Retinoic Acid Induces Apoptosis of Prostate Cancer DU145 Cells through Cdk5 Overactivation
about
Cyclin-dependent kinase 5 acts as a critical determinant of AKT-dependent proliferation and regulates differential gene expression by the androgen receptor in prostate cancer cellsRetinoic acid and cancer treatment.Retinal Targets ALDH Positive Cancer Stem Cell and Alters the Phenotype of Highly Metastatic Osteosarcoma Cells.Emodin and Aloe-Emodin Suppress Breast Cancer Cell Proliferation through ER α Inhibition.Decitabine and SAHA-induced apoptosis is accompanied by survivin downregulation and potentiated by ATRA in p53-deficient cells.Cdk5 links with DNA damage response and cancer.Combination treatment of all-trans retinoic acid (ATRA) and γ-secretase inhibitor (DAPT) cause growth inhibition and apoptosis induction in the human gastric cancer cell line.
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P2860
Retinoic Acid Induces Apoptosis of Prostate Cancer DU145 Cells through Cdk5 Overactivation
description
2012 nî lūn-bûn
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name
Retinoic Acid Induces Apoptosi ...... ls through Cdk5 Overactivation
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Retinoic Acid Induces Apoptosi ...... ls through Cdk5 Overactivation
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type
label
Retinoic Acid Induces Apoptosi ...... ls through Cdk5 Overactivation
@ast
Retinoic Acid Induces Apoptosi ...... ls through Cdk5 Overactivation
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prefLabel
Retinoic Acid Induces Apoptosi ...... ls through Cdk5 Overactivation
@ast
Retinoic Acid Induces Apoptosi ...... ls through Cdk5 Overactivation
@en
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P2860
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Retinoic Acid Induces Apoptosi ...... ls through Cdk5 Overactivation
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P2093
Chien-Te Ku
Chih-Yang Huang
Chuan-Mu Chen
Eugene Lin
Mei-Chih Chen
Shih-Lan Hsu
P2860
P304
P356
10.1155/2012/580736
P577
2012-12-13T00:00:00Z