Low levels of mammalian TGF-beta1 are protective against malaria parasite infection, a paradox clarified in the mosquito host
about
MAPK ERK signaling regulates the TGF-beta1-dependent mosquito response to Plasmodium falciparumSustained activation of Akt elicits mitochondrial dysfunction to block Plasmodium falciparum infection in the mosquito hostTranslational systems approaches to the biology of inflammation and healing.In vivo, in vitro, and in silico studies suggest a conserved immune module that regulates malaria parasite transmission from mammals to mosquitoesHuman host-derived cytokines associated with Plasmodium vivax transmission from acute malaria patients to Anopheles darlingi mosquitoes in the Peruvian Amazon
P2860
Low levels of mammalian TGF-beta1 are protective against malaria parasite infection, a paradox clarified in the mosquito host
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
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2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
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2007年學術文章
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2007年學術文章
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name
Low levels of mammalian TGF-be ...... clarified in the mosquito host
@ast
Low levels of mammalian TGF-be ...... clarified in the mosquito host
@en
type
label
Low levels of mammalian TGF-be ...... clarified in the mosquito host
@ast
Low levels of mammalian TGF-be ...... clarified in the mosquito host
@en
prefLabel
Low levels of mammalian TGF-be ...... clarified in the mosquito host
@ast
Low levels of mammalian TGF-be ...... clarified in the mosquito host
@en
P2093
P2860
P1476
Low levels of mammalian TGF-be ...... clarified in the mosquito host
@en
P2093
Matthew J Lieber
Naresh Singh
Ruben Zamora
Shirley Luckhart
Yoram Vodovotz
P2860
P304
P356
10.1016/J.EXPPARA.2007.08.013
P577
2007-09-02T00:00:00Z