Reactivation of Kaposi's sarcoma-associated herpesvirus from latency requires MEK/ERK, JNK and p38 multiple mitogen-activated protein kinase pathways
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Dangerous liaisons: molecular basis for a syndemic relationship between Kaposi's sarcoma and P. falciparum malariaKSHV Genome Replication and MaintenancePhosphoproteomic analyses reveal signaling pathways that facilitate lytic gammaherpesvirus replicationActivation of PI3K/AKT and ERK MAPK signal pathways is required for the induction of lytic cycle replication of Kaposi's sarcoma-associated herpesvirus by herpes simplex virus type 1KSHV reactivation from latency requires Pim-1 and Pim-3 kinases to inactivate the latency-associated nuclear antigen LANADistinct roles for extracellular signal-regulated kinase 1 (ERK1) and ERK2 in the structure and production of a primate gammaherpesvirusPhosphorylation of eukaryotic translation initiation factor 4B (EIF4B) by open reading frame 45/p90 ribosomal S6 kinase (ORF45/RSK) signaling axis facilitates protein translation during Kaposi sarcoma-associated herpesvirus (KSHV) lytic replicationRegulation of NF-kappaB inhibitor IkappaBalpha and viral replication by a KSHV microRNAThe AP-1 binding sites located in the pol gene intragenic regulatory region of HIV-1 are important for viral replicationActivation of Kaposi's sarcoma-associated herpesvirus (KSHV) by inhibitors of class III histone deacetylases: identification of sirtuin 1 as a regulator of the KSHV life cycle.Reactive oxygen species hydrogen peroxide mediates Kaposi's sarcoma-associated herpesvirus reactivation from latencyEBV microRNA BART 18-5p targets MAP3K2 to facilitate persistence in vivo by inhibiting viral replication in B cells.Novel changes in NF-{kappa}B activity during progression and regression phases of hyperplasia: role of MEK, ERK, and p38.Exploitation of the complement system by oncogenic Kaposi's sarcoma-associated herpesvirus for cell survival and persistent infectionCell membrane-bound Kaposi's sarcoma-associated herpesvirus-encoded glycoprotein B promotes virus latency by regulating expression of cellular Egr-1Phosphorylation of the chromatin binding domain of KSHV LANAHuman tumor-associated viruses and new insights into the molecular mechanisms of cancer.KSHV LANA--the master regulator of KSHV latency.Systemically circulating viral and tumor-derived microRNAs in KSHV-associated malignancies.Mechanisms of Kaposi's Sarcoma-Associated Herpesvirus Latency and ReactivationMolecular biology of KSHV lytic reactivation.Molecular biology of Kaposi's sarcoma-associated herpesvirus and related oncogenesisExtracellular Hsp90 serves as a co-factor for NF-κB activation and cellular pathogenesis induced by an oncogenic herpesvirus.Extracellular Hsp90 serves as a co-factor for MAPK activation and latent viral gene expression during de novo infection by KSHV.A novel role of hydrogen peroxide in Kaposi sarcoma-associated herpesvirus reactivation.Celecoxib Inhibits the Lytic Activation of Kaposi's Sarcoma-Associated Herpesvirus through Down-Regulation of RTA Expression by Inhibiting the Activation of p38 MAPK.Cellular MicroRNA Let-7a Suppresses KSHV Replication through Targeting MAP4K4 Signaling Pathways.ORF45-Mediated Prolonged c-Fos Accumulation Accelerates Viral Transcription during the Late Stage of Lytic Replication of Kaposi's Sarcoma-Associated Herpesvirus.Varicella-Zoster virus ORF12 protein triggers phosphorylation of ERK1/2 and inhibits apoptosis.Oncogenic Herpesvirus Utilizes Stress-Induced Cell Cycle Checkpoints for Efficient Lytic ReplicationAn APE1 inhibitor reveals critical roles of the redox function of APE1 in KSHV replication and pathogenic phenotypesAmplification of JNK signaling is necessary to complete the murine gammaherpesvirus 68 lytic replication cycle.Kaposi's sarcoma-associated herpesvirus suppression of DUSP1 facilitates cellular pathogenesis following de novo infection.Kaposi's sarcoma-associated herpesvirus latent gene vFLIP inhibits viral lytic replication through NF-kappaB-mediated suppression of the AP-1 pathway: a novel mechanism of virus control of latency.Ets-1 is required for the activation of VEGFR3 during latent Kaposi's sarcoma-associated herpesvirus infection of endothelial cells.Genetic disruption of KSHV major latent nuclear antigen LANA enhances viral lytic transcriptional program.Integrated analyses of genome-wide DNA occupancy and expression profiling identify key genes and pathways involved in cellular transformation by a Marek's disease virus oncoprotein, Meq.Genome-wide identification of binding sites for Kaposi's sarcoma-associated herpesvirus lytic switch protein, RTA.The inflammatory kinase MAP4K4 promotes reactivation of Kaposi's sarcoma herpesvirus and enhances the invasiveness of infected endothelial cellsTumor viruses and cancer biology: Modulating signaling pathways for therapeutic intervention.
P2860
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P2860
Reactivation of Kaposi's sarcoma-associated herpesvirus from latency requires MEK/ERK, JNK and p38 multiple mitogen-activated protein kinase pathways
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
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2007年學術文章
@zh-hant
name
Reactivation of Kaposi's sarco ...... ivated protein kinase pathways
@ast
Reactivation of Kaposi's sarco ...... ivated protein kinase pathways
@en
type
label
Reactivation of Kaposi's sarco ...... ivated protein kinase pathways
@ast
Reactivation of Kaposi's sarco ...... ivated protein kinase pathways
@en
prefLabel
Reactivation of Kaposi's sarco ...... ivated protein kinase pathways
@ast
Reactivation of Kaposi's sarco ...... ivated protein kinase pathways
@en
P2093
P2860
P1433
P1476
Reactivation of Kaposi's sarco ...... ivated protein kinase pathways
@en
P2093
Adetola Olalekan Ajibade
Fengchun Ye
Jianping Xie
Kurt Kuhne
P2860
P304
P356
10.1016/J.VIROL.2007.09.040
P407
P577
2007-10-26T00:00:00Z