CX(3)CR1(+) macrophages support IL-22 production by innate lymphoid cells during infection with Citrobacter rodentium.
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Role of the normal gut microbiotaColonization of C57BL/6 Mice by a Potential Probiotic Bifidobacterium bifidum Strain under Germ-Free and Specific Pathogen-Free Conditions and during Experimental ColitisNotch2-dependent classical dendritic cells orchestrate intestinal immunity to attaching-and-effacing bacterial pathogensMyD88 signaling in dendritic cells and the intestinal epithelium controls immunity against intestinal infection with C. rodentium.CX₃CR1⁺ mononuclear phagocytes support colitis-associated innate lymphoid cell production of IL-22.Polarizing the T helper 17 response in Citrobacter rodentium infection via expression of resistin-like molecule α.The interplay between the gut microbiota and the immune system.Aryl hydrocarbon receptor promotes RORγt⁺ group 3 ILCs and controls intestinal immunity and inflammation.A balanced IL-1β activity is required for host response to Citrobacter rodentium infection.Intestinal intraepithelial lymphocyte-enterocyte crosstalk regulates production of bactericidal angiogenin 4 by Paneth cells upon microbial challenge.CX3CR1 is dispensable for control of mucosal Candida albicans infections in mice and humans.Interleukin-22: immunobiology and pathology.SLAMF4 Is a Negative Regulator of Expansion of Cytotoxic Intraepithelial CD8+ T Cells That Maintains Homeostasis in the Small IntestineIL-10-producing intestinal macrophages prevent excessive antibacterial innate immunity by limiting IL-23 synthesisCross-talk between type 3 innate lymphoid cells and the gut microbiota in inflammatory bowel disease.Interleukin-1 (IL-1) signaling in intestinal stromal cells controls KC/ CXCL1 secretion, which correlates with recruitment of IL-22- secreting neutrophils at early stages of Citrobacter rodentium infection.Interleukin-7 produced by intestinal epithelial cells in response to Citrobacter rodentium infection plays a major role in innate immunity against this pathogen.IL-17a and IL-22 Induce Expression of Antimicrobials in Gastrointestinal Epithelial Cells and May Contribute to Epithelial Cell Defense against Helicobacter pyloriTir Triggers Expression of CXCL1 in Enterocytes and Neutrophil Recruitment during Citrobacter rodentium InfectionIntestinal macrophages arising from CCR2(+) monocytes control pathogen infection by activating innate lymphoid cells.The cell surface receptor Slamf6 modulates innate immune responses during Citrobacter rodentium-induced colitis.Fluorescent reporter systems for tracking probiotic lactic acid bacteria and bifidobacteria.Epithelial-intrinsic IKKα expression regulates group 3 innate lymphoid cell responses and antibacterial immunity.Innate lymphoid cell interactions with microbiota: implications for intestinal health and disease.Innate lymphoid cells in the initiation, regulation and resolution of inflammationHomeostatic PPARα Signaling Limits Inflammatory Responses to Commensal Microbiota in the Intestine.Emerging roles for antigen presentation in establishing host-microbiome symbiosisThe AIM2 inflammasome is a central regulator of intestinal homeostasis through the IL-18/IL-22/STAT3 pathway.Distinct Kinase-Independent Role of RIPK3 in CD11c+ Mononuclear Phagocytes in Cytokine-Induced Tissue RepairExpression of fluorescent proteins in bifidobacteria for analysis of host-microbe interactions.Regulation of the immune system by the resident intestinal bacteria.New friendships and old feuds: relationships between innate lymphoid cells and microbial communities.Innate lymphoid cells: from border protection to the initiation of inflammatory diseases.Development and regulation of RORγt(+) innate lymphoid cells.Innate lymphoid cells in intestinal immunity and inflammation.Macrophages in gastrointestinal homeostasis and inflammation.Citrobacter rodentium: a model enteropathogen for understanding the interplay of innate and adaptive components of type 3 immunity.Vitamin A Controls the Presence of RORγ+ Innate Lymphoid Cells and Lymphoid Tissue in the Small Intestine.CX3CR1⁺ cells facilitate the activation of CD4 T cells in the colonic lamina propria during antigen-driven colitis.Group 3 Innate Lymphoid Cells: Communications Hubs of the Intestinal Immune System.
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CX(3)CR1(+) macrophages support IL-22 production by innate lymphoid cells during infection with Citrobacter rodentium.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
CX(3)CR1(+) macrophages suppor ...... on with Citrobacter rodentium.
@ast
CX(3)CR1(+) macrophages suppor ...... on with Citrobacter rodentium.
@en
type
label
CX(3)CR1(+) macrophages suppor ...... on with Citrobacter rodentium.
@ast
CX(3)CR1(+) macrophages suppor ...... on with Citrobacter rodentium.
@en
prefLabel
CX(3)CR1(+) macrophages suppor ...... on with Citrobacter rodentium.
@ast
CX(3)CR1(+) macrophages suppor ...... on with Citrobacter rodentium.
@en
P2093
P2860
P356
P1433
P1476
CX(3)CR1(+) macrophages suppor ...... on with Citrobacter rodentium.
@en
P2093
C U Riedel
K Radulovic
P2860
P2888
P304
P356
10.1038/MI.2012.61
P577
2012-08-01T00:00:00Z