New perspectives on central and peripheral immune responses to acute traumatic brain injury. - Wikidata - awgldk - TriplyDB

New perspectives on central and peripheral immune responses to acute traumatic brain injury.

New perspectives on central and peripheral immune responses to acute traumatic brain injury.

http://www.wikidata.org/entity/Q36478408

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Inhibition of cathepsin S produces neuroprotective effects after traumatic brain injury in mice Impaired Pituitary Axes Following Traumatic Brain Injury Bridge between neuroimmunity and traumatic brain injury Inflammation and neuroprotection in traumatic brain injury Emerging Roles for the Immune System in Traumatic Brain Injury Inflammatory reaction after traumatic brain injury: therapeutic potential of targeting cell-cell communication by chemokines Cerebrospinal Fluid Markers of Macrophage and Lymphocyte Activation After Traumatic Brain Injury in Children Analysis of the Role of CX3CL1 (Fractalkine) and Its Receptor CX3CR1 in Traumatic Brain and Spinal Cord Injury: Insight into Recent Advances in Actions of Neurochemokine Agents.Effects of progesterone vs. dexamethasone on brain oedema and inflammatory responses following experimental brain resection.Anti-Inflammatory and Neuroprotective Role of Natural Product Securinine in Activated Glial Cells: Implications for Parkinson's Disease.Suppressed cytokine expression immediatey following traumatic brain injury in neonatal rats indicates an expeditious endogenous anti-inflammatory response IL-1β associations with posttraumatic epilepsy development: a genetics and biomarker cohort study.The p38alpha mitogen-activated protein kinase limits the CNS proinflammatory cytokine response to systemic lipopolysaccharide, potentially through an IL-10 dependent mechanism Traumatic brain injury causes selective, CD74-dependent peripheral lymphocyte activation that exacerbates neurodegeneration.Berberine protects against neuronal damage via suppression of glia-mediated inflammation in traumatic brain injury.Clinical evidence of inflammation driving secondary brain injury: a systematic review.Motor, visual and emotional deficits in mice after closed-head mild traumatic brain injury are alleviated by the novel CB2 inverse agonist SMM-189 Prion infection of mouse brain reveals multiple new upregulated genes involved in neuroinflammation or signal transduction.Peripheral Blood Mitochondrial DNA as a Biomarker of Cerebral Mitochondrial Dysfunction following Traumatic Brain Injury in a Porcine Model Galectin-3 released in response to traumatic brain injury acts as an alarmin orchestrating brain immune response and promoting neurodegeneration.Ccr2 deletion dissociates cavity size and tau pathology after mild traumatic brain injury Long-Term Consequences of Traumatic Brain Injury: Current Status of Potential Mechanisms of Injury and Neurological Outcomes Regnase-1 in microglia negatively regulates high mobility group box 1-mediated inflammation and neuronal injury.In vivo microdialysis sampling of cytokines from rat hippocampus: comparison of cannula implantation procedures Ventilator-Associated Pneumonia in Pediatric Traumatic Brain Injury.Older Age Results in Differential Gene Expression after Mild Traumatic Brain Injury and Is Linked to Imaging Differences at Acute Follow-up.Mild Traumatic Brain Injury Produces Neuron Loss That Can Be Rescued by Modulating Microglial Activation Using a CB2 Receptor Inverse Agonist.Risk of Coronary Artery Disease in Patients With Traumatic Intracranial Hemorrhage: A Nationwide, Population-Based Cohort Study.Neuroinflammation in animal models of traumatic brain injury.Differences in the Cellular Response to Acute Spinal Cord Injury between Developing and Mature Rats Highlights the Potential Significance of the Inflammatory Response.Inflammation in neurodegenerative diseases--an update.Influence of the extracellular matrix on endogenous and transplanted stem cells after brain damage Blood-brain barrier dysfunction following traumatic brain injury.Anti-inflammatory and immunomodulatory mechanisms of atorvastatin in a murine model of traumatic brain injury.Minocycline plus N-acteylcysteine induces remyelination, synergistically protects oligodendrocytes, and modifies neuroinflammation in a rat model of mild traumatic brain injury.Genome-Wide Changes in Peripheral Gene Expression following Sports-Related Concussion Pathophysiology Associated with Traumatic Brain Injury: Current Treatments and Potential Novel Therapeutics.Natural Compounds as a Therapeutic Intervention following Traumatic Brain Injury: The Role of Phytochemicals.Magnetic micelles for DNA delivery to rat brains after mild traumatic brain injury.A flow cytometric approach to analyzing mature and progenitor endothelial cells following traumatic brain injury.

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New perspectives on central and peripheral immune responses to acute traumatic brain injury.

http://www.wikidata.org/entity/Q36478408

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2012 nî lūn-bûn

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New perspectives on central an ...... acute traumatic brain injury.

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1742-2094-9-236

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Journal of Neuroinflammation

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New perspectives on central an ...... acute traumatic brain injury.

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Mahasweta Das

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Shyam S Mohapatra

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Subhra Mohapatra

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Creative Commons Attribution 2.0 Generic

P2860

The occult aftermath of boxing

Nosocomial infections and immunity: lesson from brain-injured patients

Modulation of thalamic nociceptive processing after spinal cord injury through remote activation of thalamic microglia by cysteine cysteine chemokine ligand 21

Function of the chemokine receptor CXCR4 in haematopoiesis and in cerebellar development

CINC-1 is an acute-phase protein induced by focal brain injury causing leukocyte mobilization and liver injury

Central nervous system injury triggers hepatic CC and CXC chemokine expression that is associated with leukocyte mobilization and recruitment to both the central nervous system and the liver

The CC chemokine CCL20 and its receptor CCR6.

[Apoptosis in multiple organs of rats in early stage of polytrauma combined with shock]

Minocycline treatment reduces delayed oligodendrocyte death, attenuates axonal dieback, and improves functional outcome after spinal cord injury.

Lipids and lipidomics in brain injury and diseases

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traumatic brain injury

hemic and immune systems

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3526406

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