Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20.
about
The kinase IKKα inhibits activation of the transcription factor NF-κB by phosphorylating the regulatory molecule TAX1BP1A20 is an early responding negative regulator of Toll-like receptor 5 signalling in intestinal epithelial cells during inflammationInterferon regulatory factor 3 inhibits astrocyte inflammatory gene expression through suppression of the proinflammatory miR-155 and miR-155*Ubiquitin in the activation and attenuation of innate antiviral immunityThe role of ubiquitylation in immune defence and pathogen evasionHost innate immune responses to sepsisNoncoding RNAs regulate NF-κB signaling to modulate blood vessel inflammationModulation of Intestinal TLR4-Inflammatory Signaling Pathways by Probiotic Microorganisms: Lessons Learned from Lactobacillus jensenii TL2937Ubiquitin in the immune systemInnate immune signaling in defense against intestinal microbesLinear ubiquitination in immunityDoes antigen masking by ubiquitin chains protect from the development of autoimmune diseases?A20 deficiency causes spontaneous neuroinflammation in miceThe Trypanosoma cruzi protease cruzain mediates immune evasionRole for B-cell adapter for PI3K (BCAP) as a signaling adapter linking Toll-like receptors (TLRs) to serine/threonine kinases PI3K/AktARIH2 is essential for embryogenesis, and its hematopoietic deficiency causes lethal activation of the immune systemInflammatory bowel diseaseSignaling network of dendritic cells in response to pathogens: a community-input supported knowledgebaseRNF11, a new piece in the A20 puzzle.Hypomorphic A20 expression confers susceptibility to psoriasis.Oncogenic activation of NF-kappaB.CD47 and TLR-2 cross-talk regulates neutrophil transmigration.The regulation of TGF-β/SMAD signaling by protein deubiquitination.Molecular control of steady-state dendritic cell maturation and immune homeostasis.Mechanisms of intestinal inflammation and development of associated cancers: lessons learned from mouse models.Pathogenetic importance and therapeutic implications of NF-κB in lymphoid malignancies.Toll-like receptors in kidney disease.Enterocyte-specific A20 deficiency sensitizes to tumor necrosis factor-induced toxicity and experimental colitis.TANK is a negative regulator of Toll-like receptor signaling and is critical for the prevention of autoimmune nephritis.TNFAIP3 maintains intestinal barrier function and supports epithelial cell tight junctions.The ubiquitin modifying enzyme A20 restricts B cell survival and prevents autoimmunity.MTA1 coregulator regulates LPS response via MyD88-dependent signalingToll-like receptors in inflammatory bowel diseases: a decade later.Intestinal epithelial expression of TNFAIP3 results in microbial invasion of the inner mucus layer and induces colitis in IL-10-deficient mice.Ubiquitin becomes ubiquitous in cancer: emerging roles of ubiquitin ligases and deubiquitinases in tumorigenesis and as therapeutic targets.Ubiquitin makes its mark on immune regulation.A20 Ameliorates Intracerebral Hemorrhage-Induced Inflammatory Injury by Regulating TRAF6 Polyubiquitination.Genetic relationships between A20/TNFAIP3, chronic inflammation and autoimmune disease.Immunoregulatory effect of bifidobacteria strains in porcine intestinal epithelial cells through modulation of ubiquitin-editing enzyme A20 expressionA20 restricts wnt signaling in intestinal epithelial cells and suppresses colon carcinogenesis.
P2860
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P2860
Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20.
@ast
Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20.
@en
type
label
Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20.
@ast
Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20.
@en
prefLabel
Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20.
@ast
Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20.
@en
P2093
P2860
P50
P356
P1476
Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20
@en
P2093
Barbara A Malynn
Bettina Lee
Nataliya Shifrin
Osamu Hitotsumatsu
Rommel Advincula
P2860
P304
P356
10.1084/JEM.20071108
P407
P577
2008-02-11T00:00:00Z