Fam83h null mice support a neomorphic mechanism for human ADHCAI
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FAM83 proteins: Fostering new interactions to drive oncogenic signaling and therapeutic resistanceFAM83H and casein kinase I regulate the organization of the keratin cytoskeleton and formation of desmosomes.Casein kinase 1 is recruited to nuclear speckles by FAM83H and SONNovel FAM83H mutations in patients with amelogenesis imperfecta.Evolutionary analysis of FAM83H in vertebrates.Family with sequence similarity 83, member B is a predictor of poor prognosis and a potential therapeutic target for lung adenocarcinoma expressing wild-type epidermal growth factor receptor.The FAM83 family of proteins: from pseudo-PLDs to anchors for CK1 isoforms.
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Q28077347-25CBFAA5-41B3-4F13-A2A6-4C0B83A2277BQ36934120-DEA1DC7C-6387-41BB-A0E4-28DEFF149358Q37294029-80C35287-C2AC-4C07-9768-9CFAFA82DE23Q40488428-5E7C981B-1655-4EDD-8B04-61B7E56120E2Q40953441-184D57DE-ABFB-48DE-BDB0-A3F0E4272F17Q50053792-EC3E60ED-C33B-4A25-BDEB-F8367C26BE0BQ54976961-A63A1AC0-F75B-47F6-B27C-894E5A3DEADA
P2860
Fam83h null mice support a neomorphic mechanism for human ADHCAI
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2015 nî lūn-bûn
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2015年の論文
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name
Fam83h null mice support a neomorphic mechanism for human ADHCAI
@ast
Fam83h null mice support a neomorphic mechanism for human ADHCAI
@en
type
label
Fam83h null mice support a neomorphic mechanism for human ADHCAI
@ast
Fam83h null mice support a neomorphic mechanism for human ADHCAI
@en
prefLabel
Fam83h null mice support a neomorphic mechanism for human ADHCAI
@ast
Fam83h null mice support a neomorphic mechanism for human ADHCAI
@en
P2093
P2860
P356
P1476
Fam83h null mice support a neomorphic mechanism for human ADHCAI
@en
P2093
Amelia S Richardson
Charles E Smith
Figen Seymen
James P Simmer
Jan C-C Hu
Jung-Wook Kim
Koray Gencay
Mine Koruyucu
P2860
P356
10.1002/MGG3.178
P577
2015-09-21T00:00:00Z