Epstein-Barr virus lytic transactivator Zta enhances chemotactic activity through induction of interleukin-8 in nasopharyngeal carcinoma cells. - Wikidata - awgldk - TriplyDB

Epstein-Barr virus lytic transactivator Zta enhances chemotactic activity through induction of interleukin-8 in nasopharyngeal carcinoma cells.

Epstein-Barr virus lytic transactivator Zta enhances chemotactic activity through induction of interleukin-8 in nasopharyngeal carcinoma cells.

http://www.wikidata.org/entity/Q36498181

about

Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesis The Epstein-Barr Virus (EBV) in T Cell and NK Cell Lymphomas: Time for a Reassessment.Evaluation of a prediction protocol to identify potential targets of epigenetic reprogramming by the cancer associated Epstein Barr virus Arsenic trioxide inhibits EBV reactivation and promotes cell death in EBV-positive lymphoma cells.Epigenetic control of viral life-cycle by a DNA-methylation dependent transcription factor.The Epstein-Barr virus BZLF1 protein inhibits tumor necrosis factor receptor 1 expression through effects on cellular C/EBP proteins Modulation of lung inflammation by the Epstein-Barr virus protein Zta.A new model of Epstein-Barr virus infection reveals an important role for early lytic viral protein expression in the development of lymphomas.Epstein-Barr virus Zta upregulates matrix metalloproteinases 3 and 9 that synergistically promote cell invasion in vitro.Epstein-Barr virus Zta-induced immunomodulators from nasopharyngeal carcinoma cells upregulate interleukin-10 production from monocytes Epstein-Barr virus transcription factor Zta acts through distal regulatory elements to directly control cellular gene expression.Recent advances in underlying pathologies provide insight into interleukin-8 expression-mediated inflammation and angiogenesis.Epstein-Barr virus latent membrane protein 2A promotes invasion of nasopharyngeal carcinoma cells through ERK/Fra-1-mediated induction of matrix metalloproteinase 9 As an independent unfavorable prognostic factor, IL-8 promotes metastasis of nasopharyngeal carcinoma through induction of epithelial-mesenchymal transition and activation of AKT signaling Expression of interleukin-8, interleukin-10 and Epstein-Barr viral-load as prognostic indicator in nasopharyngeal carcinoma.EBV Zta protein induces the expression of interleukin-13, promoting the proliferation of EBV-infected B cells and lymphoblastoid cell lines.Switching of EBV cycles between latent and lytic states.Regulation of Epstein-Barr virus reactivation from latency.Photodynamic therapy-mediated modulation of inflammatory cytokine production by Epstein-Barr virus-infected nasopharyngeal carcinoma cells.Inhibition of Epstein-Barr virus lytic cycle by an ethyl acetate subfraction separated from Polygonum cuspidatum root and its major component, emodin.Epstein-Barr virus deubiquitinase downregulates TRAF6-mediated NF-κB signaling during productive replication.Initiation of lytic DNA replication in Epstein-Barr virus: search for a common family mechanism.Lytic EBV infection investigated by detection of Soluble Epstein-Barr virus ZEBRA in the serum of patients with PTLD.Inhibition of Epstein-Barr virus reactivation in nasopharyngeal carcinoma cells by dietary sulforaphane.Specific NFkappaB subunit activation and kinetics of cytokine induction in adenoviral keratitis.Expression of CXCR1 (interleukin-8 receptor) in murine macrophages after staphylococcus aureus infection and its possible implication on intracellular survival correlating with cytokines and bacterial anti-oxidant enzymes.Herpes simplex virus type 1 infection activates the Epstein-Barr virus replicative cycle via a CREB-dependent mechanism.Macrophage migration inhibitory factor contributes angiogenesis by up-regulating IL-8 and correlates with poor prognosis of patients with primary nasopharyngeal carcinoma.Interplay Between Microenvironmental Abnormalities and Infectious Agents in Tumorigenesis.Epstein-Barr Virus as a Promising Immunotherapeutic Target for Nasopharyngeal Carcinoma Treatment.The Immunomodulatory Capacity of an Epstein-Barr Virus Abortive Lytic Cycle: Potential Contribution to Viral Tumorigenesis.Interplay of Viral Infection, Host Cell Factors and Tumor Microenvironment in the Pathogenesis of Nasopharyngeal Carcinoma.

P2860

Q28079510-A59D800D-A0B9-4BB9-AA94-A542DEFB1B09 Q30278553-6A2BA8D2-5067-4B5A-A541-C7C1DDB09457 Q33535848-E1DEC74D-1447-4ED2-954C-DFEC2BAE3234 Q33823359-D8FBA340-90E2-49BD-8672-6123D316B202 Q34055803-7B00D062-4D73-4619-8DFD-8910B5847AFC Q34291099-51D8FA53-F026-4DEE-8AAE-049276B59A40 Q34426098-AD50BA4C-2E16-44ED-A11A-5817F2A2CE57 Q34457976-8F9E545B-8709-4497-B399-DFE8DEA5556B Q34586675-F5414F9B-C0A6-4046-ABEF-BA9B9652D680 Q35077651-5CEE4806-4021-4551-9E4F-D4ECBB23F901 Q35476446-74E8A6BF-E3EB-4CE4-9887-F4B35C6A7D24 Q35659991-C7275DF1-9A1C-4466-8A68-5038B4C8FF47 Q36086653-E7D863E1-0174-4EFA-8406-C512F610DA34 Q36118892-EAABE86B-D8A5-4D0E-92D0-78DFC95F753B Q36713194-10F489A7-0E89-44FE-A88C-B87CED062153 Q37262460-75D45279-A96F-4E99-938E-0BED2D0351E2 Q38171455-3DAD54D8-83D9-483B-A6A8-A0E59A713872 Q38208532-396CD1CF-C745-46AC-ACCE-B420493DB979 Q39026506-BB2AB73C-2C51-41C6-A85A-BC2274F7A63A Q39032271-850C79DE-7369-432A-A231-A0816495D6C0 Q39789708-A3290812-7A06-4029-A908-6FD3F780A6FF Q39914140-299916BD-4E53-4FA3-A902-F04E1F2E271C Q40056807-3FD7EF75-B2C5-48BF-9B7E-D0D2FAA733E3 Q42717841-66AAA1EF-DB92-4F54-A251-98D0182136CF Q42948998-5DE04025-5089-4F62-83D1-FA717A3DF38F Q44516269-48B43E22-D040-4C5A-97E2-8E49204A3ABB Q45360917-35F90BAE-7E27-43B8-B51A-E81833C94969 Q46003105-12DCDE92-59F5-4765-9976-6DAD0CE8F3D9 Q47606249-F55DCB5C-11FE-41AC-B687-A9AF24488441 Q49787173-18566FBE-04FA-452B-9589-B225ABDE7295 Q54245037-C7734BA0-5C3B-488E-9F1C-F1D2CFF84292 Q55313422-853D71E9-2957-4E1C-BDCB-8A30929FA277

P2860

Epstein-Barr virus lytic transactivator Zta enhances chemotactic activity through induction of interleukin-8 in nasopharyngeal carcinoma cells.

http://www.wikidata.org/entity/Q36498181

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年学术文章

@wuu

2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh

2008年學術文章

@zh-hant

name

Epstein-Barr virus lytic trans ...... asopharyngeal carcinoma cells.

@ast

Epstein-Barr virus lytic trans ...... asopharyngeal carcinoma cells.

@en

type

label

Epstein-Barr virus lytic trans ...... asopharyngeal carcinoma cells.

@ast

Epstein-Barr virus lytic trans ...... asopharyngeal carcinoma cells.

@en

prefLabel

Epstein-Barr virus lytic trans ...... asopharyngeal carcinoma cells.

@ast

Epstein-Barr virus lytic trans ...... asopharyngeal carcinoma cells.

@en

P1433

Q36498181-7A3840CD-C8CB-4785-BD28-5FB2F15FA59F

P1476

Q36498181-9876E3BE-4A37-4713-8040-407B10A9ED44

P2093

Q36498181-107AB1B5-FE6B-4968-A842-3B757FB08F84

Q36498181-1D4D17E3-453A-4E81-BB2E-DABA1096CBB4

Q36498181-5696C3CD-6421-458D-9C8D-88C54B8B1C88

Q36498181-798EFB37-5E61-4AB9-AE0E-6EC107FF4F53

Q36498181-86683AFD-8D03-43AB-B125-CC5602D5CF6A

Q36498181-BDC04BBE-BE3B-4665-8AB1-35B5D96DD560

Q36498181-C81716B4-2CCD-4840-BE08-F0A4BE1074B0

Q36498181-E3EBF63E-763E-45AE-8F07-400656B47F80

Q36498181-F245A162-C7DF-4777-9DE1-05ABB107867A

P2860

Q36498181-05A3A005-FECC-4D70-83DF-A8F024BC5BA6

Q36498181-0B761BA9-7B4C-4445-AB7C-8EF3D80400B9

Q36498181-13A630A4-873C-4CB6-8818-F4579AD7781F

Q36498181-13C5F524-17B9-4F66-A72B-B949ECE01A39

Q36498181-1A51E8C3-CB29-43DC-86D4-9FA9A26126B9

Q36498181-1CE003B6-D671-4AF7-A59C-AB7F0EA5A6EB

Q36498181-1F706E73-E574-4807-990D-BA37B6A5821F

Q36498181-21FD02C3-E2D9-4520-8784-286364D1F5A2

Q36498181-242BA3BD-889D-455F-A2FD-C884D3F74EB9

Q36498181-25BC1888-FDD9-47E7-ABA9-FE4FA0A62BD0

P304

Q36498181-26155802-5BC9-4FCE-8506-B9F85F7DCE7F

P31

Q36498181-5850260C-0C70-43C0-8A23-9A7F74B7B0C0

P356

Q36498181-9EBA0C39-A030-4EFF-9947-8E05FFD389C6

P407

Q36498181-EE8979C0-FB68-4806-8AFF-B23985CF1F04

P433

Q36498181-015A5B93-ED09-4F9B-AA98-985526FAEA78

P478

Q36498181-DA6447EE-A591-4FEE-BBEB-8AAAF69919C1

P577

Q36498181-E6168BC9-2237-4B96-8645-67B9BFD26A41

P698

Q36498181-085CD3E0-4C74-45BE-9853-323D1D10418F

P921

Q36498181-0CF8FDC8-28A6-4D44-8E86-757C859F41E6

Q36498181-234BA1C3-4BA6-4459-A9D9-76B16204B989

Q36498181-5a7b5915-b2fe-418d-a35b-99a64c7625b0

P932

Q36498181-6508DA4A-2CCE-4BAB-B84A-F7C34CC312E0

P356

P1433

Journal of Virology

P1476

Epstein-Barr virus lytic trans ...... asopharyngeal carcinoma cells.

@en

P2093

Ai-Li Shiau

http://www.w3.org/2001/XMLSchema#string

Ching-Hwa Tsai

http://www.w3.org/2001/XMLSchema#string

Ih-Jen Su

http://www.w3.org/2001/XMLSchema#string

Kenzo Takada

http://www.w3.org/2001/XMLSchema#string

Meichi Hsu

http://www.w3.org/2001/XMLSchema#string

Shih-Shin Chang

http://www.w3.org/2001/XMLSchema#string

Shih-Yi Wu

http://www.w3.org/2001/XMLSchema#string

Siao-Jing Lai

http://www.w3.org/2001/XMLSchema#string

Yao Chang

http://www.w3.org/2001/XMLSchema#string

P2860

Cancer and the chemokine network

Epstein-Barr virus lytic infection induces retinoic acid-responsive genes through induction of a retinol-metabolizing enzyme, DHRS9

Monoclonal antibodies detect different distribution patterns of IL-8 receptor A and IL-8 receptor B on human peripheral blood leukocytes

A distinct expression of CC chemokines by macrophages in nasopharyngeal carcinoma: implication for the intense tumor infiltration by T lymphocytes and macrophages.

Stimulus-specific regulation of chemokine expression involves differential activation of the redox-responsive transcription factors AP-1 and NF-kappaB.

Upregulation of tyrosine kinase TKT by the Epstein-Barr virus transactivator Zta

Requirement for cell-to-cell contact in Epstein-Barr virus infection of nasopharyngeal carcinoma cells and keratinocytes.

Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.

The neutrophil as a cellular source of chemokines.

Epstein-Barr virus lytic infection is required for efficient production of the angiogenesis factor vascular endothelial growth factor in lymphoblastoid cell lines.

P304

3679-3688

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1128/JVI.02301-07

http://www.w3.org/2001/XMLSchema#string

P407

P433

7

http://www.w3.org/2001/XMLSchema#string

P478

82

http://www.w3.org/2001/XMLSchema#string

P577

2008-01-30T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

18234802

http://www.w3.org/2001/XMLSchema#string

P921

Epstein–Barr virus

P932

2268478

http://www.w3.org/2001/XMLSchema#string