Nuclear ULK1 promotes cell death in response to oxidative stress through PARP1
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Impaired Autophagy and Defective Mitochondrial Function: Converging Paths on the Road to Motor Neuron DegenerationAutophagy signal transduction by ATG proteins: from hierarchies to networksLoss of ULK1 increases RPS6KB1-NCOR1 repression of NR1H/LXR-mediated Scd1 transcription and augments lipotoxicity in hepatic cellsBeyond autophagy: New roles for ULK1 in immune signaling and interferon responses.Sunitinib induces genomic instability of renal carcinoma cells through affecting the interaction of LC3-II and PARP-1.Serine/Threonine Kinase Unc-51-like Kinase-1 (Ulk1) Phosphorylates the Co-chaperone Cell Division Cycle Protein 37 (Cdc37) and Thereby Disrupts the Stability of Cdc37 Client Proteins.Canonical and noncanonical functions of ULK/Atg1.High mobility group A1 protein modulates autophagy in cancer cells.ULK1 in type I interferon response.Systematic analysis of ATG13 domain requirements for autophagy induction.Systems-Level Feedbacks of NRF2 Controlling Autophagy upon Oxidative Stress Response.The deubiquitinating enzyme USP20 stabilizes ULK1 and promotes autophagy initiation.Overexpression of ULK1 Represents a Potential Diagnostic Marker for Clear Cell Renal Carcinoma and the Antitumor Effects of SBI-0206965
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P2860
Nuclear ULK1 promotes cell death in response to oxidative stress through PARP1
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name
Nuclear ULK1 promotes cell death in response to oxidative stress through PARP1
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Nuclear ULK1 promotes cell death in response to oxidative stress through PARP1
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label
Nuclear ULK1 promotes cell death in response to oxidative stress through PARP1
@ast
Nuclear ULK1 promotes cell death in response to oxidative stress through PARP1
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prefLabel
Nuclear ULK1 promotes cell death in response to oxidative stress through PARP1
@ast
Nuclear ULK1 promotes cell death in response to oxidative stress through PARP1
@en
P2093
P2860
P356
P1476
Nuclear ULK1 promotes cell death in response to oxidative stress through PARP1
@en
P2093
A Phillips
B J Winborn
R Kriwacki
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P2888
P304
P356
10.1038/CDD.2015.88
P577
2015-07-03T00:00:00Z