Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice - Wikidata - awgldk - TriplyDB

Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice

Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice

http://www.wikidata.org/entity/Q36490737

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Targeted therapeutics in SLE: emerging strategies to modulate the interferon pathway Breakdown of Immune Tolerance in Systemic Lupus Erythematosus by Dendritic Cells Predominant Role of Plasmacytoid Dendritic Cells in Stimulating Systemic Autoimmunity TLRs and interferons: a central paradigm in autoimmunity Modelling clinical systemic lupus erythematosus: similarities, differences and success stories Contribution of dendritic cells to the autoimmune pathology of systemic lupus erythematosus Type I interferon is a therapeutic target for virus-induced lethal vascular damage.Autoimmunity-associated protein tyrosine phosphatase PEP negatively regulates IFN-α receptor signaling NKG2D+CD4+ T Cells Kill Regulatory T Cells in a NKG2D-NKG2D Ligand- Dependent Manner in Systemic Lupus Erythematosus.Type I interferon in the pathogenesis of lupus.Pivotal Functions of Plasmacytoid Dendritic Cells in Systemic Autoimmune Pathogenesis.Early, transient depletion of plasmacytoid dendritic cells ameliorates autoimmunity in a lupus model.Targeting of type I interferon in systemic autoimmune diseases Requirements for innate immune pathways in environmentally induced autoimmunity.The effect of the autoimmunity-associated gene, PTPN22, on a BXSB-derived model of lupus S1PR1-mediated IFNAR1 degradation modulates plasmacytoid dendritic cell interferon-α autoamplification Essential requirement for IRF8 and SLC15A4 implicates plasmacytoid dendritic cells in the pathogenesis of lupus.A Regulatory Feedback between Plasmacytoid Dendritic Cells and Regulatory B Cells Is Aberrant in Systemic Lupus Erythematosus The multifaceted biology of plasmacytoid dendritic cells.Development of Th17-Associated Interstitial Kidney Inflammation in Lupus-Prone Mice Lacking the Gene Encoding STAT-1.IFNα Inducible Models of Murine SLE.IL-21 is a double-edged sword in the systemic lupus erythematosus-like disease of BXSB.Yaa mice Interferon-γ and systemic autoimmunity.New approaches for the treatment of lupus nephritis in the 21st century: from the laboratory to the clinic.Microglia-dependent synapse loss in type I interferon-mediated lupus.The Role of IFN-β during the Course of Sepsis Progression and Its Therapeutic Potential.The Role of STAT Signaling Pathways in the Pathogenesis of Systemic Lupus Erythematosus.Clinical Pharmacokinetics and Pharmacodynamics of Biologic Therapeutics for Treatment of Systemic Lupus Erythematosus.Human pDCs display sex-specific differences in type I interferon subtypes and interferon α/β receptor expression.The multiple pathways to autoimmunity.A panel of synthetic antibodies that selectively recognize and antagonize members of the interferon alpha family.Efficacy of a Virus-Like Nanoparticle As Treatment for a Chronic Viral Infection Is Hindered by IRAK1 Regulation and Antibody Interference.Molecular and cellular discoveries in inflammatory dermatoses.TANK-Binding Kinase 1-Dependent Responses in Health and Autoimmunity.Lupus acceleration by a MAVS-activating RNA virus requires endosomal TLR signaling and host genetic predisposition

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Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice

http://www.wikidata.org/entity/Q36490737

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

@zh-sg

2012年學術文章

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2012年學術文章

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2012年學術文章

@zh-hant

name

Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice

@ast

Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice

@en

type

label

Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice

@ast

Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice

@en

prefLabel

Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice

@ast

Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice

@en

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P356

JIMMUNOL.1201477

P1433

Journal of Immunology

P1476

Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed mice

@en

P2093

Argyrios N Theofilopoulos

http://www.w3.org/2001/XMLSchema#string

Brian R Lawson

http://www.w3.org/2001/XMLSchema#string

Dwight H Kono

http://www.w3.org/2001/XMLSchema#string

Robert D Schreiber

http://www.w3.org/2001/XMLSchema#string

Roberto Baccala

http://www.w3.org/2001/XMLSchema#string

Rosana Gonzalez-Quintial

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P2860

SCF(HOS) ubiquitin ligase mediates the ligand-induced down-regulation of the interferon-alpha receptor

Differential viral induction of distinct interferon-alpha genes by positive feedback through interferon regulatory factor-7

Intracellular nucleic acid sensors and autoimmunity

Efficient T cell activation via a Toll-Interleukin 1 Receptor-independent pathway

IRFs: master regulators of signalling by Toll-like receptors and cytosolic pattern-recognition receptors

Apoptosis and interferons: role of interferon-stimulated genes as mediators of apoptosis

Longitudinal expression of type I interferon responsive genes in systemic lupus erythematosus.

Interferon-alpha treatment of female (NZW x BXSB)F(1) mice mimics some but not all features associated with the Yaa mutation.

Fas ligand-induced apoptosis.

Deficiency of the type I interferon receptor protects mice from experimental lupus.

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5976-5984

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10.4049/JIMMUNOL.1201477

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12

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23175700

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3530925

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