C1q-induced LRP1B and GPR6 proteins expressed early in Alzheimer disease mouse models, are essential for the C1q-mediated protection against amyloid-β neurotoxicity.
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The Role of Inflammatory Mediators in the Pathogenesis of Alzheimer's DiseaseComplement activation in the context of stem cells and tissue repairMicroglia communication: Parallels between aging and Alzheimer's diseaseThe ischemic environment drives microglia and macrophage functionMacrophages migrate in an activation-dependent manner to chemokines involved in neuroinflammation.New insights on the role of microglia in synaptic pruning in health and diseaseComplement modulation of T cell immune responses during homeostasis and disease.Versatility of the complement system in neuroinflammation, neurodegeneration and brain homeostasisComplement protein C1q modulates neurite outgrowth in vitro and spinal cord axon regeneration in vivo.Variation in complement protein C1q is not a major contributor to cognitive impairment in Parkinson's disease.Complement 5a Enhances Hepatic Metastases of Colon Cancer via Monocyte Chemoattractant Protein-1-mediated Inflammatory Cell Infiltration.Application of quantitative trait locus mapping and transcriptomics to studies of the senescence-accelerated phenotype in rats.RNA-sequencing reveals transcriptional up-regulation of Trem2 in response to bexarotene treatment.Active immunization against complement factor C5a: a new therapeutic approach for Alzheimer's diseaseAnalysis of the Putative Role of CR1 in Alzheimer's Disease: Genetic Association, Expression and Function.Fundamental role of C1q in autoimmunity and inflammationA dramatic increase of C1q protein in the CNS during normal aging.What does complement do in Alzheimer's disease? Old molecules with new insights.Complement and microglia mediate early synapse loss in Alzheimer mouse models.Modulation of p25 and inflammatory pathways by fisetin maintains cognitive function in Alzheimer's disease transgenic miceDissecting the Contribution of Vascular Alterations and Aging to Alzheimer's Disease.Microglia-Mediated Inflammation and Neurodegenerative Disease.Microglia and Monocyte-Derived Macrophages in StrokeCalreticulin contributes to C1q-dependent recruitment of microglia in the leech Hirudo medicinalis following a CNS injuryChaperones as Suppressors of Protein Misfolded Oligomer ToxicityC1q and HMGB1 reciprocally regulate human macrophage polarizationC1q: A fresh look upon an old molecule.Microglia and C9orf72 in neuroinflammation and ALS and frontotemporal dementia.Association Between Microglia, Inflammatory Factors, and Complement with Loss of Hippocampal Mossy Fiber Synapses Induced by Trimethyltin.Cell-specific deletion of C1qa identifies microglia as the dominant source of C1q in mouse brain.Autophagy is involved in oral rAAV/Aβ vaccine-induced Aβ clearance in APP/PS1 transgenic mice.Prevention of C5aR1 signaling delays microglial inflammatory polarization, favors clearance pathways and suppresses cognitive loss.C5a Increases the Injury to Primary Neurons Elicited by Fibrillar Amyloid Beta.An Update on Non-CB1, Non-CB2 Cannabinoid Related G-Protein-Coupled Receptors.Immunity and inflammation in neurodegenerative diseases.
P2860
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P2860
C1q-induced LRP1B and GPR6 proteins expressed early in Alzheimer disease mouse models, are essential for the C1q-mediated protection against amyloid-β neurotoxicity.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
C1q-induced LRP1B and GPR6 pro ...... ainst amyloid-β neurotoxicity.
@ast
C1q-induced LRP1B and GPR6 pro ...... ainst amyloid-β neurotoxicity.
@en
type
label
C1q-induced LRP1B and GPR6 pro ...... ainst amyloid-β neurotoxicity.
@ast
C1q-induced LRP1B and GPR6 pro ...... ainst amyloid-β neurotoxicity.
@en
prefLabel
C1q-induced LRP1B and GPR6 pro ...... ainst amyloid-β neurotoxicity.
@ast
C1q-induced LRP1B and GPR6 pro ...... ainst amyloid-β neurotoxicity.
@en
P2093
P2860
P356
P1476
C1q-induced LRP1B and GPR6 pro ...... ainst amyloid-β neurotoxicity.
@en
P2093
Andrea J Tenner
Francisca Benavente
Marie E Benoit
Michael X Hernandez
Minhan L Dinh
Osvaldo Vasquez
P2860
P304
P356
10.1074/JBC.M112.400168
P407
P577
2012-11-13T00:00:00Z