Mesenchymal cell targeting by TNF as a common pathogenic principle in chronic inflammatory joint and intestinal diseases. - Wikidata - awgldk - TriplyDB

Mesenchymal cell targeting by TNF as a common pathogenic principle in chronic inflammatory joint and intestinal diseases.

Mesenchymal cell targeting by TNF as a common pathogenic principle in chronic inflammatory joint and intestinal diseases.

http://www.wikidata.org/entity/Q36502842

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The Role of the Transcriptional Regulation of Stromal Cells in Chronic Inflammation Targeting TNF receptors in rheumatoid arthritis Murine models of inflammatory bowel disease (IBD): challenges of modeling human disease Redeeming an old foe: protective as well as pathophysiological roles for tumor necrosis factor in inflammatory bowel disease The control of inflammation via the phosphorylation and dephosphorylation of tristetraprolin: a tale of two phosphatases Whole-body deletion of LPS-induced TNF-α factor (LITAF) markedly improves experimental endotoxic shock and inflammatory arthritis Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility.Inflammatory bowel disease Higher Levels of Secretory IgA Are Associated with Low Disease Activity Index in Patients with Reactive Arthritis and Undifferentiated Spondyloarthritis TNF-α stimulates colonic myofibroblast migration via COX-2 and Hsp27 Extensive phenotypic characterization of a new transgenic mouse reveals pleiotropic perturbations in physiology due to mesenchymal hGH minigene expression.IL-6-dependent PGE2 secretion by mesenchymal stem cells inhibits local inflammation in experimental arthritis Molecular control of steady-state dendritic cell maturation and immune homeostasis.A crucial role for tumor necrosis factor receptor 1 in synovial lining cells and the reticuloendothelial system in mediating experimental arthritis Intestinal dendritic cells in the pathogenesis of inflammatory bowel disease.Intestinal myofibroblast-specific Tpl2-Cox-2-PGE2 pathway links innate sensing to epithelial homeostasis.Small intestine inflammation in Roquin-mutant and Roquin-deficient mice.TNF-α and LPA promote synergistic expression of COX-2 in human colonic myofibroblasts: role of LPA-mediated transactivation of upregulated EGFR.Intestinal epithelial cells as producers but not targets of chronic TNF suffice to cause murine Crohn-like pathology TNF-α potentiates lysophosphatidic acid-induced COX-2 expression via PKD in human colonic myofibroblasts.Intestinal myofibroblasts: targets for stem cell therapy Intestinal stricture in Crohn's disease.Tristetraprolin (TTP) coordinately regulates primary and secondary cellular responses to proinflammatory stimuli Protein kinase D1 mediates synergistic MMP-3 expression induced by TNF-α and bradykinin in human colonic myofibroblasts.Genome-wide association study of ankylosing spondylitis identifies non-MHC susceptibility loci.Remission of collagen-induced arthritis through combination therapy of microfracture and transplantation of thermogel-encapsulated bone marrow mesenchymal stem cells.Inflammatory bowel disease associated arthropathy Prolonged tumor necrosis factor α primes fibroblast-like synoviocytes in a gene-specific manner by altering chromatin.Inflammation, Innate Immunity, and the Intestinal Stromal Cell Niche: Opportunities and Challenges.A Feedback Loop between Inflammation and Zn Uptake.Spontaneous retrotransposon insertion into TNF 3'UTR causes heart valve disease and chronic polyarthritis.TNF-α induces upregulation of EGFR expression and signaling in human colonic myofibroblasts.Update on biologic pathways in inflammatory bowel disease and their therapeutic relevance.Expression of A20 by dendritic cells preserves immune homeostasis and prevents colitis and spondyloarthritis.IKKβ in intestinal mesenchymal cells promotes initiation of colitis-associated cancer.Mesenchymal stem cells can improve anal pressures after anal sphincter injury.Nuclear factor-κB modulates osteogenesis of periodontal ligament stem cells through competition with β-catenin signaling in inflammatory microenvironments.CollagenVI-Cre mice: A new tool to target stromal cells in secondary lymphoid organs.Protein kinase D mediates synergistic expression of COX-2 induced by TNF-{alpha} and bradykinin in human colonic myofibroblasts.Commercial development of cell-based therapeutics: strategic considerations along the drug to tissue spectrum.

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P2860

Mesenchymal cell targeting by TNF as a common pathogenic principle in chronic inflammatory joint and intestinal diseases.

http://www.wikidata.org/entity/Q36502842

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Dimitris L Kontoyiannis

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Dirk Elewaut

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Maria Apostolaki

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Peggy Jacques

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P2860

Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis

Generalized lacZ expression with the ROSA26 Cre reporter strain

Update on spondyloarthropathies

Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection

Z/EG, a double reporter mouse line that expresses enhanced green fluorescent protein upon Cre-mediated excision

Genetic dissection of the cellular pathways and signaling mechanisms in modeled tumor necrosis factor-induced Crohn's-like inflammatory bowel disease.

Myofibroblasts. II. Intestinal subepithelial myofibroblasts.

Fibroblasts regulate the switch from acute resolving to chronic persistent inflammation.

How important are T cells in chronic rheumatoid synovitis?: II. T cell-independent mechanisms from beginning to end.

Matrix metalloproteinases-3, -8, -9 as markers of disease activity and joint damage progression in early rheumatoid arthritis

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331-337

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2

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205

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Marietta Armaka

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