssessment of feeding behavior in laboratory miceHindbrain nucleus tractus solitarius glucagon-like peptide-1 receptor signaling reduces appetitive and motivational aspects of feeding.Parallel reinforcement pathways for conditioned food aversions in the honeybee.A comparison between taste avoidance and conditioned disgust reactions induced by ethanol and lithium chloride in preweanling rats.Characterization of deoxynivalenol-induced anorexia using mouse bioassay.Comparison of anorectic and emetic potencies of deoxynivalenol (vomitoxin) to the plant metabolite deoxynivalenol-3-glucoside and synthetic deoxynivalenol derivatives EN139528 and EN139544.Behavioral patterns associated with chemotherapy-induced emesis: a potential signature for nausea in musk shrews.Incretins and amylin: neuroendocrine communication between the gut, pancreas, and brain in control of food intake and blood glucose.GLP-1 neurons in the nucleus of the solitary tract project directly to the ventral tegmental area and nucleus accumbens to control for food intake.Brain Fos expression during 48 h after cisplatin treatment: neural pathways for acute and delayed visceral sickness.Computerized detection and analysis of cancer chemotherapy-induced emesis in a small animal model, musk shrew.Glutamate Receptors in the Central Nucleus of the Amygdala Mediate Cisplatin-Induced Malaise and Energy Balance Dysregulation through Direct Hindbrain Projections.Predicting the emetic liability of novel chemical entities: a comparative studyVarenicline for smoking cessation: nausea severity and variation in nicotinic receptor genes.Galantamine, an acetylcholinesterase inhibitor and positive allosteric modulator of nicotinic acetylcholine receptors, attenuates nicotine taking and seeking in ratsEndocannabinoid regulation of nausea is mediated by 2-arachidonoylglycerol (2-AG) in the rat visceral insular cortex.Comparison of emetic potencies of the 8-ketotrichothecenes deoxynivalenol, 15-acetyldeoxynivalenol, 3-acetyldeoxynivalenol, fusarenon X, and nivalenol.Responsiveness to nicotine of neurons of the caudal nucleus of the solitary tract correlates with the neuronal projection target.CXCL12 sensitizes vago-vagal reflex neurons in the dorsal medullaIncreased liking for a solution is not necessary for the attenuation of neophobia in rats.Peptide YY3-36 and 5-hydroxytryptamine mediate emesis induction by trichothecene deoxynivalenol (vomitoxin).Citrus aurantium and Rhodiola rosea in combination reduce visceral white adipose tissue and increase hypothalamic norepinephrine in a rat model of diet-induced obesityDonepezil, an acetylcholinesterase inhibitor, attenuates nicotine self-administration and reinstatement of nicotine seeking in rats.Opportunities for the replacement of animals in the study of nausea and vomitingChemotherapy-induced kaolin intake is increased by lesion of the lateral parabrachial nucleus of the rat.Musk shrews selectively bred for motion sickness display increased anesthesia-induced vomiting.
P2860
Q21090773-6E71F0A7-17EC-4CAA-8AB3-D9526D4032DAQ24649937-6C7DEAE2-565C-4D03-A5C2-874FB46E0F09Q26738288-675847FA-8E4E-45F9-9485-CE4AB5DA3D1FQ26830195-C326B2E2-C94F-42C2-A902-D155819870A9Q26852714-CB6ACB77-6875-402D-B965-C74AF148E9D3Q27311258-FE45EDAE-DFAE-41A6-B781-E95C9553E2F4Q27321366-C0A40B81-1AFD-4142-A14A-EA14ED660DDDQ28301264-324F1FCD-04DF-4178-A14C-C3372E472754Q30433586-E5D47777-1B17-48F3-ACDD-2BC32150CE0BQ30436557-91DC674A-1261-4AA5-9F10-C2112021B743Q30487697-57CBC4E0-FC01-49BB-A660-5D139D3B1738Q30834914-EACD02BD-D09E-46A6-88F1-CE8AD1A075A7Q33155571-30CD8826-0CE1-4873-AD67-994E88340572Q33967946-D854962E-CDC7-4883-95CE-0EDD02D93A22Q34046745-08626C82-9696-4372-B8AA-37F0A76136A4Q34062928-FFDCC825-EDF5-4AFD-9FDD-2D225983D802Q34444350-71833BCC-A4FB-459C-B671-C4D49E89F52CQ34666371-4E28E243-1FCC-4419-BB9B-55BF392B6A6EQ35070893-40A0022B-2DD4-429A-941F-48C73397D389Q35102616-76CDC8C7-6158-4C60-B301-CFE833EDD297Q35114037-9629E0DD-EB1E-4C55-98F3-2B4327ADFB99Q35690970-0132FDBF-247E-47C0-8C18-7C0AA25D1D0BQ35744558-7A31B7E0-2204-45AC-B730-746E0BF56478Q35781990-5E220B13-6953-4BAF-8C49-C21EB0B3855AQ35871181-858E0755-E36C-42D9-89BF-DF75035903FCQ35916433-B36E9745-9F45-4CC8-AB14-F99BF3FA2C67Q36025372-8C91601B-D358-46E0-8421-74BFF24DED98Q36118485-471CD2A5-6C83-41F9-BBCF-B28D6711763DQ36174102-2781D8AB-0515-4E1A-82AB-7D4AC0C50EFAQ36421863-9BB9EB19-7448-41F1-90E4-AC0E3A9435F6Q36509050-6083F774-0573-4905-B0D5-1100FA9B07D3Q36532483-481129C8-FE6E-4B32-82C3-ABBF63092AA8Q36635641-B553446F-5CB2-45B4-90A0-08BDB6648439Q36665830-A664B9C2-FAF2-4C5B-86BA-8D5E6B9E0556Q36769372-DA385026-0920-47D8-BEEB-BFE9C828FF2BQ37254830-096E2A04-76F6-4A4C-9874-6B1CE5286131Q37282357-9A4241EA-946B-4DB6-B03F-F6F119B33C97Q37332618-B9064574-A0F9-43AF-9BCB-2A2F25D8E8EFQ37422065-55FF1F41-D2C4-41A8-ABFA-0ECF80A54CE0Q37460036-A0C5814B-F21E-48D1-9A3D-11E2EE911776
P2860
Signals for nausea and emesis: Implications for models of upper gastrointestinal diseases.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
Signals for nausea and emesis: ...... per gastrointestinal diseases.
@ast
Signals for nausea and emesis: ...... per gastrointestinal diseases.
@en
type
label
Signals for nausea and emesis: ...... per gastrointestinal diseases.
@ast
Signals for nausea and emesis: ...... per gastrointestinal diseases.
@en
prefLabel
Signals for nausea and emesis: ...... per gastrointestinal diseases.
@ast
Signals for nausea and emesis: ...... per gastrointestinal diseases.
@en
P2860
P1476
Signals for nausea and emesis: Implications for models of upper gastrointestinal diseases
@en
P2860
P304
P356
10.1016/J.AUTNEU.2006.01.008
P577
2006-03-23T00:00:00Z