Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice - Wikidata - awgldk - TriplyDB

Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice

Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice

http://www.wikidata.org/entity/Q36438370

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Contribution of nitric oxide synthases 1, 2, and 3 to airway hyperresponsiveness and inflammation in a murine model of asthma Aspergillus antigen induces robust Th2 cytokine production, inflammation, airway hyperreactivity and fibrosis in the absence of MCP-1 or CCR2 An immune basis for lung parenchymal destruction in chronic obstructive pulmonary disease and emphysema Environmental contributions to the allergic asthma epidemic.Identification of antigenic epitopes on human allergens: studies with HLA transgenic mice Challenges and Current Efforts in the Development of Biomarkers for Chronic Inflammatory and Remodeling Conditions of the Lungs Airway fibrinogenolysis and the initiation of allergic inflammation NO2 inhalation induces maturation of pulmonary CD11c+ cells that promote antigenspecific CD4+ T cell polarization Gamma-glutamyl leukotrienase, a novel endothelial membrane protein, is specifically responsible for leukotriene D(4) formation in vivo Transfer of in vivo primed transgenic T cells supports allergic lung inflammation and FIZZ1 and Ym1 production in an IL-4Rα and STAT6 dependent manner.Decreased allergic lung inflammatory cell egression and increased susceptibility to asphyxiation in MMP2-deficiency Eosinophils and allergic airway disease: there is more to the story Respiratory tract allergic disease and atopy: experimental evidence for a fungal infectious etiology Particulate matter-induced airway hyperresponsiveness is lymphocyte dependent IL-33 mediates multi-walled carbon nanotube (MWCNT)-induced airway hyper-reactivity via the mobilization of innate helper cells in the lung.Interleukin-8 receptor modulates IgE production and B-cell expansion and trafficking in allergen-induced pulmonary inflammation.The adaptor protein insulin receptor substrate 2 inhibits alternative macrophage activation and allergic lung inflammation Link between allergic asthma and airway mucosal infection suggested by proteinase-secreting household fungi.IL-33 and thymic stromal lymphopoietin mediate immune pathology in response to chronic airborne allergen exposure Importance of cytokines in murine allergic airway disease and human asthma.Anisakis pegreffii-induced airway hyperresponsiveness is mediated by gamma interferon in the absence of interleukin-4 receptor alpha responsiveness.Adoptive transfer of IL-4Rα+ macrophages is sufficient to enhance eosinophilic inflammation in a mouse model of allergic lung inflammation.Programmed Death-1 antibody blocks therapeutic effects of T-regulatory cells in cockroach antigen-induced allergic asthma.New insights into the role of cytokines in asthma The AGC kinase inhibitor H89 attenuates airway inflammation in mouse models of asthma Dendritic cells and B cells: unexpected partners in Th2 development.Airway epithelial indoleamine 2,3-dioxygenase inhibits CD4+ T cells during Aspergillus fumigatus antigen exposure.Effect of a CD4-depleting antibody on the development of Cryptococcus neoformans-induced allergic bronchopulmonary mycosis in mice.A reversible, non-invasive method for airway resistance measurements and bronchoalveolar lavage fluid sampling in mice.B cells play key roles in th2-type airway immune responses in mice exposed to natural airborne allergens Beta-arrestin-2 regulates the development of allergic asthma Proteinases as molecular adjuvants in allergic airway disease.Chronic airway hyperreactivity, goblet cell hyperplasia, and peribronchial fibrosis during allergic airway disease induced by Aspergillus fumigatus.Matrix metalloproteinase-9 deficiency impairs cellular infiltration and bronchial hyperresponsiveness during allergen-induced airway inflammation Persistence of asthma requires multiple feedback circuits involving type 2 innate lymphoid cells and IL-33.Alpha-4 integrins and VCAM-1, but not MAdCAM-1, are essential for recruitment of mast cell progenitors to the inflamed lung Long-Acting Beta Agonists Enhance Allergic Airway Disease Evolution of the Immune Response to Chronic Airway Colonization with Aspergillus fumigatus Hyphae Airway epithelial regulation of allergic sensitization in asthma.Type 2 immunity is controlled by IL-4/IL-13 expression in hematopoietic non-eosinophil cells of the innate immune system.

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P2860

Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice

http://www.wikidata.org/entity/Q36438370

description

1998 nî lūn-bûn

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1998年の論文

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年学术文章

@zh-my

1998年学术文章

@zh-sg

1998年學術文章

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1998年學術文章

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1998年學術文章

@zh-hant

name

Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice

@ast

Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice

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type

label

Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice

@ast

Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice

@en

prefLabel

Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice

@ast

Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice

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Molecular Medicine

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Requirements for allergen-induced airway hyperreactivity in T and B cell-deficient mice

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P2093

D B Corry

http://www.w3.org/2001/XMLSchema#string

D M Rennick

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D Sheppard

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G Grünig

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H Hadeiba

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M L Warnock

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R M Locksley

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V P Kurup

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P2860

Eotaxin: a potent eosinophil chemoattractant cytokine detected in a guinea pig model of allergic airways inflammation

Interleukin-4, -5, and -6 and tumor necrosis factor-alpha in normal and asthmatic airways: evidence for the human mast cell as a source of these cytokines

Active anaphylaxis in IgE-deficient mice

RAG-1-deficient mice have no mature B and T lymphocytes

RAG-2-deficient mice lack mature lymphocytes owing to inability to initiate V(D)J rearrangement

Eosinophil recruitment into the respiratory epithelium following antigenic challenge in hyper-IgE mice is accompanied by interleukin 5-dependent bronchial hyperresponsiveness.

A genome-wide search for quantitative trait loci underlying asthma.

Eosinophils express interleukin 5 and granulocyte macrophage-colony-stimulating factor mRNA at sites of allergic inflammation in asthmatics.

Transfer of allergic airway responses with antigen-primed CD4+ but not CD8+ T cells in brown Norway rats.

Allergen-induced bronchial hyperreactivity and eosinophilic inflammation occur in the absence of IgE in a mouse model of asthma.

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2230383

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