Hepatitis C virus utilizes VLDLR as a novel entry pathway.
about
HCV-Induced Oxidative Stress: Battlefield-Winning StrategyTriglyceride Rich Lipoprotein -LPL-VLDL Receptor and Lp(a)-VLDL Receptor Pathways for Macrophage Foam Cell Formation.Regulated Entry of Hepatitis C Virus into Hepatocytes.Lipoprotein Receptors Redundantly Participate in Entry of Hepatitis C Virus.PCSK9 and infection: A potentially useful or dangerous association?Modeling Viral Spread.Claudins in viral infection: from entry to spread.Hepatitis C virus and proprotein convertase subtilisin/kexin type 9: a detrimental interaction to increase viral infectivity and disrupt lipid metabolism.Treatment-Induced Viral Cure of Hepatitis C Virus-Infected Patients Involves a Dynamic Interplay among three Important Molecular Players in Lipid Homeostasis: Circulating microRNA (miR)-24, miR-223, and Proprotein Convertase Subtilisin/Kexin Type 9.Association of a 3' Untranslated Region Polymorphism in PCSK9 with HIV Viral Load and CD4+ Levels in HIV/Hepatitis C Virus Co-Infected Women.CD81 Receptor Regions outside the Large Extracellular Loop Determine Hepatitis C Virus Entry into Hepatoma Cells.
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Hepatitis C virus utilizes VLDLR as a novel entry pathway.
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2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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name
Hepatitis C virus utilizes VLDLR as a novel entry pathway.
@ast
Hepatitis C virus utilizes VLDLR as a novel entry pathway.
@en
type
label
Hepatitis C virus utilizes VLDLR as a novel entry pathway.
@ast
Hepatitis C virus utilizes VLDLR as a novel entry pathway.
@en
prefLabel
Hepatitis C virus utilizes VLDLR as a novel entry pathway.
@ast
Hepatitis C virus utilizes VLDLR as a novel entry pathway.
@en
P2093
P2860
P356
P1476
Hepatitis C virus utilizes VLDLR as a novel entry pathway.
@en
P2093
Hironori Nishitsuji
Hiroshi Takaku
Kazuo Sugiyama
Kunitada Shimotohno
Saneyuki Ujino
Takayuki Hishiki
P2860
P304
P356
10.1073/PNAS.1506524113
P407
P577
2015-12-23T00:00:00Z