Lipopolysaccharide potentiates polychlorinated biphenyl-induced disruption of the blood-brain barrier via TLR4/IRF-3 signaling
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Contributions of nonhematopoietic cells and mediators to immune responses: implications for immunotoxicologyTLR4 signaling is involved in brain vascular toxicity of PCB153 bound to nanoparticles.Melatonin protects against blood-brain barrier damage by inhibiting the TLR4/ NF-κB signaling pathway after LPS treatment in neonatal rats.Exercise attenuates PCB-induced changes in the mouse gut microbiome.Lipid rafts regulate PCB153-induced disruption of occludin and brain endothelial barrier function through protein phosphatase 2A and matrix metalloproteinase-2Does generalized hypo-oxygenation (hypoxia) allow endotoxin into the brain through the blood brain barrier, thus increasing the risk for Parkinson disease?2,3',4,4',5-Pentachlorobiphenyl impairs insulin-induced NO production partly through excessive ROS production in endothelial cells.PCB 118-induced endothelial cell apoptosis is partially mediated by excessive ROS production.Intestinal exposure to PCB 153 induces inflammation via the ATM/NEMO pathway.
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P2860
Lipopolysaccharide potentiates polychlorinated biphenyl-induced disruption of the blood-brain barrier via TLR4/IRF-3 signaling
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Lipopolysaccharide potentiates ...... rrier via TLR4/IRF-3 signaling
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Lipopolysaccharide potentiates ...... rrier via TLR4/IRF-3 signaling
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Lipopolysaccharide potentiates ...... rrier via TLR4/IRF-3 signaling
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Lipopolysaccharide potentiates ...... rrier via TLR4/IRF-3 signaling
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Lipopolysaccharide potentiates ...... rrier via TLR4/IRF-3 signaling
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Lipopolysaccharide potentiates ...... rrier via TLR4/IRF-3 signaling
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Lipopolysaccharide potentiates ...... rrier via TLR4/IRF-3 signaling
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Jeong June Choi
Michal Toborek
Sung Yong Eum
Yean Jung Choi
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P304
P356
10.1016/J.TOX.2012.08.001
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2012-08-13T00:00:00Z