The repertoire of CD4+ CD28- T cells in rheumatoid arthritis.
about
Aging, autoimmunity and arthritis: T-cell senescence and contraction of T-cell repertoire diversity - catalysts of autoimmunity and chronic inflammationThe expansion of CD4+CD28- T cells in patients with rheumatoid arthritisRheumatoid arthritis is an independent risk factor for multi-vessel coronary artery disease: a case control studyThe story of CD4+ CD28- T cells revisited: solved or still ongoing?Human IgG1 antibodies antagonizing activating receptor NKG2D on natural killer cellsPeripheral and site-specific CD4(+) CD28(null) T cells from rheumatoid arthritis patients show distinct characteristics.Implication of gammadelta T cells in the human immune response to cytomegalovirusImmune aging and rheumatoid arthritis.Diagnostic role of tests for T cell receptor (TCR) genesPeripheral CD4CD8 double positive T cells with a distinct helper cytokine profile are increased in rheumatoid arthritis.Synergy or independence? Deciphering the interaction of HLA Class I and NK cell KIR alleles in early HIV-1 disease progression.Uncoupling of T-cell effector functions by inhibitory killer immunoglobulin-like receptors.Selective activation of the c-Jun NH2-terminal protein kinase signaling pathway by stimulatory KIR in the absence of KARAP/DAP12 in CD4+ T cells.Skewed distribution of proinflammatory CD4+CD28null T cells in rheumatoid arthritis.Association of Elevations of Specific T Cell and Monocyte Subpopulations in Rheumatoid Arthritis With Subclinical Coronary Artery AtherosclerosisCardiovascular co-morbidity in rheumatic diseases.The janus head of T cell aging - autoimmunity and immunodeficiency.T cells in rheumatoid arthritis.Heat shock proteins 60 and 70 specific proinflammatory and cytotoxic response of CD4+CD28null cells in chronic kidney disease.Persistent arthritis in Borrelia burgdorferi-infected HLA-DR4-positive CD28-negative mice post-antibiotic treatment.Developments in the scientific understanding of rheumatoid arthritis.Expansions of Cytotoxic CD4+CD28- T Cells Drive Excess Cardiovascular Mortality in Rheumatoid Arthritis and Other Chronic Inflammatory Conditions and Are Triggered by CMV Infection.The life (and death) of CD4+ CD28(null) T cells in inflammatory diseases.DNA damage, metabolism and aging in pro-inflammatory T cells: Rheumatoid arthritis as a model system.CD4+CD28null T Lymphocytes are Associated with the Development of Atrial Fibrillation after Elective Cardiac Surgery.The host cellular immune response to cytomegalovirus targets the endothelium and is associated with increased arterial stiffness in ANCA-associated vasculitisCD28 pro-atherogenic CD4 T-cells explain the link between CMV infection and an increased risk of cardiovascular death
P2860
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P2860
The repertoire of CD4+ CD28- T cells in rheumatoid arthritis.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
The repertoire of CD4+ CD28- T cells in rheumatoid arthritis.
@ast
The repertoire of CD4+ CD28- T cells in rheumatoid arthritis.
@en
type
label
The repertoire of CD4+ CD28- T cells in rheumatoid arthritis.
@ast
The repertoire of CD4+ CD28- T cells in rheumatoid arthritis.
@en
prefLabel
The repertoire of CD4+ CD28- T cells in rheumatoid arthritis.
@ast
The repertoire of CD4+ CD28- T cells in rheumatoid arthritis.
@en
P2093
P2860
P1433
P1476
The repertoire of CD4+ CD28- T cells in rheumatoid arthritis.
@en
P2093
C M Weyand
J J Goronzy
P B Martens
P2860
P304
P407
P577
1996-09-01T00:00:00Z