Ontogeny of T cell tolerance to peripherally expressed antigens.
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Opinion: Satisfaction (not) guaranteed: re-evaluating the use of animal models of type 1 diabetesA replicating modified vaccinia tiantan strain expressing an avian-derived influenza H5N1 hemagglutinin induce broadly neutralizing antibodies and cross-clade protective immunity in miceLow Antigen Dose in Adjuvant-Based Vaccination Selectively Induces CD4 T Cells with Enhanced Functional Avidity and Protective Efficacy.In vivo cyclophosphamide and IL-2 treatment impedes self-antigen-induced effector CD4 cell tolerization: implications for adoptive immunotherapy.Effector CD4 cells are tolerized upon exposure to parenchymal self-antigen.Peripheral Tolerization of Effector and Memory T Cells: Implications for Autoimmunity and Tumor-Immunity.Activation of CD1d-restricted T cells protects NOD mice from developing diabetes by regulating dendritic cell subsets.The use of mouse models to better understand mechanisms of autoimmunity and toleranceAssumptions of the tumor 'escape' hypothesis.Prospects for immunotherapy of malignant disease.Autoreactive T cells bypass negative selection and respond to self-antigen stimulation during infection.In situ tolerance within the central nervous system as a mechanism for preventing autoimmunity.Pancreatic lymph nodes are required for priming of beta cell reactive T cells in NOD mice.Uncoupling of proliferative potential and gain of effector function by CD8(+) T cells responding to self-antigens.Physiological beta cell death triggers priming of self-reactive T cells by dendritic cells in a type-1 diabetes model.Phenotypic and functional analysis of CD8(+) T cells undergoing peripheral deletion in response to cross-presentation of self-antigen.Central tolerance: good but imperfect.CD8 T cell ignorance or tolerance to islet antigens depends on antigen dose.Memory-like CD8+ and CD4+ T cells cooperate to break peripheral tolerance under lymphopenic conditions.The cell biology of cross-presentation and the role of dendritic cell subsets.Animal models of human type 1 diabetes.T cells with low avidity for a tissue-restricted antigen routinely evade central and peripheral tolerance and cause autoimmunity.Antigen presentation events in autoimmune diabetes.Enhanced CD8 T cell cross-presentation by macrophages with targeted disruption of STAT3Abortive activation precedes functional deletion of CD8+ T cells following encounter with self-antigens expressed by resting B cells in vivo.Peripheral Deletion of CD8 T Cells Requires p38 MAPK in Cross-Presenting Dendritic Cells.Peripheral antigen display by lymph node stroma promotes T cell tolerance to intestinal self
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P2860
Ontogeny of T cell tolerance to peripherally expressed antigens.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Ontogeny of T cell tolerance to peripherally expressed antigens.
@ast
Ontogeny of T cell tolerance to peripherally expressed antigens.
@en
type
label
Ontogeny of T cell tolerance to peripherally expressed antigens.
@ast
Ontogeny of T cell tolerance to peripherally expressed antigens.
@en
prefLabel
Ontogeny of T cell tolerance to peripherally expressed antigens.
@ast
Ontogeny of T cell tolerance to peripherally expressed antigens.
@en
P2093
P2860
P356
P1476
Ontogeny of T cell tolerance to peripherally expressed antigens.
@en
P2093
P2860
P304
P356
10.1073/PNAS.96.7.3854
P407
P577
1999-03-01T00:00:00Z