Matrix metalloproteinase-dependent shedding of intercellular adhesion molecule-5 occurs with long-term potentiation.
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Diverse impact of acute and long-term extracellular proteolytic activity on plasticity of neuronal excitabilityActivity dependent CAM cleavage and neurotransmissionCopper signaling in the mammalian nervous system: synaptic effectsSynaptic circuit remodelling by matrix metalloproteinases in health and diseaseMMPs and soluble ICAM-5 increase neuronal excitability within in vitro networks of hippocampal neuronsSoluble ICAM-5, a product of activity dependent proteolysis, increases mEPSC frequency and dendritic expression of GluA1Synaptically released matrix metalloproteinase activity in control of structural plasticity and the cell surface distribution of GluA1-AMPA receptorsICAM-5 affects spine maturation by regulation of NMDA receptor binding to α-actinin.Intercellular Adhesion Molecular-5 as Marker in HIV Associated Neurocognitive Disorder.Multifaceted Roles of Metzincins in CNS Physiology and Pathology: From Synaptic Plasticity and Cognition to Neurodegenerative DisordersSynaptic loss and retention of different classic cadherins with LTP-associated synaptic structural remodeling in vivo.Matrix metalloproteinases, synaptic injury, and multiple sclerosisMatrix metalloproteinases regulate the formation of dendritic spine head protrusions during chemically induced long-term potentiation.Metzincin proteases and their inhibitors: foes or friends in nervous system physiology?Interactions of HIV and drugs of abuse: the importance of glia, neural progenitors, and host genetic factors.DP-b99 modulates matrix metalloproteinase activity and neuronal plasticity.MT5-MMP, ADAM-10, and N-cadherin act in concert to facilitate synapse reorganization after traumatic brain injuryProteolytic regulation of synaptic plasticity in the mouse primary visual cortex: analysis of matrix metalloproteinase 9 deficient miceMatrix metalloproteinase-9 and -2 enhance the ligand sensitivity of photoreceptor cyclic nucleotide-gated channels.The TARC/sICAM5 Ratio in Patient Plasma is a Candidate Biomarker for Drug Resistant Epilepsy.Interactions between ICAM-5 and β1 integrins regulate neuronal synapse formationTransient ECM protease activity promotes synaptic plasticity.Lectican proteoglycans, their cleaving metalloproteinases, and plasticity in the central nervous system extracellular microenvironment.Protease induced plasticity: matrix metalloproteinase-1 promotes neurostructural changes through activation of protease activated receptor 1Subcellular localization of intercellular adhesion molecule-5 (telencephalin) in the visual cortex is not developmentally regulated in the absence of matrix metalloproteinase-9.Matrix Metalloprotease 3 Activity Supports Hippocampal EPSP-to-Spike Plasticity Following Patterned Neuronal Activity via the Regulation of NMDAR Function and Calcium Flux.Resveratrol regulates neuro-inflammation and induces adaptive immunity in Alzheimer's disease.Extracellular matrix molecules, their receptors, and secreted proteases in synaptic plasticity.How can microbial interactions with the blood-brain barrier modulate astroglial and neuronal function?Matrix metalloproteinase dependent cleavage of cell adhesion molecules in the pathogenesis of CNS dysfunction with HIV and methamphetamine.Proteolytic remodeling of the synaptic cell adhesion molecules (CAMs) by metzincins in synaptic plasticity.Matrix metalloproteinase-9 involvement in the structural plasticity of dendritic spines.Implication of matrix metalloproteinases in regulating neuronal disorder.MMP-9 in translation: from molecule to brain physiology, pathology, and therapy.Interactions between intercellular adhesion molecule-5 positive elements and their surroundings in the rodent visual cortex.Dopamine increases NMDA-stimulated calcium flux in striatopallidal neurons through a matrix metalloproteinase-dependent mechanism.Activity-dependent proteolytic cleavage of cell adhesion molecules regulates excitatory synaptic development and function.Methamphetamine-associated cleavage of the synaptic adhesion molecule intercellular adhesion molecule-5.Neural functions of matrix metalloproteinases: plasticity, neurogenesis, and diseaseBuilding and remodeling synapses.
P2860
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P2860
Matrix metalloproteinase-dependent shedding of intercellular adhesion molecule-5 occurs with long-term potentiation.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Matrix metalloproteinase-depen ...... s with long-term potentiation.
@ast
Matrix metalloproteinase-depen ...... s with long-term potentiation.
@en
type
label
Matrix metalloproteinase-depen ...... s with long-term potentiation.
@ast
Matrix metalloproteinase-depen ...... s with long-term potentiation.
@en
prefLabel
Matrix metalloproteinase-depen ...... s with long-term potentiation.
@ast
Matrix metalloproteinase-depen ...... s with long-term potentiation.
@en
P2093
P2860
P1433
P1476
Matrix metalloproteinase-depen ...... s with long-term potentiation.
@en
P2093
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2009.12.061
P407
P577
2010-01-04T00:00:00Z