tPA contributes to impaired NMDA cerebrovasodilation after traumatic brain injury through activation of JNK MAPK.

tPA contributes to impaired NMDA cerebrovasodilation after traumatic brain injury through activation of JNK MAPK.

Item
http://www.wikidata.org/entity/Q36469555
Linking traumatic brain injury to chronic traumatic encephalopathy: identification of potential mechanisms leading to neurofibrillary tangle developmentSubacute intranasal administration of tissue plasminogen activator promotes neuroplasticity and improves functional recovery following traumatic brain injury in ratsXG-102 administered to healthy male volunteers as a single intravenous infusion: a randomized, double-blind, placebo-controlled, dose-escalating study.The tissue-type plasminogen activator-plasminogen activator inhibitor 1 complex promotes neurovascular injury in brain trauma: evidence from mice and humansPlasmin-dependent modulation of the blood-brain barrier: a major consideration during tPA-induced thrombolysis?Glucagon protects against impaired NMDA-mediated cerebrovasodilation and cerebral autoregulation during hypotension after brain injury by activating cAMP protein kinase A and inhibiting upregulation of tPA.tPA-S481A prevents neurotoxicity of endogenous tPA in traumatic brain injury.Combination Therapies for Traumatic Brain Injury: Retrospective ConsiderationsCombination therapy with glucagon and a novel plasminogen activator inhibitor-1-derived peptide enhances protection against impaired cerebrovasodilation during hypotension after traumatic brain injury through inhibition of ERK and JNK MAPKRBC-coupled tPA Prevents Whereas tPA Aggravates JNK MAPK-Mediated Impairment of ATP- and Ca-Sensitive K Channel-Mediated Cerebrovasodilation After Cerebral PhotothrombosistPA-S(481)A prevents impairment of cerebrovascular autoregulation by endogenous tPA after traumatic brain injury by upregulating p38 MAPK and inhibiting ET-1.Interactions between Sirt1 and MAPKs regulate astrocyte activation induced by brain injury in vitro and in vivo.K channel impairment determines sex and age differences in epinephrine-mediated outcomes after brain injury.tPA Modulation of the Blood-Brain Barrier: A Unifying Explanation for the Pleiotropic Effects of tPA in the CNS.Sex and Age Differences in Epinephrine Mechanisms and Outcomes after Brain Injury.Tissue-Type Plasminogen Activator-A296-299 Prevents Impairment of Cerebral Autoregulation After Stroke Through Lipoprotein-Related Receptor-Dependent Increase in cAMP and p38.tPA variant tPA-A296-299 Prevents impairment of cerebral autoregulation and necrosis of hippocampal neurons after stroke by inhibiting upregulation of ET-1.
P2860

tPA contributes to impaired NMDA cerebrovasodilation after traumatic brain injury through activation of JNK MAPK.

Item
http://www.wikidata.org/entity/Q36469555
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
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2011年學術文章
@zh-hant
name
tPA contributes to impaired NM ...... hrough activation of JNK MAPK.
@ast
tPA contributes to impaired NM ...... hrough activation of JNK MAPK.
@en
type
label
tPA contributes to impaired NM ...... hrough activation of JNK MAPK.
@ast
tPA contributes to impaired NM ...... hrough activation of JNK MAPK.
@en
prefLabel
tPA contributes to impaired NM ...... hrough activation of JNK MAPK.
@ast
tPA contributes to impaired NM ...... hrough activation of JNK MAPK.
@en
P1433
P1476
P2093
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P577
P698
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tPA contributes to impaired NM ...... through activation of JNK MAPK
@en
P2093
P2860
P304
P31
P356
10.1179/016164110X12807570509853
P433
P478
P50
P577
P698
P921
P932