RAGE signaling sustains inflammation and promotes tumor development. - Wikidata - awgldk - TriplyDB

RAGE signaling sustains inflammation and promotes tumor development.

RAGE signaling sustains inflammation and promotes tumor development.

http://www.wikidata.org/entity/Q36502858

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S100A9 interaction with TLR4 promotes tumor growth The secreted protein S100A7 (psoriasin) is induced by telomere dysfunction in human keratinocytes independently of a DNA damage response and cell cycle regulators Receptor for advanced glycation end products and its involvement in inflammatory diseases Gene from a psoriasis susceptibility locus primes the skin for inflammation Proinflammatory S100 proteins regulate the accumulation of myeloid-derived suppressor cells RAGE, carboxylated glycans and S100A8/A9 play essential roles in colitis-associated carcinogenesis Aggressiveness Niche: Can It Be the Foster Ground for Cancer Metastasis Precursors?Molecular signaling involving intrinsically disordered proteins in prostate cancer Involvement of HMGB1 in Resistance to Tumor Vessel-Targeted, Monoclonal Antibody-Based Immunotherapy PAMPs and DAMPs: signal 0s that spur autophagy and immunity Tumour hypoxia promotes melanoma growth and metastasis via High Mobility Group Box-1 and M2-like macrophages Damage-associated molecular patterns in cancer: a double-edged sword Immature myeloid cells directly contribute to skin tumor development by recruiting IL-17-producing CD4+ T cells IL-17 mediated inflammation promotes tumor growth and progression in the skin Common interactions between S100A4 and S100A9 defined by a novel chemical probe Deficiency of the Kruppel-like factor KLF4 correlates with increased cell proliferation and enhanced skin tumorigenesis Glyoxalase 1-419C>A variant is associated with oxidative stress: implications in prostate cancer progression Topical application of a platelet activating factor receptor agonist suppresses phorbol ester-induced acute and chronic inflammation and has cancer chemopreventive activity in mouse skin S100A8/A9 stimulates keratinocyte proliferation in the development of squamous cell carcinoma of the skin via the receptor for advanced glycation-end products Blocking the Interactions between Calcium-Bound S100A12 Protein and the V Domain of RAGE Using Tranilast Zinc replenishment reverses overexpression of the proinflammatory mediator S100A8 and esophageal preneoplasia in the rat TIRAP, an adaptor protein for TLR2/4, transduces a signal from RAGE phosphorylated upon ligand binding The multiple faces of RAGE--opportunities for therapeutic intervention in aging and chronic disease Commercial processed soy-based food product contains glycated and glycoxidated lunasin proteoforms Apoptotic cell recognition receptors and scavenger receptors.Proteomic and pathway analyses reveal a network of inflammatory genes associated with differences in skin tumor promotion susceptibility in DBA/2 and C57BL/6 mice.Disturbances in metabolic, transport and structural genes in experimental colonic inflammation in the rat: a longitudinal genomic analysis.The receptor for advanced glycation end products promotes pancreatic carcinogenesis and accumulation of myeloid-derived suppressor cells.Identification of the Rage-dependent gene regulatory network in a mouse model of skin inflammation.Deletion of bone marrow-derived receptor for advanced glycation end products inhibits atherosclerotic plaque progression.The danger signal S100B integrates pathogen- and danger-sensing pathways to restrain inflammation TLR4-mediated skin carcinogenesis is dependent on immune and radioresistant cells.CD14(+)S100A9(+) monocytic myeloid-derived suppressor cells and their clinical relevance in non-small cell lung cancer Alternatively spliced RAGEv1 inhibits tumorigenesis through suppression of JNK signaling S100A9 a new marker for monocytic human myeloid-derived suppressor cells.Opposing function of MYBBP1A in proliferation and migration of head and neck squamous cell carcinoma cells.Longistatin in tick saliva blocks advanced glycation end-product receptor activation.HMGB1 enhances immune suppression by facilitating the differentiation and suppressive activity of myeloid-derived suppressor cells The association of HMGB1 expression with clinicopathological significance and prognosis in hepatocellular carcinoma: a meta-analysis and literature review.RAGE mediates S100A7-induced breast cancer growth and metastasis by modulating the tumor microenvironment

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P2860

RAGE signaling sustains inflammation and promotes tumor development.

http://www.wikidata.org/entity/Q36502858

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2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

RAGE signaling sustains inflammation and promotes tumor development.

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RAGE signaling sustains inflammation and promotes tumor development.

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RAGE signaling sustains inflammation and promotes tumor development.

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RAGE signaling sustains inflammation and promotes tumor development.

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RAGE signaling sustains inflammation and promotes tumor development.

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RAGE signaling sustains inflammation and promotes tumor development.

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P1433

Journal of Experimental Medicine

P1476

RAGE signaling sustains inflammation and promotes tumor development.

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P2093

Alexander Enk

http://www.w3.org/2001/XMLSchema#string

Angelika Bierhaus

http://www.w3.org/2001/XMLSchema#string

Astrid Riehl

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Bernd Arnold

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Gerhard Fürstenberger

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Jochen Hess

http://www.w3.org/2001/XMLSchema#string

Julia Németh

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Karin Müller-Decker

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Moritz Durchdewald

http://www.w3.org/2001/XMLSchema#string

Peter Angel

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P2860

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

The Hallmarks of Cancer

Inflammation and cancer

Diabetes-associated sustained activation of the transcription factor nuclear factor-kappaB

Mrp8 and Mrp14 are endogenous activators of Toll-like receptor 4, promoting lethal, endotoxin-induced shock

Tumour-mediated upregulation of chemoattractants and recruitment of myeloid cells predetermines lung metastasis

NF-kappaB inhibition: a double-edged sword in cancer?

Deficiency of either cyclooxygenase (COX)-1 or COX-2 alters epidermal differentiation and reduces mouse skin tumorigenesis

Paradoxical roles of the immune system during cancer development

NF-kappaB: linking inflammation and immunity to cancer development and progression

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275-285

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10.1084/JEM.20070679

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2

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205

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Christoffer Gebhardt

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2008-01-21T00:00:00Z

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5642685

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18208974

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2271015

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